{"title":"All Products","description":"","products":[{"product_id":"kpv-10mg","title":"KPV Peptide","description":"KPV (5MG) \nOverview\nThis blend combines KPV, creating a powerful regenerative and healing formula for muscle, tendon, joint, and skin repair.\nKey Benefits:\n✔ Accelerates wound healing and tissue repair\n✔ Reduces inflammation and oxidative stress\n✔ Supports joint, muscle, and skin regeneration\n✔ Boosts collagen production and angiogenesis\nMechanism of Action:\n\n\n\nKPV: KP-V: Rapidly lowers inflammation and supports gut and tissue repair by modulating immune pathways.\n\n\n\nCitations:\n\nSorg, H. et al. “GHK-Cu peptide and its wound healing effects,” J Dermatol Sci, 2017.\nNixon, J. et al. “BPC-157 and tendon healing,” J Orthop Res, 2016.\nGoldstein, A. et al. “Thymosin beta-4 and its role in regenerative medicine,” Regen Med, 2015.","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989559972073,"sku":"PS062-1-1-2","price":19000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/KPV-10mg.jpg?v=1778509454"},{"product_id":"kpv-5mg","title":"KPV 5mg","description":"KPV (5MG) \nOverview\nThis blend combines KPV, creating a powerful regenerative and healing formula for muscle, tendon, joint, and skin repair.\nKey Benefits:\n✔ Accelerates wound healing and tissue repair\n✔ Reduces inflammation and oxidative stress\n✔ Supports joint, muscle, and skin regeneration\n✔ Boosts collagen production and angiogenesis\nMechanism of Action:\n\n\n\nKPV: KP-V: Rapidly lowers inflammation and supports gut and tissue repair by modulating immune pathways.\n\n\n\nCitations:\n\nSorg, H. et al. “GHK-Cu peptide and its wound healing effects,” J Dermatol Sci, 2017.\nNixon, J. et al. “BPC-157 and tendon healing,” J Orthop Res, 2016.\nGoldstein, A. et al. “Thymosin beta-4 and its role in regenerative medicine,” Regen Med, 2015.","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560004841,"sku":"PS062-1-1","price":19000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/KPV-5mg.jpg?v=1778509455"},{"product_id":"igf-1-lr3-1mg","title":"IGF-1 LR3 .1mg","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nIGF-1 LR3 (Insulin-Like Growth Factor-1 Long R3) is a synthetic modified form of IGF-1, designed to extend its biological activity by preventing binding to IGF-binding proteins. This modification makes IGF-1 LR3 up to 120 times longer-lasting than standard IGF-1, significantly enhancing its effects on cell growth, metabolism, and muscle repair.\u003cbr\u003e\nResearch suggests IGF-1 LR3 may:\u003c\/p\u003e\n\n\u003cp\u003eStimulate cell division and tissue growth, affecting muscles, bones, nerves, and vital organs.\u003cbr\u003e\nEnhance fat metabolism, leading to reduced adipose tissue and improved glucose regulation.\u003cbr\u003e\nSupport muscle repair and hypertrophy, by counteracting myostatin’s inhibitory effects.\u003cbr\u003e\nPromote longevity and cellular maintenance, by reducing age-related tissue degeneration.\u003cbr\u003e\nProtect against glucocorticoid-induced muscle loss, supporting muscle preservation in individuals undergoing corticosteroid therapy.\u003c\/p\u003e\n\n\u003cp\u003eDue to these properties, IGF-1 LR3 has been explored in muscle-wasting conditions, diabetes research, and anti-aging therapies.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\nIGF-1 LR3 and Cell Growth\u003c\/p\u003e\n\n\u003cp\u003eIGF-1 LR3 is a potent stimulator of cell division and proliferation.\u003cbr\u003e\nPrimarily affects muscles and bones, but also influences liver, kidney, nerve, skin, lung, and blood tissues.\u003cbr\u003e\nLonger circulation time compared to IGF-1, making it more effective in promoting cell maturation and tissue regeneration.\u003cbr\u003e\nPotential application: Regenerative medicine and muscle recovery.\u003c\/p\u003e\n\n\u003cp\u003eIGF-1 LR3 and Fat Metabolism\u003c\/p\u003e\n\n\u003cp\u003eIGF-1 LR3 binds to insulin receptors, enhancing glucose uptake in muscle, liver, and nerve cells.\u003cbr\u003e\nThis leads to lower blood sugar levels, triggering fat breakdown and a net decrease in adipose tissue.\u003cbr\u003e\nPotential application: Obesity management and diabetes therapy.\u003c\/p\u003e\n\n\u003cp\u003eIGF-1 LR3 and Muscle Growth (Myostatin Inhibition)\u003c\/p\u003e\n\n\u003cp\u003eMyostatin is a natural inhibitor of muscle growth—IGF-1 LR3 blocks its effects, allowing enhanced muscle repair and hypertrophy.\u003cbr\u003e\nResearch suggests IGF-1 LR3 may be beneficial for conditions such as Duchenne Muscular Dystrophy (DMD) and muscle atrophy due to immobility or chronic illness.\u003cbr\u003e\nPotential application: Muscle-wasting disorder treatment and sports performance research.\u003c\/p\u003e\n\n\u003cp\u003eIGF-1 LR3 and Longevity Research\u003c\/p\u003e\n\n\u003cp\u003eIGF-1 LR3 supports tissue repair and maintenance, offering protective benefits against age-related decline.\u003cbr\u003e\nStudies in livestock models (cows and pigs) suggest IGF-1 LR3 can counteract cellular aging effects.\u003cbr\u003e\nOngoing research in mice explores IGF-1 LR3’s role in preventing dementia, muscle loss, and kidney disease.\u003cbr\u003e\nPotential application: Anti-aging and age-related disease prevention.\u003c\/p\u003e\n\n\u003cp\u003eIGF-1 LR3 and Glucocorticoid Protection\u003c\/p\u003e\n\n\u003cp\u003eGlucocorticoids are commonly used anti-inflammatory drugs, but long-term use can cause muscle wasting, fat gain, and bone density loss.\u003cbr\u003e\nIGF-1 LR3 counteracts these negative effects, allowing for more effective corticosteroid therapy with reduced side effects.\u003cbr\u003e\nPotential application: Muscle preservation in patients undergoing glucocorticoid therapy.\u003c\/p\u003e\n\n\u003cp\u003eSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eMolecular Formula: C₄₀₀H₆₂₅N₁₁₁O₁₁₅S₉\u003cbr\u003e\nMolecular Weight: 9117.5 g\/mol\u003cbr\u003e\nAmino Acid Sequence:\u003cbr\u003e\nMFPAMPLSSL FVNGPRTLCG AELVDALQFV CGDRGFYFNK PTGYGSSSRR APQTGIVDEC CFRSCDLRRL EMYCAPLKPA KSA\u003cbr\u003e\nCAS Registry Number: 946870-92-4\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003c\/p\u003e\n\n\u003cp\u003eAdipose Tissue-Derived Stem Cell Secreted IGF-1 Protects Myoblasts from the Negative Effect of Myostatin. Hindawi Journal.\u003cbr\u003e\nN. Li, Q. Yang, R. G. Walker, T. B. Thompson, M. Du, and B. D. Rodgers. Myostatin Attenuation In Vivo Reduces Adiposity, but Activates Adipogenesis. Endocrinology (2016).\u003cbr\u003e\nE. Corpas, S. M. Harman, and M. R. Blackman. Human growth hormone and human aging. Endocr. Rev. (1993).\u003cbr\u003e\nW. E. Sonntag, A. Csiszar, R. deCabo, L. Ferrucci, and Z. Ungvari. Diverse roles of growth hormone and insulin-like growth factor-1 in mammalian aging: progress and controversies. J. Gerontol. A. Biol. Sci. Med. Sci. (2012).\u003cbr\u003e\nIGF-I\/IGFBP system: metabolism outline and physical exercise. NCBI.\u003cbr\u003e\nB. Y. Hanaoka, C. A. Peterson, C. Horbinski, and L. J. Crofford. Implications of glucocorticoid therapy in idiopathic inflammatory myopathies. Nat. Rev. Rheumatol. (2012).\u003cbr\u003e\nA. Philippou, A. Halapas, M. Maridaki, M. Koutsilieris. Expression of IGF-1 isoforms after exercise-induced muscle damage in humans. J. Musculoskelet Neuronal Interact. (2007).\u003cbr\u003e\nA. Philippou, E. Papageorgiou, G. Bogdanis, A. Halapas. Characterization of the MGF E peptide actions in vitro. In Vivo (2009).\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560037609,"sku":"PS049-1","price":19000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/IGF-1-LR3-1mg-1.jpg?v=1778509456"},{"product_id":"5-amino-1mq-5mg","title":"5 AMINO-1MQ 5MG","description":"\u003cp\u003e5-Amino 1mq – 5mg Vial\u003c\/p\u003e\n\n\u003cp\u003eResearch-Grade Synthetic Compound\u003cbr\u003e\nContents: 5mg of 5-Amino 1mq lyophilized powder\u003cbr\u003e\nForm: Freeze-Dried Powder\u003cbr\u003e\nPurity: ≥99% (HPLC Verified)\u003c\/p\u003e\n\n\u003cp\u003ePackaging: Supplied in a sterile, sealed vial with tamper-evident cap for laboratory use.\u003cbr\u003e\nNotice: This product is provided strictly for laboratory research use only. It is not intended for human or animal consumption, nor for diagnostic or therapeutic use.\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"One-time purchase","offer_id":51137696858345,"sku":"PS061-2","price":19000.0,"currency_code":"PKR","in_stock":true},{"title":"20%  Discount Subscribe","offer_id":51137696891113,"sku":"PS061-1","price":19000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/5-Amino-1MQ-5mg.jpg?v=1778509456"},{"product_id":"selank-10mg","title":"Selank 10MG","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nSelank is a synthetic analogue of tuftsin, a naturally occurring immunomodulatory peptide. Originally developed in Russia, it has been extensively studied for its anxiolytic (anti-anxiety), nootropic (cognitive-enhancing), and immune-boosting properties.\u003cbr\u003e\nResearch suggests that Selank may:\u003c\/p\u003e\n\n\u003cp\u003eReduce anxiety without causing sedation or addiction.\u003cbr\u003e\nEnhance memory, learning, and cognitive function.\u003cbr\u003e\nModulate immune system responses, aiding in stress-induced immune dysfunction.\u003cbr\u003e\nRegulate inflammatory cytokines, particularly by suppressing IL-6, a key marker of inflammation.\u003cbr\u003e\nIncrease brain-derived neurotrophic factor (BDNF), supporting neuroplasticity and brain health.\u003c\/p\u003e\n\n\u003cp\u003eDue to its effects on the GABAergic system, Selank is considered a potential alternative to benzodiazepines for treating anxiety-related disorders—without the risk of dependence or withdrawal.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\nSelank and Anxiety Reduction\u003cbr\u003e\nSelank interacts with the GABAergic system, similar to benzodiazepines, but with key distinctions:\u003c\/p\u003e\n\n\u003cp\u003eEnhances GABA receptor sensitivity, promoting a calming effect.\u003cbr\u003e\nDoes not cause dependence, withdrawal symptoms, or cognitive impairment, unlike traditional anxiolytics.\u003cbr\u003e\nIncreases the effectiveness of diazepam (Valium) when combined, suggesting a synergistic mechanism.\u003c\/p\u003e\n\n\u003cp\u003eClinical trials conducted in Russia demonstrated that Selank:\u003c\/p\u003e\n\n\u003cp\u003eSignificantly reduced anxiety and improved mood in patients with generalized anxiety disorder (GAD) and neurasthenia.\u003c\/p\u003e\n\n\u003cp\u003eThese findings highlight Selank’s potential for treating anxiety disorders without the sedation or addiction risksassociated with benzodiazepines.\u003cbr\u003e\nSelank and Cognitive Enhancement\u003cbr\u003e\nSelank has been shown to improve learning, memory retention, and recall through multiple mechanisms:\u003c\/p\u003e\n\n\u003cp\u003eEnhances hippocampal gene expression, supporting neuroplasticity.\u003cbr\u003e\nIncreases neuropeptides that promote long-term memory formation.\u003cbr\u003e\nProtects against cognitive decline after brain injury or stress exposure.\u003c\/p\u003e\n\n\u003cp\u003eAnimal studies indicate that Selank enhances cognitive function even in subjects without anxiety, suggesting direct nootropic effects.\u003cbr\u003e\nSelank and Immune System Modulation\u003cbr\u003e\nSelank is unique among nootropic peptides due to its immune-modulating properties:\u003c\/p\u003e\n\n\u003cp\u003eSuppresses IL-6, a pro-inflammatory cytokine associated with anxiety and depression.\u003cbr\u003e\nRegulates T-helper cell activity, helping to maintain immune balance.\u003cbr\u003e\nDemonstrates antiviral potential, possibly aiding recovery from respiratory infections.\u003c\/p\u003e\n\n\u003cp\u003eThese findings suggest that Selank may benefit individuals with stress-related immune suppression.\u003cbr\u003e\nSelank and Pain Perception\u003cbr\u003e\nSelank influences the endogenous opioid system by:\u003c\/p\u003e\n\n\u003cp\u003ePreventing the breakdown of enkephalins, the body’s natural pain-killing peptides.\u003cbr\u003e\nReducing stress-related hyperalgesia, a condition where anxiety or chronic stress amplifies pain sensitivity.\u003c\/p\u003e\n\n\u003cp\u003eThis mechanism mirrors the effects of opioid receptor modulators and certain antidepressants, making Selank a potential alternative treatment for chronic pain conditions.\u003cbr\u003e\nSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eMolecular Formula: C₃₃H₅₇N₁₁O₉\u003cbr\u003e\nMolecular Weight: 751.887 g\/mol\u003cbr\u003e\nAmino Acid Sequence: Thr-Lys-Pro-Arg-Pro-Gly-Pro\u003cbr\u003e\nCAS Registry Number: 129954-34-3\u003cbr\u003e\nPubChem Identifier: 11765600\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003c\/p\u003e\n\n\u003cp\u003eVolkova, A. et al. Selank Administration Affects the Expression of Some Genes Involved in GABAergic Neurotransmission. Front. Pharmacol. (2016).\u003cbr\u003e\nKasian, A. et al. Peptide Selank Enhances the Effect of Diazepam in Reducing Anxiety in Unpredictable Chronic Mild Stress Conditions in Rats. Behavioural Neurology (2017).\u003cbr\u003e\nZozulya, A. A. et al. The inhibitory effect of Selank on enkephalin-degrading enzymes as a possible mechanism of its anxiolytic activity. Bull. Exp. Biol. Med. (2001).\u003cbr\u003e\nSokolov, O. Y. et al. Effects of Selank on behavioral reactions and activities of plasma enkephalin-degrading enzymes in mice with different phenotypes of emotional and stress reactions. Bull. Exp. Biol. Med. (2003).\u003cbr\u003e\nUchakina, O. N. et al. Immunomodulatory effects of Selank in patients with anxiety-asthenic disorders. Zh. Nevrol. Psikhiatr. (2008).\u003cbr\u003e\nKolomin, T. et al. Expression of inflammation-related genes in mouse spleen under tuftsin analog Selank. Regul. Pept. (2011).\u003cbr\u003e\nSemenova, T. et al. Effect of Selank on cognitive processes after damage inflicted to the cerebral catecholamine system during early ontogeny. Bull. Exp. Biol. Med. (2014).\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560168681,"sku":"PS033-1","price":19000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/SELANK.png?v=1778509462"},{"product_id":"semax-10mg","title":"Semax 10mg","description":"OVERVIEW\nSemax is a synthetic derivative of adrenocorticotropic hormone (ACTH), originally developed in Russia for its neuroprotective, cognitive-enhancing, and immune-boosting properties. Research suggests that Semax may:\n\nIncrease brain-derived neurotrophic factor (BDNF), supporting neuroplasticity and learning.\nEnhance immune function and reduce inflammation.\nProtect neurons from ischemic damage, aiding in stroke and traumatic brain injury (TBI) recovery.\nImprove cardiovascular function, potentially enhancing blood circulation.\nAct as an antidepressant and anxiolytic (anti-anxiety agent) by modulating serotonin, dopamine, and BDNF levels.\n\nDue to its ability to support mental performance, reduce stress, and promote neurological recovery, Semax is widely researched in neurology, cognitive enhancement, and immune regulation.\nRESEARCH\nSemax and Stroke Recovery\nSemax has been extensively studied for its neuroprotective effects in stroke and traumatic brain injury (TBI).\n\nIn rat models of stroke, Semax activated 24 genes linked to vascular function, promoting neurogenesis and protecting against ischemic damage.\nClinical trials in stroke patients showed that those receiving Semax experienced faster motor function recovery and improved neurological outcomes.\nBDNF elevation facilitated brain plasticity and functional rehabilitation.\n\nThese findings suggest Semax could be a promising treatment for post-stroke recovery and neurodegenerative conditions.\nSemax and Cognitive Enhancement\nSemax has been studied for its potential as a nootropic (cognitive enhancer):\n\nIncreases BDNF levels in the hippocampus, improving learning and memory formation.\nEnhances prefrontal cortex activity, potentially boosting problem-solving skills and attention span.\nProtects against cognitive decline, possibly preventing age-related memory loss.\n\nThese neuroprotective and cognitive-enhancing effects make Semax a promising compound for cognitive longevity and brain health.\nSemax and the Default Mode Network (DMN)\n\nFunctional MRI studies suggest that Semax activates the Default Mode Network (DMN), a system responsible for:\n\nSocial cognition\nEnvironmental awareness\nResting-state brain activity\n\n\nEnhanced DMN function is linked to better focus, cognitive flexibility, and mental clarity.\nDMN disruptions are associated with neurodegenerative diseases like Alzheimer’s, suggesting Semax may play a neuroprotective role.\n\nSemax and Depression\/Anxiety\nSemax may offer antidepressant and anxiolytic effects by modulating serotonin, dopamine, and BDNF levels.\n\nAnimal studies show that Semax increases BDNF production, reducing depressive symptoms.\nUnlike SSRIs, Semax may work faster by directly increasing neurotrophic support, rather than altering serotonin levels alone.\nPotential as an adjunct therapy with antidepressants for enhanced efficacy.\n\nSemax and Pain Management\nResearch suggests Semax may help regulate pain perception by influencing the opioid system:\n\nPrevents the breakdown of enkephalins, the brain’s natural painkillers.\nMay provide an alternative approach for chronic pain management.\n\nSemax and Immune System Support\n\nSuppresses IL-6, a key inflammatory cytokine involved in stress response and immune dysfunction.\nEnhances T-helper cell activity, leading to better immune system balance.\nPotential antiviral properties, suggesting possible applications in infection recovery.\n\nSTRUCTURE\n\nMolecular Formula: C₃₉H₅₄N₁₀O₁₀S\nMolecular Weight: 854.99 g\/mol\nAmino Acid Sequence: Ac-Met-Glu-His-Phe-Pro-Gly-Pro\nCAS Registry Number: 80714-61-0\nPubChem Identifier: 122178\nSynonyms: Pro-Gly-Pro-ACTH, ACTH(4-10) analog\n\nCITATIONS\n\nLebedeva, I. S. et al. Effects of Semax on the Default Mode Network of the Brain. Bulletin of Experimental Biology and Medicine (2018).\nMars, R. B. et al. On the relationship between the default mode network and the social brain. Front. Hum. Neurosci. (2012).\nMedvedeva, E. V. et al. The peptide Semax affects the expression of genes related to the immune and vascular systems in rat brain focal ischemia. BMC Genomics (2014).\nGusev, E. I. et al. The efficacy of Semax in the treatment of patients at different stages of ischemic stroke.Zhurnal Nevrologii i Psikhiatrii (2018).\nAgapova, T. I. et al. Effect of Semax on BDNF and NGF gene expression in the rat hippocampus and frontal cortex. Molecular Genetics, Microbiology, and Virology (2008).\nScantlebury, M. H. et al. Adrenocorticotropic Hormone Protects Learning and Memory Function in Epileptic Mice. Neuroscience Letters (2017).\nDeltheil, T. et al. Behavioral and serotonergic consequences of increasing hippocampus BDNF protein levels.Neuropharmacology (2008).\nBobyntsev, I. I. et al. Influence of ACTG4-7-PGP (Semax) on the morphofunctional state of hepatocytes in chronic stress. Bulletin of Experimental Biology and Medicine (2017).\nBobyntsev, I. I. et al. The effect of ACTH-4-7-PGP peptide on lipid peroxidation and stress response in rats.Research Gate (2015).","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560201449,"sku":"PS034-1","price":19000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/Semax-5mg-scaled.jpg?v=1778509460"},{"product_id":"glp1-t-15mg","title":"GLP1-T 15MG","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nOriginally developed for type 2 diabetes management, Tirzepatide has demonstrated additional benefits, including cardiovascular protection and effective weight loss support. This synthetic peptide is derived from gastric inhibitory polypeptide (GIP) while also acting as a glucagon-like peptide-1 (GLP-1) receptor agonist, allowing it to:\u003c\/p\u003e\n\n\u003cp\u003eRegulate blood sugar levels\u003cbr\u003e\nEnhance insulin sensitivity\u003cbr\u003e\nPromote satiety\u003cbr\u003e\nFacilitate weight loss\u003c\/p\u003e\n\n\u003cp\u003eRESEARCH\u003cbr\u003e\nTirzepatide is a synthetic analogue of gastric inhibitory polypeptide (GIP), designed to stimulate insulin release and support the management of type 2 diabetes and non-alcoholic fatty liver disease. Composed of 39 amino acids, this peptide functions by activating both GIP and GLP-1 receptors, leading to improved insulin regulation and metabolic balance.\u003cbr\u003e\nLong-term use of Tirzepatide has been shown to increase adiponectin levels by up to 26%, contributing to:\u003c\/p\u003e\n\n\u003cp\u003eReduced hunger and cravings\u003cbr\u003e\nLower fasting insulin levels\u003cbr\u003e\nGreater insulin sensitivity\u003c\/p\u003e\n\n\u003cp\u003eThese metabolic effects result in:\u003c\/p\u003e\n\n\u003cp\u003eSignificant weight loss (approximately 11 kg or 25 lbs)\u003cbr\u003e\nEnhanced glucose control\u003cbr\u003e\nDecreased fat storage\u003cbr\u003e\nLower cardiovascular risk factors\u003c\/p\u003e\n\n\u003cp\u003eSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eAmino Acid Sequence: YE-Aib-GTFTSDYSI-Aib-LDKIAQ AFVQWLIAGGPSSGAPPPS\u003cbr\u003e\n(Aib is a non-standard amino acid: H H-C(CH3)COOH)\u003cbr\u003e\nMolecular Formula: C₂₂₅H₃₄₈N₄₈O₆₈\u003cbr\u003e\nMolecular Weight: 4813.527 g\/mol\u003cbr\u003e\nPubChem Identifier: 156588324\u003cbr\u003e\nCAS Registry Number: 2023788-19-2\u003cbr\u003e\nAlternative Names: P1206, LY3298176\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003cbr\u003e\nSource: PubChem\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560234217,"sku":"PS003-1","price":19000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/glp1t15mg.png?v=1778509463"},{"product_id":"kisspeptin-10-10mg","title":"Kisspeptin-10 10mg","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nKisspeptin-10 (KP-10) is a naturally occurring peptide that plays a critical role in reproductive hormone regulation. It serves as the primary trigger for the release of gonadotropin-releasing hormone (GnRH), which regulates key reproductive hormones such as testosterone, estrogen, and luteinizing hormone (LH).\u003cbr\u003e\nBeyond its well-established role in reproductive function, emerging research suggests Kisspeptin-10 may also contribute to:\u003c\/p\u003e\n\n\u003cp\u003eEnhancing fertility and reproductive health\u003cbr\u003e\nIncreasing testosterone levels in males\u003cbr\u003e\nRegulating mood, motivation, and behavior\u003cbr\u003e\nModulating energy balance and metabolism\u003cbr\u003e\nSuppressing tumor growth and cancer metastasis\u003c\/p\u003e\n\n\u003cp\u003eThese potential benefits make KP-10 a promising candidate for therapeutic applications in reproductive medicine, endocrinology, neurology, and oncology.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\nReproductive Hormone Regulation\u003c\/p\u003e\n\n\u003cp\u003eKisspeptin-10 initiates the release of GnRH, which stimulates LH and FSH secretion from the pituitary gland.\u003cbr\u003e\nThis regulatory function makes it essential for puberty onset, fertility, and overall reproductive function.\u003cbr\u003e\nPotential application: Treatment of delayed puberty, hypogonadism, and infertility.\u003c\/p\u003e\n\n\u003cp\u003eTestosterone Enhancement\u003c\/p\u003e\n\n\u003cp\u003eClinical studies show that KP-10 significantly increases testosterone levels in men within 90 minutes of administration.\u003cbr\u003e\nThis occurs by modulating LH pulse frequency, which fine-tunes the natural release of sex hormones.\u003cbr\u003e\nPotential application: Hormonal therapy and fertility enhancement in males.\u003c\/p\u003e\n\n\u003cp\u003eMood, Motivation, and Libido\u003c\/p\u003e\n\n\u003cp\u003eResearch links Kisspeptin to brain regions involved in reward-seeking behavior, motivation, and emotional regulation.\u003cbr\u003e\nA clinical study on 29 men found that KP-10 administration enhanced limbic brain activity, suggesting benefits in treating low libido and mood disorders.\u003cbr\u003e\nPotential application: Treatment for hypoactive sexual desire disorder (HSDD) and mood-related conditions.\u003c\/p\u003e\n\n\u003cp\u003eEnergy Balance and Metabolism\u003c\/p\u003e\n\n\u003cp\u003eKisspeptin neurons are highly sensitive to nutritional status, influencing GnRH release based on energy availability.\u003cbr\u003e\nStudies in genetically modified mice lacking Kiss1 receptors showed weight gain and reduced energy expenditure, indicating a role in obesity regulation.\u003cbr\u003e\nPotential application: Obesity management and metabolic health support.\u003c\/p\u003e\n\n\u003cp\u003eAnti-Cancer Research\u003c\/p\u003e\n\n\u003cp\u003eKisspeptin-10 has been linked to tumor suppression by reducing metastatic spread in various cancers.\u003cbr\u003e\nA groundbreaking study found that KP-10 suppressed melanoma metastasis by 95%.\u003cbr\u003e\nAltered kisspeptin levels have also been observed in breast, prostate, bladder, ovarian, and thyroid cancers, suggesting a role in tumor migration suppression.\u003cbr\u003e\nPotential application: Development of kisspeptin-based cancer therapies.\u003c\/p\u003e\n\n\u003cp\u003eMemory and Cognitive Function\u003c\/p\u003e\n\n\u003cp\u003eResearch suggests Kisspeptin analogs may enhance memory retention and cognitive flexibility.\u003cbr\u003e\nStudies in mice indicate that KP-10 counteracts learning impairments associated with alcohol exposure.\u003cbr\u003e\nPotential application: Neurological research and cognitive function support.\u003c\/p\u003e\n\n\u003cp\u003eKidney and Cardiovascular Health\u003c\/p\u003e\n\n\u003cp\u003eKisspeptin receptors have been identified in the kidneys, where they support glomerular development and function.\u003cbr\u003e\nKP-10 has been linked to blood vessel function and cardiac output regulation, making it a potential therapeutic target for cardiovascular diseases.\u003cbr\u003e\nPotential application: Cardiovascular health and kidney disease treatment.\u003c\/p\u003e\n\n\u003cp\u003eSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eAmino Acid Sequence: YNWNSFGLRF\u003cbr\u003e\nMolecular Formula: C₆₃H₈₃N₁₇O₁₄\u003cbr\u003e\nMolecular Weight: 1302.4 g\/mol\u003cbr\u003e\nCAS Registry Number: 388138-21-4\u003cbr\u003e\nSynonyms: KISS-1, Protein KISS-1, Metastin, KP-10 Peptide\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003c\/p\u003e\n\n\u003cp\u003eW. S. Dhillo et al. Kisspeptin-54 stimulates the hypothalamic-pituitary-gonadal axis in human males. J. Clin. Endocrinol. Metab. (2005).\u003cbr\u003e\nJ. T. George et al. Kisspeptin-10 is a potent stimulator of LH and increases pulse frequency in men. J. Clin. Endocrinol. Metab. (2011).\u003cbr\u003e\nC. J. L. Harter, G. S. Kavanagh, and J. T. Smith. The role of kisspeptin neurons in reproduction and metabolism. J. Endocrinol. (2018).\u003cbr\u003e\nE. J. Mead et al. Kisspeptins: a multifunctional peptide system with a role in reproduction, cancer, and the cardiovascular system. Br. J. Pharmacol. (2007).\u003cbr\u003e\nT. Ly, S. Harihar, and D. R. Welch. KISS1 in metastatic cancer research and treatment: potential and paradoxes. Cancer Metastasis Rev. (2020).\u003cbr\u003e\nP. Pazarci et al. The effects of daylight exposure on melatonin levels, Kiss1 expression, and melanoma formation in mice. Croat. Med. J. (2020).\u003cbr\u003e\nE. Gibula-Tarlowska and J. H. Kotlinska. Kissorphin improves spatial memory and cognitive flexibility impairment induced by ethanol treatment in the Barnes maze task in rats. Behav. Pharmacol. (2020).\u003cbr\u003e\nA. N. Comninos et al. Kisspeptin modulates sexual and emotional brain processing in humans. J. Clin. Invest. (2017).\u003cbr\u003e\nM. Bhattacharya and A. V. Babwah. Kisspeptin: Beyond the Brain. Endocrinology (2015).\u003cbr\u003e\nAntitumor efficacy of Kisspeptin in human malignant mesothelioma cells. PubMed Central (PMC).\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560266985,"sku":"PS052-1","price":19000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/kisspeptin-10-scaled.jpg?v=1778509462"},{"product_id":"cagrilintide-5mg-semaglutide-5mg-blend","title":"Cagrilintide (5mg) + Semaglutide (5mg) Blend","description":"Cagrilintide (5mg) + Semaglutide (5mg) Blend\nOverview\nThis dual-peptide weight management formulation combines Cagrilintide (an amylin analog) and Semaglutide (a GLP-1 receptor agonist) to enhance appetite suppression, fat loss, and metabolic control.\nKey Benefits:\n✔ Suppresses appetite and promotes satiety\n✔ Enhances fat oxidation and weight loss\n✔ Regulates blood sugar levels and insulin sensitivity\n✔ Supports cardiovascular and metabolic health\nMechanism of Action:\n\nCagrilintide: Mimics amylin, reducing hunger and food intake.\nSemaglutide: Activates GLP-1 receptors, slowing gastric emptying and improving insulin function.\n\nCitations:\n\nLau, J. et al. “Cagrilintide and its synergistic effects with GLP-1 receptor agonists,” Nat Metab, 2021.\nWilding, J. et al. “Semaglutide’s role in weight management,” N Engl J Med, 2021.","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560299753,"sku":"PS063","price":19000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/CAGRILINTIDE-SEMAGLUTIDE-10MG-scaled.jpg?v=1778509463"},{"product_id":"glow-8211-bpc-10mg-ghk-50mg-tb-10mg","title":"Wolverine Blend — BPC-157 (10mg) + TB-500 (10mg)","description":"BPC-157 (10mg) + GHK-Cu (50mg) + TB-500 (10mg) Blend\nGLOW\nOverview\nThis blend combines BPC-157, GHK-Cu, and TB-500, creating a powerful regenerative and healing formula for muscle, tendon, joint, and skin repair.\nKey Benefits:\n✔ Accelerates wound healing and tissue repair\n✔ Reduces inflammation and oxidative stress\n✔ Supports joint, muscle, and skin regeneration\n✔ Boosts collagen production and angiogenesis\nMechanism of Action:\n\nBPC-157: Heals the gut, tendons, and muscles via angiogenesis and cell migration.\nGHK-Cu: Stimulates collagen synthesis, tissue repair, and anti-aging effects.\nTB-500: Enhances cell migration and blood vessel formation, expediting injury recovery.\n\nCitations:\n\nSorg, H. et al. “GHK-Cu peptide and its wound healing effects,” J Dermatol Sci, 2017.\nNixon, J. et al. “BPC-157 and tendon healing,” J Orthop Res, 2016.\nGoldstein, A. et al. “Thymosin beta-4 and its role in regenerative medicine,” Regen Med, 2015.","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560332521,"sku":"PS062","price":25053.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/Wolverine_Blend_BPC-157_10mg_TB-500_10mg_01_jpg.jpg?v=1781622908"},{"product_id":"nad-500mg","title":"NAD+ 500mg","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nNAD+ (Nicotinamide Adenine Dinucleotide) is a vital coenzyme found in every living cell, essential for energy production, DNA repair, and cellular function. It fluctuates between oxidized (NAD⁺) and reduced (NADH) forms, supporting mitochondrial health and longevity.\u003cbr\u003e\nKey Benefits of NAD+ Research:\u003cbr\u003e\n✔️ Boosts mitochondrial energy production\u003cbr\u003e\n✔️ Supports brain health and cognitive function\u003cbr\u003e\n✔️ Promotes longevity and cellular repair\u003cbr\u003e\n✔️ Enhances cardiovascular and metabolic health\u003cbr\u003e\n✔️ Reduces inflammation and supports immune function\u003cbr\u003e\nNAD+ is widely studied for its role in aging, neuroprotection, and metabolic disorders.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\n1. Mitochondrial Health \u0026amp; Energy Production\u003c\/p\u003e\n\n\u003cp\u003eNAD+ is essential for ATP production in mitochondria via the electron transport chain.\u003cbr\u003e\nDeclining NAD+ levels contribute to mitochondrial dysfunction, leading to fatigue, aging, and metabolic decline.\u003cbr\u003e\nRestoring NAD+ improves energy production and reduces oxidative stress.\u003cbr\u003e\nPotential application: Anti-aging, energy optimization, and mitochondrial health support.\u003c\/p\u003e\n\n\u003cp\u003e2. Brain Health \u0026amp; Cognitive Function\u003c\/p\u003e\n\n\u003cp\u003eNAD+ activates sirtuins and PGC-1α, key regulators of brain cell repair and neuroprotection.\u003cbr\u003e\nIt protects neurons from oxidative stress and inflammation, helping prevent Alzheimer’s, Parkinson’s, and cognitive decline.\u003cbr\u003e\nResearch suggests NAD+ enhances memory, learning, and focus in aging populations.\u003cbr\u003e\nPotential application: Brain function, neuroprotection, and cognitive longevity.\u003c\/p\u003e\n\n\u003cp\u003e3. Longevity \u0026amp; Anti-Aging\u003c\/p\u003e\n\n\u003cp\u003eNAD+ activates sirtuins (SIRT1 \u0026amp; SIRT3), enzymes linked to lifespan extension.\u003cbr\u003e\nIt maintains genomic stability by repairing DNA damage, slowing the aging process.\u003cbr\u003e\nNAD+ precursors delay age-related decline in muscle strength and vascular health.\u003cbr\u003e\nPotential application: Longevity, anti-aging, and cellular rejuvenation.\u003c\/p\u003e\n\n\u003cp\u003e4. Cardiovascular \u0026amp; Metabolic Health\u003c\/p\u003e\n\n\u003cp\u003eNAD+ supports blood vessel flexibility and prevents atherosclerosis.\u003cbr\u003e\nIt enhances insulin sensitivity, reducing the risk of Type 2 diabetes.\u003cbr\u003e\nHigher NAD+ levels protect against obesity and metabolic syndrome in animal studies.\u003cbr\u003e\nPotential application: Heart health, diabetes prevention, and metabolic balance.\u003c\/p\u003e\n\n\u003cp\u003e5. Inflammation \u0026amp; Immune Support\u003c\/p\u003e\n\n\u003cp\u003eNAD+ regulates inflammatory pathways, reducing chronic inflammation linked to metabolic disorders and autoimmune diseases.\u003cbr\u003e\nIt enhances immune function, helping fight infections and age-related immune decline.\u003cbr\u003e\nPotential application: Immune support and inflammation reduction.\u003c\/p\u003e\n\n\u003cp\u003e6. Addiction Recovery \u0026amp; Mood Regulation\u003c\/p\u003e\n\n\u003cp\u003eNAD+ levels are depleted in cases of chronic drug and alcohol use.\u003cbr\u003e\nSupplementation has been shown to reduce withdrawal symptoms and cravings.\u003cbr\u003e\nStudies suggest NAD+ supports mood stability and resilience against stress.\u003cbr\u003e\nPotential application: Addiction recovery and mental wellness.\u003c\/p\u003e\n\n\u003cp\u003eSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eMolecular Formula: C₂₁H₂₇N₇O₁₄P₂\u003cbr\u003e\nMolecular Weight: 663.43 g\/mol\u003cbr\u003e\nCAS Number: 53-84-9\u003cbr\u003e\nSynonyms: Nicotinamide Adenine Dinucleotide, β-NAD, Endopride\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003c\/p\u003e\n\n\u003cp\u003eA. P. Gomes et al. Declining NAD+ disrupts nuclear-mitochondrial communication during aging. Cell (2013).\u003cbr\u003e\nS. Imai \u0026amp; L. Guarente. NAD+ and sirtuins in aging and disease. Trends Cell Biol. (2014).\u003cbr\u003e\nN. Sun, R. J. Youle \u0026amp; T. Finkel. The mitochondrial basis of aging. Mol. Cell (2016).\u003cbr\u003e\nR. T. Matthews et al. Coenzyme Q10 and NAD+ effects on brain mitochondrial function. PNAS (1998).\u003cbr\u003e\nC. Shan et al. Protective effects of NAD+ in Parkinson’s disease models. Prog. Neuropsychopharmacol. Biol. Psychiatry (2019).\u003cbr\u003e\nD. C. Maddison \u0026amp; F. Giorgini. The kynurenine pathway and neurodegeneration. Semin. Cell Dev. Biol. (2015).\u003cbr\u003e\nA. Garten et al. Roles of NAMPT and NAD+ metabolism in health and disease. Nat. Rev. Endocrinol. (2015).\u003cbr\u003e\nS. Yamaguchi \u0026amp; J. Yoshino. Adipose tissue NAD+ in obesity and insulin resistance. BioEssays (2017).\u003cbr\u003e\nD. Stipp. Beyond Resveratrol: The Anti-Aging NAD+ Fad. Scientific American (2019).\u003cbr\u003e\nS. Imai. NAD World 2.0: Systemic Regulation of Aging and Longevity. npj Systems Biology and Applications(2016).\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560365289,"sku":"PS061","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/NAD-500mg-scaled.jpg?v=1778509465"},{"product_id":"reconstitution-solution","title":"Reconstitution Solution","description":"Bacteriostatic Water (Bac Water)\nOverview\nBacteriostatic Water is sterile water containing 0.9% benzyl alcohol, used for diluting peptides and medications before injection.\nKey Benefits:\n✔ Prevents bacterial growth in multi-use vials\n✔ Prolongs shelf-life of reconstituted peptides\n✔ Safe for subcutaneous and intramuscular injections\nCitations:\n\nU.S. Pharmacopeia, “Bacteriostatic Water for Injection,” USP 43-NF38, 2020.","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560398057,"sku":"PS060","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/RECONSTITION-SOLUTION-3ML.png?v=1778509470"},{"product_id":"glutathione-gsh-1500mg","title":"Glutathione (GSH) 1500mg","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nL-Glutathione (GSH) is a powerful antioxidant and detoxifier that protects cells from oxidative stress, supports immune health, and plays a key role in anti-aging. It is a tripeptide composed of cysteine, glutamate, and glycine, essential for maintaining cellular redox balance and detoxification.\u003cbr\u003e\nKey Benefits of L-Glutathione Research:\u003cbr\u003e\n✔️ Supports brain health and reduces neurodegenerative risk\u003cbr\u003e\n✔️ Protects cartilage and joints from oxidative damage\u003cbr\u003e\n✔️ Enhances immune system function\u003cbr\u003e\n✔️ Improves skin health and brightens complexion\u003cbr\u003e\n✔️ Aids liver detoxification and heavy metal removal\u003cbr\u003e\nGSH is widely studied in longevity research, immune function, and chronic disease prevention.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\n1. Neuroprotection \u0026amp; Brain Health\u003c\/p\u003e\n\n\u003cp\u003eL-Glutathione shields neurons from oxidative stress, reducing the risk of neurodegenerative diseases like Alzheimer’s and Parkinson’s.\u003cbr\u003e\nLow GSH levels correlate with cognitive decline and increased oxidative brain damage.\u003cbr\u003e\nPotential application: Brain health support, cognitive enhancement, and neuroprotection.\u003c\/p\u003e\n\n\u003cp\u003e2. Cartilage \u0026amp; Joint Health\u003c\/p\u003e\n\n\u003cp\u003eGSH plays a role in cartilage regeneration and protects against osteoporosis and osteoarthritis.\u003cbr\u003e\nIt regulates inflammation and oxidative stress, preventing joint degeneration.\u003cbr\u003e\nPotential application: Joint health, arthritis prevention, and cartilage repair.\u003c\/p\u003e\n\n\u003cp\u003e3. Eye Health \u0026amp; Vision Protection\u003c\/p\u003e\n\n\u003cp\u003eGlutathione is present in high concentrations in the eye, particularly in the retina and lens.\u003cbr\u003e\nIt prevents cataracts and age-related macular degeneration (AMD) by neutralizing free radicals and UV damage.\u003cbr\u003e\nPotential application: Vision preservation and eye disease prevention.\u003c\/p\u003e\n\n\u003cp\u003e4. Immune System Enhancement\u003c\/p\u003e\n\n\u003cp\u003eGSH is critical for T-cell function and white blood cell activation, boosting immune defense.\u003cbr\u003e\nIt has been studied for its role in improving immune response in HIV and viral infections.\u003cbr\u003e\nPotential application: Immune support and infection prevention.\u003c\/p\u003e\n\n\u003cp\u003e5. Skin Health \u0026amp; Anti-Aging\u003c\/p\u003e\n\n\u003cp\u003eGlutathione reduces oxidative stress, promoting youthful skin, improved elasticity, and reduced wrinkles.\u003cbr\u003e\n**It lightens pigmentation by inhibiting melanin production, resulting in a brighter, more even complexion.\u003cbr\u003e\nPotential application: Skin rejuvenation, anti-aging, and hyperpigmentation reduction.\u003c\/p\u003e\n\n\u003cp\u003e6. Liver Detoxification \u0026amp; Heavy Metal Removal\u003c\/p\u003e\n\n\u003cp\u003eL-Glutathione is essential for detoxifying harmful toxins, including heavy metals, alcohol, and environmental pollutants.\u003cbr\u003e\nIt enhances liver function and prevents oxidative liver damage.\u003cbr\u003e\nPotential application: Liver detoxification, alcohol recovery, and toxin elimination.\u003c\/p\u003e\n\n\u003cp\u003e7. Oxidative Stress \u0026amp; Inflammation Control\u003c\/p\u003e\n\n\u003cp\u003eGSH neutralizes harmful free radicals that contribute to chronic inflammation, aging, and disease progression.\u003cbr\u003e\nLow GSH levels are associated with diabetes, cardiovascular disease, and metabolic disorders.\u003cbr\u003e\nPotential application: Inflammation reduction and metabolic health support.\u003c\/p\u003e\n\n\u003cp\u003eSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eAmino Acid Sequence: γ-L-Glutamyl-L-cysteinyl-glycine\u003cbr\u003e\nMolecular Formula: C₁₀H₁₇N₃O₆S\u003cbr\u003e\nMolecular Weight: 307.33 g\/mol\u003cbr\u003e\nCAS Registry Number: 170-18-8\u003cbr\u003e\nSynonyms: GSH, Glutathione, Isethion\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003c\/p\u003e\n\n\u003cp\u003eH. J. Forman et al. Glutathione: overview of its protective roles, measurement, and biosynthesis. Mol. Aspects Med. (2009).\u003cbr\u003e\nG. Teskey et al. Glutathione as a Marker for Human Disease. Adv. Clin. Chem. (2018).\u003cbr\u003e\nS. Tsunoda et al. Protein folding and glutathione depletion in the endoplasmic reticulum. eLife (2014).\u003cbr\u003e\nJ. H. Wu \u0026amp; G. Batist. Glutathione and therapeutic potentials. Biochim. Biophys. Acta (2013).\u003cbr\u003e\nT. S. Nagapan et al. L-Glutathione prevents UVB-induced melanogenesis and oxidative stress. Exp. Anim. (2019).\u003cbr\u003e\nA. Bansal \u0026amp; M. C. Simon. Glutathione metabolism in cancer progression. J. Cell Biol. (2018).\u003cbr\u003e\nP. Maher. The effects of stress and aging on glutathione metabolism. Ageing Res. Rev. (2005).\u003cbr\u003e\nV. N. Reddy. Glutathione and its function in the lens. Exp. Eye Res. (1990).\u003cbr\u003e\nS. Weschawalit et al. Glutathione’s anti-aging and antimelanogenic effects. Clin. Cosmet. Investig. Dermatol. (2017).\u003cbr\u003e\nW. Dröge \u0026amp; R. Breitkreutz. Glutathione and immune function. Proc. Nutr. Soc. (2000).\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560430825,"sku":"PS059","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/L-GLUTATHION-GSH-1500MG-scaled.jpg?v=1778509466"},{"product_id":"melatonin-10mg","title":"Melatonin 10mg","description":"OVERVIEW\nMelatonin is a naturally occurring indoleamine primarily produced by the pineal gland, playing a crucial role in regulating circadian rhythms, sleep-wake cycles, and neuroendocrine functions. It is widely studied for its potential impact on sleep regulation, oxidative stress management, and immune system modulation. Research suggests that melatonin may:\n\nRegulate sleep patterns by influencing the body’s internal clock\nAct as a potent antioxidant, reducing oxidative damage at the cellular level\nSupport immune function and inflammatory response regulation\nInfluence metabolic and neuroprotective processes\nPlay a role in reproductive hormone modulation\n\nMelatonin functions by binding to melatonin receptors (MT1 and MT2), which are distributed in the brain and peripheral tissues, helping synchronize biological processes to environmental light-dark cycles.\nRESEARCH\nMelatonin and Sleep Regulation\nMelatonin is most well-known for its role in controlling the circadian rhythm and sleep-wake cycles. Research findings indicate:\n\nIt signals the body when it is time to sleep, aiding in sleep onset and quality.\nMT1 and MT2 receptor activation influences the sleep phase and duration.\nStudies suggest that melatonin may improve sleep patterns in circadian rhythm disorders.\n\nMelatonin as an Antioxidant\nMelatonin is a powerful antioxidant with the ability to neutralize free radicals and protect cells from oxidative damage. Key research findings include:\n\nIt enhances the activity of other antioxidants, such as glutathione and superoxide dismutase (SOD).\nMelatonin can cross the blood-brain barrier, offering neuroprotective benefits.\nStudies suggest its role in reducing age-related oxidative stress and cellular damage.\n\nMelatonin and Immune Function\nMelatonin has been studied for its immunomodulatory properties, with research suggesting:\n\nIt enhances immune cell function and supports the body’s defense mechanisms.\nMelatonin may play a role in regulating inflammatory cytokine production.\nPotential benefits in reducing inflammation-associated conditions have been explored.\n\nMelatonin and Metabolism\nMelatonin has been implicated in metabolic regulation, with research indicating:\n\nIt may influence insulin sensitivity and glucose homeostasis.\nStudies have explored its role in obesity and metabolic disorders.\nIts effects on energy expenditure and fat oxidation are areas of active research.\n\nMelatonin and Neuroprotection\nMelatonin has been investigated for its potential role in cognitive health and neuroprotection. Research highlights:\n\nIt may help protect neurons from oxidative stress and mitochondrial dysfunction.\nStudies suggest a potential role in neurodegenerative disease models.\nMelatonin’s anti-inflammatory properties may contribute to brain health and cognitive function.\n\nSTRUCTURE\n\nMolecular Formula: C₁₃H₁₆N₂O₂\nMolecular Weight: 232.2783 g\/mol\nCAS Registry Number: 73-31-4\nPurity: 99% HPLC\n\nRESEARCH USE ONLY\nMelatonin is strictly intended for research and laboratory purposes. It is not for human or veterinary use and is not intended for therapeutic or diagnostic applications.","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560463593,"sku":"PS058","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/Melatonin-10mg-scaled.jpg?v=1778509468"},{"product_id":"mots-c-40mg","title":"MOTS-C 40mg","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nMOTS-c is a mitochondria-derived peptide (MDP) composed of 16 amino acids that plays a critical role in metabolic regulation, mitochondrial function, and cellular stress response. Unlike traditional peptides, MOTS-c is synthesized within mitochondria and translocates to the nucleus, where it regulates gene expression related to energy metabolism and longevity.\u003cbr\u003e\nKey Benefits of MOTS-c Research:\u003cbr\u003e\n✔️ Enhances muscle metabolism and exercise capacity\u003cbr\u003e\n✔️ Promotes fat oxidation and prevents obesity\u003cbr\u003e\n✔️ Improves insulin sensitivity and helps prevent Type 2 diabetes\u003cbr\u003e\n✔️ Protects against age-related diseases and enhances longevity\u003cbr\u003e\n✔️ Supports cardiovascular and bone health\u003cbr\u003e\nMOTS-c is being actively studied in metabolic disorders, aging research, and mitochondrial medicine.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\n1. Muscle Metabolism \u0026amp; Exercise Performance\u003c\/p\u003e\n\n\u003cp\u003eMOTS-c enhances AMPK activation, improving glucose uptake in muscle tissue.\u003cbr\u003e\nStudies in mice show that MOTS-c increases endurance and muscle recovery post-exercise.\u003cbr\u003e\nPotential application: Exercise enhancement, muscle recovery, and metabolic health support.\u003c\/p\u003e\n\n\u003cp\u003e2. Fat Metabolism \u0026amp; Obesity Prevention\u003c\/p\u003e\n\n\u003cp\u003eMOTS-c stimulates brown fat activation, promoting fat oxidation over storage.\u003cbr\u003e\nResearch indicates that MOTS-c supplementation prevents adipose tissue dysfunction and white fat accumulation.\u003cbr\u003e\nPotential application: Obesity treatment, fat metabolism regulation, and ketogenic diet support.\u003c\/p\u003e\n\n\u003cp\u003e3. Insulin Sensitivity \u0026amp; Diabetes Prevention\u003c\/p\u003e\n\n\u003cp\u003eLow MOTS-c levels are correlated with insulin resistance, particularly in obese individuals.\u003cbr\u003e\nMOTS-c supplementation enhances glucose metabolism and insulin sensitivity, making it a potential therapy for Type 2 diabetes.\u003cbr\u003e\nPotential application: Diabetes prevention, metabolic syndrome treatment, and insulin resistance improvement.\u003c\/p\u003e\n\n\u003cp\u003e4. Longevity \u0026amp; Aging Research\u003c\/p\u003e\n\n\u003cp\u003eMOTS-c levels decline with age, and genetic studies link its variants to exceptional longevity (especially in Japanese centenarians).\u003cbr\u003e\nMOTS-c supplementation has been associated with improved mitochondrial function and protection against age-related diseases.\u003cbr\u003e\nPotential application: Longevity enhancement, anti-aging therapy, and mitochondrial function support.\u003c\/p\u003e\n\n\u003cp\u003e5. Cardiovascular Health\u003c\/p\u003e\n\n\u003cp\u003eMOTS-c improves endothelial function, reducing inflammation and oxidative stress in blood vessels.\u003cbr\u003e\nClinical research shows lower MOTS-c levels correlate with a higher risk of coronary artery disease.\u003cbr\u003e\nPotential application: Cardiovascular disease prevention, blood vessel protection, and heart health improvement.\u003c\/p\u003e\n\n\u003cp\u003e6. Bone Health \u0026amp; Osteoporosis Prevention\u003c\/p\u003e\n\n\u003cp\u003eMOTS-c activates the TGF-β\/SMAD signaling pathway, stimulating osteoblast activity and Type I collagen production.\u003cbr\u003e\nStudies suggest that MOTS-c enhances bone density and reduces fracture risk.\u003cbr\u003e\nPotential application: Osteoporosis prevention and bone regeneration therapy.\u003c\/p\u003e\n\n\u003cp\u003e7. Metabolic Stress \u0026amp; Mitochondrial Function\u003c\/p\u003e\n\n\u003cp\u003eMOTS-c acts as a mitochondrial stress response peptide, regulating nuclear gene expression to counteract metabolic dysfunction.\u003cbr\u003e\nIt influences glucose metabolism and antioxidant defense, offering protection against metabolic diseases.\u003cbr\u003e\nPotential application: Mitochondrial medicine, metabolic disease treatment, and cellular stress response regulation.\u003c\/p\u003e\n\n\u003cp\u003eSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eAmino Acid Sequence: Met-Arg-Trp-Gln-Glu-Met-Gly-Tyr-Ile-Phe-Tyr-Pro-Arg-Lys-Leu-Arg\u003cbr\u003e\nMolecular Formula: C₁₀₁H₁₅₂N₂₈O₂₂S₂\u003cbr\u003e\nMolecular Weight: 2174.64 g\/mol\u003cbr\u003e\nCAS Registry Number: 1627580-64-6\u003cbr\u003e\nSynonyms: Mitochondrial Open Reading Frame of the 12S rRNA-c (MT-RNR1), MOTS-c Peptide\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003c\/p\u003e\n\n\u003cp\u003eC. Lee, K. H. Kim, \u0026amp; P. Cohen. MOTS-c: A novel mitochondrial-derived peptide regulating muscle and fat metabolism. Free Radic. Biol. Med. (2016).\u003cbr\u003e\nH. Lu et al. MOTS-c peptide regulates adipose homeostasis to prevent ovariectomy-induced metabolic dysfunction. J. Mol. Med. Berl. Ger. (2019).\u003cbr\u003e\nK. H. Kim et al. MOTS-c translocates to the nucleus to regulate nuclear gene expression in response to metabolic stress. Cell Metab. (2018).\u003cbr\u003e\nS.-J. Kim et al. MOTS-c enhances insulin sensitivity and regulates plasma metabolites. Physiol. Rep. (2019).\u003cbr\u003e\nL. R. Cataldo et al. Plasma MOTS-c levels are associated with insulin sensitivity in lean but not obese individuals. J. Investig. Med. (2018).\u003cbr\u003e\nN. Che et al. MOTS-c improves osteoporosis by promoting Type I collagen synthesis in osteoblasts via TGF-β\/SMAD signaling pathway. Eur. Rev. Med. Pharmacol. Sci. (2019).\u003cbr\u003e\nN. Fuku et al. MOTS-c as a key player in exceptional longevity. Aging Cell (2015).\u003cbr\u003e\nQ. Qin et al. Downregulation of circulating MOTS-c levels in patients with coronary endothelial dysfunction. Int. J. Cardiol. (2018).\u003cbr\u003e\nY. Yang et al. MOTS-c’s role in cardiovascular disease: Recent updates. Biomed. Pharmacother. (2019).\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560496361,"sku":"PS057","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/mots-c-40mg-scaled.jpg?v=1778509468"},{"product_id":"mots-c-10mg","title":"MOTS-C 10mg","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nMOTS-c is a mitochondria-derived peptide (MDP) composed of 16 amino acids that plays a critical role in metabolic regulation, mitochondrial function, and cellular stress response. Unlike traditional peptides, MOTS-c is synthesized within mitochondria and translocates to the nucleus, where it regulates gene expression related to energy metabolism and longevity.\u003cbr\u003e\nKey Benefits of MOTS-c Research:\u003cbr\u003e\n✔️ Enhances muscle metabolism and exercise capacity\u003cbr\u003e\n✔️ Promotes fat oxidation and prevents obesity\u003cbr\u003e\n✔️ Improves insulin sensitivity and helps prevent Type 2 diabetes\u003cbr\u003e\n✔️ Protects against age-related diseases and enhances longevity\u003cbr\u003e\n✔️ Supports cardiovascular and bone health\u003cbr\u003e\nMOTS-c is being actively studied in metabolic disorders, aging research, and mitochondrial medicine.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\n1. Muscle Metabolism \u0026amp; Exercise Performance\u003c\/p\u003e\n\n\u003cp\u003eMOTS-c enhances AMPK activation, improving glucose uptake in muscle tissue.\u003cbr\u003e\nStudies in mice show that MOTS-c increases endurance and muscle recovery post-exercise.\u003cbr\u003e\nPotential application: Exercise enhancement, muscle recovery, and metabolic health support.\u003c\/p\u003e\n\n\u003cp\u003e2. Fat Metabolism \u0026amp; Obesity Prevention\u003c\/p\u003e\n\n\u003cp\u003eMOTS-c stimulates brown fat activation, promoting fat oxidation over storage.\u003cbr\u003e\nResearch indicates that MOTS-c supplementation prevents adipose tissue dysfunction and white fat accumulation.\u003cbr\u003e\nPotential application: Obesity treatment, fat metabolism regulation, and ketogenic diet support.\u003c\/p\u003e\n\n\u003cp\u003e3. Insulin Sensitivity \u0026amp; Diabetes Prevention\u003c\/p\u003e\n\n\u003cp\u003eLow MOTS-c levels are correlated with insulin resistance, particularly in obese individuals.\u003cbr\u003e\nMOTS-c supplementation enhances glucose metabolism and insulin sensitivity, making it a potential therapy for Type 2 diabetes.\u003cbr\u003e\nPotential application: Diabetes prevention, metabolic syndrome treatment, and insulin resistance improvement.\u003c\/p\u003e\n\n\u003cp\u003e4. Longevity \u0026amp; Aging Research\u003c\/p\u003e\n\n\u003cp\u003eMOTS-c levels decline with age, and genetic studies link its variants to exceptional longevity (especially in Japanese centenarians).\u003cbr\u003e\nMOTS-c supplementation has been associated with improved mitochondrial function and protection against age-related diseases.\u003cbr\u003e\nPotential application: Longevity enhancement, anti-aging therapy, and mitochondrial function support.\u003c\/p\u003e\n\n\u003cp\u003e5. Cardiovascular Health\u003c\/p\u003e\n\n\u003cp\u003eMOTS-c improves endothelial function, reducing inflammation and oxidative stress in blood vessels.\u003cbr\u003e\nClinical research shows lower MOTS-c levels correlate with a higher risk of coronary artery disease.\u003cbr\u003e\nPotential application: Cardiovascular disease prevention, blood vessel protection, and heart health improvement.\u003c\/p\u003e\n\n\u003cp\u003e6. Bone Health \u0026amp; Osteoporosis Prevention\u003c\/p\u003e\n\n\u003cp\u003eMOTS-c activates the TGF-β\/SMAD signaling pathway, stimulating osteoblast activity and Type I collagen production.\u003cbr\u003e\nStudies suggest that MOTS-c enhances bone density and reduces fracture risk.\u003cbr\u003e\nPotential application: Osteoporosis prevention and bone regeneration therapy.\u003c\/p\u003e\n\n\u003cp\u003e7. Metabolic Stress \u0026amp; Mitochondrial Function\u003c\/p\u003e\n\n\u003cp\u003eMOTS-c acts as a mitochondrial stress response peptide, regulating nuclear gene expression to counteract metabolic dysfunction.\u003cbr\u003e\nIt influences glucose metabolism and antioxidant defense, offering protection against metabolic diseases.\u003cbr\u003e\nPotential application: Mitochondrial medicine, metabolic disease treatment, and cellular stress response regulation.\u003c\/p\u003e\n\n\u003cp\u003eSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eAmino Acid Sequence: Met-Arg-Trp-Gln-Glu-Met-Gly-Tyr-Ile-Phe-Tyr-Pro-Arg-Lys-Leu-Arg\u003cbr\u003e\nMolecular Formula: C₁₀₁H₁₅₂N₂₈O₂₂S₂\u003cbr\u003e\nMolecular Weight: 2174.64 g\/mol\u003cbr\u003e\nCAS Registry Number: 1627580-64-6\u003cbr\u003e\nSynonyms: Mitochondrial Open Reading Frame of the 12S rRNA-c (MT-RNR1), MOTS-c Peptide\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003c\/p\u003e\n\n\u003cp\u003eC. Lee, K. H. Kim, \u0026amp; P. Cohen. MOTS-c: A novel mitochondrial-derived peptide regulating muscle and fat metabolism. Free Radic. Biol. Med. (2016).\u003cbr\u003e\nH. Lu et al. MOTS-c peptide regulates adipose homeostasis to prevent ovariectomy-induced metabolic dysfunction. J. Mol. Med. Berl. Ger. (2019).\u003cbr\u003e\nK. H. Kim et al. MOTS-c translocates to the nucleus to regulate nuclear gene expression in response to metabolic stress. Cell Metab. (2018).\u003cbr\u003e\nS.-J. Kim et al. MOTS-c enhances insulin sensitivity and regulates plasma metabolites. Physiol. Rep. (2019).\u003cbr\u003e\nL. R. Cataldo et al. Plasma MOTS-c levels are associated with insulin sensitivity in lean but not obese individuals. J. Investig. Med. (2018).\u003cbr\u003e\nN. Che et al. MOTS-c improves osteoporosis by promoting Type I collagen synthesis in osteoblasts via TGF-β\/SMAD signaling pathway. Eur. Rev. Med. Pharmacol. Sci. (2019).\u003cbr\u003e\nN. Fuku et al. MOTS-c as a key player in exceptional longevity. Aging Cell (2015).\u003cbr\u003e\nQ. Qin et al. Downregulation of circulating MOTS-c levels in patients with coronary endothelial dysfunction. Int. J. Cardiol. (2018).\u003cbr\u003e\nY. Yang et al. MOTS-c’s role in cardiovascular disease: Recent updates. Biomed. Pharmacother. (2019).\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560529129,"sku":"PS056","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/mots-c-10mg-scaled.jpg?v=1778509469"},{"product_id":"thymosin-alpha-1-5mg","title":"Thymosin Alpha-1 5mg","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nThymosin Alpha-1 (Tα1) is a naturally occurring peptide derived from the thymus gland, discovered in 1972. It plays a critical role in immune system regulation, inflammation control, and disease resistance.\u003cbr\u003e\nResearch has identified Thymosin Alpha-1’s potential benefits in:\u003c\/p\u003e\n\n\u003cp\u003eEnhancing immune response and infection resistance\u003cbr\u003e\nTreating chronic viral infections, including Hepatitis B \u0026amp; C\u003cbr\u003e\nRegulating autoimmune disorders and reducing inflammation\u003cbr\u003e\nImproving vaccine efficacy in immunocompromised individuals\u003cbr\u003e\nSupporting cancer therapy by boosting immune surveillance\u003c\/p\u003e\n\n\u003cp\u003eDue to its immunomodulatory properties, Thymosin Alpha-1 is widely studied for applications in immunotherapy, infectious disease management, and oncology.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\nImmune Modulation \u0026amp; Infection Control\u003c\/p\u003e\n\n\u003cp\u003eTα1 enhances T-cell maturation and activation, strengthening the adaptive immune response.\u003cbr\u003e\nStimulates dendritic cells, promoting better antigen presentation and immune memory.\u003cbr\u003e\nResearch shows that Tα1 can restore immune function even in individuals lacking a functional thymus, preventing recurrent infections.\u003cbr\u003e\nPotential application: Immunotherapy, infection prevention, and immune recovery support.\u003c\/p\u003e\n\n\u003cp\u003eHepatitis B \u0026amp; C Treatment\u003c\/p\u003e\n\n\u003cp\u003eTα1 is approved in 35+ countries as a treatment for chronic Hepatitis B \u0026amp; C.\u003cbr\u003e\nIt reduces viral replication, enhances immune response, and improves patient recovery rates.\u003cbr\u003e\nClinical studies suggest that when combined with interferon-alpha, Tα1 enhances viral clearance and treatment efficacy.\u003cbr\u003e\nPotential application: Chronic viral infection management, including Hepatitis B, Hepatitis C, and emerging viral threats.\u003c\/p\u003e\n\n\u003cp\u003eCancer Therapy \u0026amp; Tumor Immunity\u003c\/p\u003e\n\n\u003cp\u003eTα1 is under investigation as an adjuvant therapy for melanoma, lung cancer, and liver cancer.\u003cbr\u003e\nBoosts T-cell function and tumor surveillance, improving immune recovery post-chemotherapy.\u003cbr\u003e\nClinical trials suggest improved survival rates and reduced tumor progression with Tα1 therapy.\u003cbr\u003e\nPotential application: Cancer immunotherapy and adjunct treatment for chemotherapy-induced immune suppression.\u003c\/p\u003e\n\n\u003cp\u003eSepsis \u0026amp; Septic Shock\u003c\/p\u003e\n\n\u003cp\u003eSepsis is a life-threatening immune overreaction to infection, leading to organ failure and high mortality rates.\u003cbr\u003e\nTα1 has been shown to modulate immune response, prevent excessive inflammation, and reduce sepsis-related mortality.\u003cbr\u003e\nEarly studies suggest Tα1 could be an adjunct therapy for severe infections and immune dysregulation disorders.\u003cbr\u003e\nPotential application: Sepsis treatment and critical care medicine.\u003c\/p\u003e\n\n\u003cp\u003eVaccine Response Enhancement\u003c\/p\u003e\n\n\u003cp\u003eTα1 increases vaccine efficacy, particularly in immunocompromised individuals.\u003cbr\u003e\nStudies suggest Tα1 enhances antibody production and immune memory, improving response to vaccines such as avian influenza and HIV vaccines.\u003cbr\u003e\nPotential application: Improving vaccine effectiveness in at-risk populations, including the elderly and immunocompromised patients.\u003c\/p\u003e\n\n\u003cp\u003eNeuroprotection \u0026amp; Cognitive Function\u003c\/p\u003e\n\n\u003cp\u003eRecent research suggests Tα1 may promote nerve growth and enhance cognitive function.\u003cbr\u003e\nAnimal studies show improved learning, memory, and neuroplasticity following Tα1 administration.\u003cbr\u003e\nPotential application: Neurodegenerative disease research, cognitive enhancement, and brain health support.\u003c\/p\u003e\n\n\u003cp\u003eAutoimmune Disease Management\u003c\/p\u003e\n\n\u003cp\u003eTα1 helps regulate immune overactivity, making it a promising therapy for rheumatoid arthritis, multiple sclerosis, and lupus.\u003cbr\u003e\nIt reduces excessive inflammation while preserving normal immune function, which is crucial in autoimmune disease treatment.\u003cbr\u003e\nPotential application: Autoimmune disorder management and inflammation control.\u003c\/p\u003e\n\n\u003cp\u003eSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eAmino Acid Sequence: Ser-Asp-Ala-Ala-Val-Asp-Thr-Ser-Ser-Glu-Ile-Thr-Thr-Lys-Asp-Leu-Lys-Glu-Lys-Lys-Glu-Val-Val-Glu-Glu-Ala-Glu-Asn\u003cbr\u003e\nMolecular Formula: C₁₂₉H₂₁₅N₃₃O₅₅\u003cbr\u003e\nMolecular Weight: 3108.315 g\/mol\u003cbr\u003e\nCAS Registry Number: 62304-98-7\u003cbr\u003e\nSynonyms: Thymalfasin\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003c\/p\u003e\n\n\u003cp\u003eR. King \u0026amp; C. Tuthill Immune Modulation with Thymosin Alpha-1 Treatment. Vitam. Horm. (2016).\u003cbr\u003e\nC. Zhang et al. Tα1 as an immune adjuvant in avian influenza vaccination. Microb. Pathog. (2018).\u003cbr\u003e\nF. Pei et al. Thymosin Alpha-1 treatment for sepsis patients. Expert Opin. Biol. Ther. (2018).\u003cbr\u003e\nG. Wang et al. Tα1 promotes neurogenesis and cognitive function in developing mice. Neurosci. Bull. (2017).\u003cbr\u003e\nL. Romani et al. Tα1 activates dendritic cells for antifungal resistance via Toll-like receptor signaling. Blood (2004).\u003cbr\u003e\nA. L. Goldstein \u0026amp; A. L. Goldstein. Emerging clinical applications of Thymosin Alpha-1. Expert Opin. Biol. Ther. (2009).\u003cbr\u003e\nM. Maio et al. Tα1 in combination with chemotherapy improves survival in metastatic melanoma. J. Clin. Oncol. (2010).\u003cbr\u003e\nR. Danielli et al. Clinical applications of Thymosin Alpha-1 in melanoma treatment. Ann. N. Y. Acad. Sci. (2012).\u003cbr\u003e\nX. Shen et al. Development of a long-acting Thymosin Alpha-1 fusion protein for breast cancer treatment.Biomed. Pharmacother. (2018).\u003cbr\u003e\nF. Wang et al. Tα1-Fc enhances immune function and reduces melanoma and breast cancer progression. Sci. Rep. (2018).\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560561897,"sku":"PS055","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/Thymosin-Alpha-5mg-scaled.jpg?v=1778509470"},{"product_id":"thymosin-alpha-1-10mg","title":"Thymosin Alpha-1 10mg","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nThymosin Alpha-1 (Tα1) is a naturally occurring peptide derived from the thymus gland, discovered in 1972. It plays a critical role in immune system regulation, inflammation control, and disease resistance.\u003cbr\u003e\nResearch has identified Thymosin Alpha-1’s potential benefits in:\u003c\/p\u003e\n\n\u003cp\u003eEnhancing immune response and infection resistance\u003cbr\u003e\nTreating chronic viral infections, including Hepatitis B \u0026amp; C\u003cbr\u003e\nRegulating autoimmune disorders and reducing inflammation\u003cbr\u003e\nImproving vaccine efficacy in immunocompromised individuals\u003cbr\u003e\nSupporting cancer therapy by boosting immune surveillance\u003c\/p\u003e\n\n\u003cp\u003eDue to its immunomodulatory properties, Thymosin Alpha-1 is widely studied for applications in immunotherapy, infectious disease management, and oncology.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\nImmune Modulation \u0026amp; Infection Control\u003c\/p\u003e\n\n\u003cp\u003eTα1 enhances T-cell maturation and activation, strengthening the adaptive immune response.\u003cbr\u003e\nStimulates dendritic cells, promoting better antigen presentation and immune memory.\u003cbr\u003e\nResearch shows that Tα1 can restore immune function even in individuals lacking a functional thymus, preventing recurrent infections.\u003cbr\u003e\nPotential application: Immunotherapy, infection prevention, and immune recovery support.\u003c\/p\u003e\n\n\u003cp\u003eHepatitis B \u0026amp; C Treatment\u003c\/p\u003e\n\n\u003cp\u003eTα1 is approved in 35+ countries as a treatment for chronic Hepatitis B \u0026amp; C.\u003cbr\u003e\nIt reduces viral replication, enhances immune response, and improves patient recovery rates.\u003cbr\u003e\nClinical studies suggest that when combined with interferon-alpha, Tα1 enhances viral clearance and treatment efficacy.\u003cbr\u003e\nPotential application: Chronic viral infection management, including Hepatitis B, Hepatitis C, and emerging viral threats.\u003c\/p\u003e\n\n\u003cp\u003eCancer Therapy \u0026amp; Tumor Immunity\u003c\/p\u003e\n\n\u003cp\u003eTα1 is under investigation as an adjuvant therapy for melanoma, lung cancer, and liver cancer.\u003cbr\u003e\nBoosts T-cell function and tumor surveillance, improving immune recovery post-chemotherapy.\u003cbr\u003e\nClinical trials suggest improved survival rates and reduced tumor progression with Tα1 therapy.\u003cbr\u003e\nPotential application: Cancer immunotherapy and adjunct treatment for chemotherapy-induced immune suppression.\u003c\/p\u003e\n\n\u003cp\u003eSepsis \u0026amp; Septic Shock\u003c\/p\u003e\n\n\u003cp\u003eSepsis is a life-threatening immune overreaction to infection, leading to organ failure and high mortality rates.\u003cbr\u003e\nTα1 has been shown to modulate immune response, prevent excessive inflammation, and reduce sepsis-related mortality.\u003cbr\u003e\nEarly studies suggest Tα1 could be an adjunct therapy for severe infections and immune dysregulation disorders.\u003cbr\u003e\nPotential application: Sepsis treatment and critical care medicine.\u003c\/p\u003e\n\n\u003cp\u003eVaccine Response Enhancement\u003c\/p\u003e\n\n\u003cp\u003eTα1 increases vaccine efficacy, particularly in immunocompromised individuals.\u003cbr\u003e\nStudies suggest Tα1 enhances antibody production and immune memory, improving response to vaccines such as avian influenza and HIV vaccines.\u003cbr\u003e\nPotential application: Improving vaccine effectiveness in at-risk populations, including the elderly and immunocompromised patients.\u003c\/p\u003e\n\n\u003cp\u003eNeuroprotection \u0026amp; Cognitive Function\u003c\/p\u003e\n\n\u003cp\u003eRecent research suggests Tα1 may promote nerve growth and enhance cognitive function.\u003cbr\u003e\nAnimal studies show improved learning, memory, and neuroplasticity following Tα1 administration.\u003cbr\u003e\nPotential application: Neurodegenerative disease research, cognitive enhancement, and brain health support.\u003c\/p\u003e\n\n\u003cp\u003eAutoimmune Disease Management\u003c\/p\u003e\n\n\u003cp\u003eTα1 helps regulate immune overactivity, making it a promising therapy for rheumatoid arthritis, multiple sclerosis, and lupus.\u003cbr\u003e\nIt reduces excessive inflammation while preserving normal immune function, which is crucial in autoimmune disease treatment.\u003cbr\u003e\nPotential application: Autoimmune disorder management and inflammation control.\u003c\/p\u003e\n\n\u003cp\u003eSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eAmino Acid Sequence: Ser-Asp-Ala-Ala-Val-Asp-Thr-Ser-Ser-Glu-Ile-Thr-Thr-Lys-Asp-Leu-Lys-Glu-Lys-Lys-Glu-Val-Val-Glu-Glu-Ala-Glu-Asn\u003cbr\u003e\nMolecular Formula: C₁₂₉H₂₁₅N₃₃O₅₅\u003cbr\u003e\nMolecular Weight: 3108.315 g\/mol\u003cbr\u003e\nCAS Registry Number: 62304-98-7\u003cbr\u003e\nSynonyms: Thymalfasin\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003c\/p\u003e\n\n\u003cp\u003eR. King \u0026amp; C. Tuthill Immune Modulation with Thymosin Alpha-1 Treatment. Vitam. Horm. (2016).\u003cbr\u003e\nC. Zhang et al. Tα1 as an immune adjuvant in avian influenza vaccination. Microb. Pathog. (2018).\u003cbr\u003e\nF. Pei et al. Thymosin Alpha-1 treatment for sepsis patients. Expert Opin. Biol. Ther. (2018).\u003cbr\u003e\nG. Wang et al. Tα1 promotes neurogenesis and cognitive function in developing mice. Neurosci. Bull. (2017).\u003cbr\u003e\nL. Romani et al. Tα1 activates dendritic cells for antifungal resistance via Toll-like receptor signaling. Blood (2004).\u003cbr\u003e\nA. L. Goldstein \u0026amp; A. L. Goldstein. Emerging clinical applications of Thymosin Alpha-1. Expert Opin. Biol. Ther. (2009).\u003cbr\u003e\nM. Maio et al. Tα1 in combination with chemotherapy improves survival in metastatic melanoma. J. Clin. Oncol. (2010).\u003cbr\u003e\nR. Danielli et al. Clinical applications of Thymosin Alpha-1 in melanoma treatment. Ann. N. Y. Acad. Sci. (2012).\u003cbr\u003e\nX. Shen et al. Development of a long-acting Thymosin Alpha-1 fusion protein for breast cancer treatment.Biomed. Pharmacother. (2018).\u003cbr\u003e\nF. Wang et al. Tα1-Fc enhances immune function and reduces melanoma and breast cancer progression. Sci. Rep. (2018).\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560594665,"sku":"PS054","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/THYMOSIN-ALPHA-10-MG.png?v=1778509474"},{"product_id":"thymalin-10mg","title":"Thymalin 10mg","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nThymalin is a synthetic version of thymulin, a peptide originally isolated from the thymus gland in 1977. It plays a critical role in immune system regulation, inflammation control, and overall longevity.\u003cbr\u003e\nResearch has identified Thymalin’s potential benefits in:\u003c\/p\u003e\n\n\u003cp\u003eEnhancing immune system function\u003cbr\u003e\nReducing inflammation and chronic pain\u003cbr\u003e\nProlonging lifespan and reducing mortality rates\u003cbr\u003e\nSupporting cardiovascular and metabolic health\u003cbr\u003e\nPromoting nervous system function and neuroprotection\u003c\/p\u003e\n\n\u003cp\u003eWith its wide-ranging effects, Thymalin has been extensively studied for its applications in immunology, anti-aging medicine, and chronic disease management.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\nImmune System Modulation\u003c\/p\u003e\n\n\u003cp\u003eThymalin enhances T-cell differentiation and increases natural killer (NK) cell activity.\u003cbr\u003e\nResearch suggests it may help restore immune balance in immunocompromised patients, including HIV and chronic immune suppression cases.\u003cbr\u003e\nIn diabetic patients with retinopathy, Thymalin boosted T-lymphocyte proliferation, reducing inflammation and slowing disease progression.\u003cbr\u003e\nPotential application: Immunotherapy, chronic inflammation treatment, and immune recovery support.\u003c\/p\u003e\n\n\u003cp\u003eAnti-Aging \u0026amp; Longevity\u003c\/p\u003e\n\n\u003cp\u003eClinical studies in elderly individuals demonstrated that long-term Thymalin administration normalized cardiovascular, immune, and nervous system functions.\u003cbr\u003e\nResearch suggests Thymalin reduces acute respiratory diseases, hypertension, osteoporosis, ischemic heart disease, and arthritis.\u003cbr\u003e\nMortality rates were reduced by up to 50% in populations receiving Thymalin therapy.\u003cbr\u003e\nPotential application: Longevity research, geriatric medicine, and chronic disease prevention.\u003c\/p\u003e\n\n\u003cp\u003eCancer Research\u003c\/p\u003e\n\n\u003cp\u003eStudies suggest Thymalin may enhance the immune system’s ability to combat cancer.\u003cbr\u003e\nWhen combined with pulsed laser therapy, Thymalin increased antibody production and immune activity, leading to higher remission rates in tumor-bearing mice.\u003cbr\u003e\nEven in sub-therapeutic doses, Thymalin demonstrated tumor-inhibiting properties, slowing cancer progression.\u003cbr\u003e\nPotential application: Cancer adjuvant therapy and tumor regression research.\u003c\/p\u003e\n\n\u003cp\u003eCardiovascular Health\u003c\/p\u003e\n\n\u003cp\u003eThymalin reduces lipid levels and T-cell dysfunction, potentially preventing and reversing atherosclerosis.\u003cbr\u003e\nAnimal studies suggest Thymalin reduces immune dysfunction that contributes to arterial plaque formation, lowering cardiovascular disease risk.\u003cbr\u003e\nPotential application: Atherosclerosis prevention, cardiovascular disease management, and lipid metabolism regulation.\u003c\/p\u003e\n\n\u003cp\u003ePostoperative Recovery \u0026amp; Infection Prevention\u003c\/p\u003e\n\n\u003cp\u003eRussian clinical trials found Thymalin highly effective in preventing post-surgical infections and inflammatory complications.\u003cbr\u003e\nMortality rates significantly decreased in high-risk patients receiving Thymalin therapy.\u003cbr\u003e\nPotential application: Postoperative recovery, infection control, and surgical immune support.\u003c\/p\u003e\n\n\u003cp\u003eNeuroprotection \u0026amp; Circadian Rhythms\u003c\/p\u003e\n\n\u003cp\u003eResearch links Thymalin to circadian rhythm regulation and seasonal immune function changes.\u003cbr\u003e\nMay help prevent age-related immune decline and lower infection susceptibility in elderly individuals.\u003cbr\u003e\nPotential application: Neurological disease prevention, circadian rhythm stabilization, and immune aging management.\u003c\/p\u003e\n\n\u003cp\u003eChronic Disease Applications\u003c\/p\u003e\n\n\u003cp\u003eTuberculosis: Clinical trials found that patients receiving Thymalin alongside standard antibiotic therapy had higher cure rates than those on antibiotics alone.\u003cbr\u003e\nKidney Disease: Research suggests Thymalin reduces inflammation and improves kidney function in patients with chronic glomerulonephritis.\u003cbr\u003e\nPsoriasis: Studies indicate that Thymalin reduces inflammation and improves skin healing in psoriasis patients.\u003cbr\u003e\nPotential application: Adjunct therapy for infectious diseases, kidney health, and autoimmune disorders.\u003c\/p\u003e\n\n\u003cp\u003eSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eAmino Acid Sequence: Pyr-Ala-Lys-Ser-Gln-Gly-Gly-Ser-Asn\u003cbr\u003e\nMolecular Formula: C₃₃H₅₄N₁₂O₁₅\u003cbr\u003e\nMolecular Weight: 858.864 g\/mol\u003cbr\u003e\nCAS Registry Number: 63958-90-7\u003cbr\u003e\nSynonyms: Thymulin, Thymic Factor, Serum Thymic Factor\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003c\/p\u003e\n\n\u003cp\u003eV. K. Khavinson \u0026amp; V. G. Morozov Peptides of the pineal gland and thymus prolong human life. Neuro Endocrinol. Lett. (2003).\u003cbr\u003e\nJ. Bach et al. Biochemical characterization of a serum thymic factor. Nature (1977).\u003cbr\u003e\nA. M. Reznichenko et al. Changes in immune cell indexes under the influence of Thymalin in patients with hyperplastic thyroid disease. Klin. Khir. (2001).\u003cbr\u003e\nT. P. Young. Immune mechanisms in HIV infection. J. Assoc. Nurses AIDS Care (2003).\u003cbr\u003e\nA. P. Kozlov \u0026amp; K. G. Moskalik. Pulsed laser radiation therapy of skin tumors. Cancer (1980).\u003cbr\u003e\nN. N. Tretiak et al. Efficacy of Thymalin and plasmapheresis in treating chronic lymphocytic leukemia patients.Lik. Sprava (1998).\u003cbr\u003e\nM. A. Karachunskiĭ et al. Cellular immunity changes in pulmonary tuberculosis patients with diabetes mellitus.Probl. Tuberk. (1997).\u003cbr\u003e\nG. V. Budazhabon et al. Thymalin’s effect on immune responses in chronic glomerulonephritis. Ter. Arkh. (1984).\u003cbr\u003e\nV. E. Ryzhenkov et al. Thymalin’s role in preventing experimental hyperlipidemia and atherosclerosis. Vopr. Med. Khim. (1988).\u003cbr\u003e\nS. Wade et al. Thymulin (Zn-facteur thymique serique) activity in anorexia nervosa patients. Am. J. Clin. Nutr. (1985).\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560627433,"sku":"PS053","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/Thymalin-10mg-scaled.jpg?v=1778509473"},{"product_id":"kisspeptin-10-5mg","title":"Kisspeptin-10 5mg","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nKisspeptin-10 (KP-10) is a naturally occurring peptide that plays a critical role in reproductive hormone regulation. It serves as the primary trigger for the release of gonadotropin-releasing hormone (GnRH), which regulates key reproductive hormones such as testosterone, estrogen, and luteinizing hormone (LH).\u003cbr\u003e\nBeyond its well-established role in reproductive function, emerging research suggests Kisspeptin-10 may also contribute to:\u003c\/p\u003e\n\n\u003cp\u003eEnhancing fertility and reproductive health\u003cbr\u003e\nIncreasing testosterone levels in males\u003cbr\u003e\nRegulating mood, motivation, and behavior\u003cbr\u003e\nModulating energy balance and metabolism\u003cbr\u003e\nSuppressing tumor growth and cancer metastasis\u003c\/p\u003e\n\n\u003cp\u003eThese potential benefits make KP-10 a promising candidate for therapeutic applications in reproductive medicine, endocrinology, neurology, and oncology.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\nReproductive Hormone Regulation\u003c\/p\u003e\n\n\u003cp\u003eKisspeptin-10 initiates the release of GnRH, which stimulates LH and FSH secretion from the pituitary gland.\u003cbr\u003e\nThis regulatory function makes it essential for puberty onset, fertility, and overall reproductive function.\u003cbr\u003e\nPotential application: Treatment of delayed puberty, hypogonadism, and infertility.\u003c\/p\u003e\n\n\u003cp\u003eTestosterone Enhancement\u003c\/p\u003e\n\n\u003cp\u003eClinical studies show that KP-10 significantly increases testosterone levels in men within 90 minutes of administration.\u003cbr\u003e\nThis occurs by modulating LH pulse frequency, which fine-tunes the natural release of sex hormones.\u003cbr\u003e\nPotential application: Hormonal therapy and fertility enhancement in males.\u003c\/p\u003e\n\n\u003cp\u003eMood, Motivation, and Libido\u003c\/p\u003e\n\n\u003cp\u003eResearch links Kisspeptin to brain regions involved in reward-seeking behavior, motivation, and emotional regulation.\u003cbr\u003e\nA clinical study on 29 men found that KP-10 administration enhanced limbic brain activity, suggesting benefits in treating low libido and mood disorders.\u003cbr\u003e\nPotential application: Treatment for hypoactive sexual desire disorder (HSDD) and mood-related conditions.\u003c\/p\u003e\n\n\u003cp\u003eEnergy Balance and Metabolism\u003c\/p\u003e\n\n\u003cp\u003eKisspeptin neurons are highly sensitive to nutritional status, influencing GnRH release based on energy availability.\u003cbr\u003e\nStudies in genetically modified mice lacking Kiss1 receptors showed weight gain and reduced energy expenditure, indicating a role in obesity regulation.\u003cbr\u003e\nPotential application: Obesity management and metabolic health support.\u003c\/p\u003e\n\n\u003cp\u003eAnti-Cancer Research\u003c\/p\u003e\n\n\u003cp\u003eKisspeptin-10 has been linked to tumor suppression by reducing metastatic spread in various cancers.\u003cbr\u003e\nA groundbreaking study found that KP-10 suppressed melanoma metastasis by 95%.\u003cbr\u003e\nAltered kisspeptin levels have also been observed in breast, prostate, bladder, ovarian, and thyroid cancers, suggesting a role in tumor migration suppression.\u003cbr\u003e\nPotential application: Development of kisspeptin-based cancer therapies.\u003c\/p\u003e\n\n\u003cp\u003eMemory and Cognitive Function\u003c\/p\u003e\n\n\u003cp\u003eResearch suggests Kisspeptin analogs may enhance memory retention and cognitive flexibility.\u003cbr\u003e\nStudies in mice indicate that KP-10 counteracts learning impairments associated with alcohol exposure.\u003cbr\u003e\nPotential application: Neurological research and cognitive function support.\u003c\/p\u003e\n\n\u003cp\u003eKidney and Cardiovascular Health\u003c\/p\u003e\n\n\u003cp\u003eKisspeptin receptors have been identified in the kidneys, where they support glomerular development and function.\u003cbr\u003e\nKP-10 has been linked to blood vessel function and cardiac output regulation, making it a potential therapeutic target for cardiovascular diseases.\u003cbr\u003e\nPotential application: Cardiovascular health and kidney disease treatment.\u003c\/p\u003e\n\n\u003cp\u003eSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eAmino Acid Sequence: YNWNSFGLRF\u003cbr\u003e\nMolecular Formula: C₆₃H₈₃N₁₇O₁₄\u003cbr\u003e\nMolecular Weight: 1302.4 g\/mol\u003cbr\u003e\nCAS Registry Number: 388138-21-4\u003cbr\u003e\nSynonyms: KISS-1, Protein KISS-1, Metastin, KP-10 Peptide\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003c\/p\u003e\n\n\u003cp\u003eW. S. Dhillo et al. Kisspeptin-54 stimulates the hypothalamic-pituitary-gonadal axis in human males. J. Clin. Endocrinol. Metab. (2005).\u003cbr\u003e\nJ. T. George et al. Kisspeptin-10 is a potent stimulator of LH and increases pulse frequency in men. J. Clin. Endocrinol. Metab. (2011).\u003cbr\u003e\nC. J. L. Harter, G. S. Kavanagh, and J. T. Smith. The role of kisspeptin neurons in reproduction and metabolism. J. Endocrinol. (2018).\u003cbr\u003e\nE. J. Mead et al. Kisspeptins: a multifunctional peptide system with a role in reproduction, cancer, and the cardiovascular system. Br. J. Pharmacol. (2007).\u003cbr\u003e\nT. Ly, S. Harihar, and D. R. Welch. KISS1 in metastatic cancer research and treatment: potential and paradoxes. Cancer Metastasis Rev. (2020).\u003cbr\u003e\nP. Pazarci et al. The effects of daylight exposure on melatonin levels, Kiss1 expression, and melanoma formation in mice. Croat. Med. J. (2020).\u003cbr\u003e\nE. Gibula-Tarlowska and J. H. Kotlinska. Kissorphin improves spatial memory and cognitive flexibility impairment induced by ethanol treatment in the Barnes maze task in rats. Behav. Pharmacol. (2020).\u003cbr\u003e\nA. N. Comninos et al. Kisspeptin modulates sexual and emotional brain processing in humans. J. Clin. Invest. (2017).\u003cbr\u003e\nM. Bhattacharya and A. V. Babwah. Kisspeptin: Beyond the Brain. Endocrinology (2015).\u003cbr\u003e\nAntitumor efficacy of Kisspeptin in human malignant mesothelioma cells. PubMed Central (PMC).\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560660201,"sku":"PS052","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/Kisspeptin-10-5mg-scaled.jpg?v=1778509473"},{"product_id":"igf-des-2mg","title":"IGF-DES 2mg","description":"IGF-DES (Insulin-Like Growth Factor-1 DES)\nOverview\nIGF-DES is a truncated form of IGF-1, which has 3-10 times higher potency in muscle growth and repair due to its enhanced receptor binding and reduced binding protein affinity.\nKey Benefits:\n✔ Rapid muscle growth and hypertrophy\n✔ Increased nitrogen retention and protein synthesis\n✔ Enhances endurance and recovery\n✔ Works locally at the site of injection\nMechanism of Action:\n\nIGF-DES bypasses IGF binding proteins, increasing its potency in muscle and tissue growth.\nStimulates hyperplasia (new muscle cell formation), unlike regular IGF-1, which only promotes hypertrophy.\n\nCitations:\n\nAdams, G. R. et al. “IGF-1 variants and their role in muscle regeneration,” J Appl Physiol, 2010.\nYang, S. Y. et al. “IGF-DES vs. IGF-1 in skeletal muscle growth,” J Cell Physiol, 2015.","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560692969,"sku":"PS050","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/IGF-DES-2MG-scaled.jpg?v=1778509474"},{"product_id":"hcg-10000iu","title":"HCG 10000IU","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nHuman Chorionic Gonadotropin (HCG) is a naturally occurring glycoprotein hormone primarily produced during pregnancy. It plays a crucial role in stimulating the production of sex hormones, particularly testosterone and estrogen. In research settings, HCG has been studied for its ability to:\u003c\/p\u003e\n\n\u003cp\u003eStimulate testosterone production by mimicking luteinizing hormone (LH)\u003cbr\u003e\nSupport spermatogenesis and fertility-related functions\u003cbr\u003e\nMaintain testicular size and function in prolonged androgen therapy research\u003cbr\u003e\nPotentially aid in weight regulation by influencing metabolic activity\u003cbr\u003e\nModulate endocrine function in hormone-related studies\u003c\/p\u003e\n\n\u003cp\u003eHCG is commonly investigated in endocrinology and reproductive research due to its ability to regulate hormone production and influence metabolic processes.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\nHCG and Testosterone Production\u003cbr\u003e\nHCG interacts with luteinizing hormone receptors in the Leydig cells of the testes, promoting the production of testosterone. Studies indicate that:\u003c\/p\u003e\n\n\u003cp\u003eHCG mimics LH, stimulating natural testosterone synthesis.\u003cbr\u003e\nIt may help preserve testicular function during long-term anabolic hormone research.\u003cbr\u003e\nResearch suggests it plays a role in hormone balance and androgen regulation.\u003c\/p\u003e\n\n\u003cp\u003eHCG and Fertility Research\u003cbr\u003e\nHCG has been extensively studied for its potential role in male and female fertility. Findings include:\u003c\/p\u003e\n\n\u003cp\u003eStimulation of sperm production in hypogonadal models.\u003cbr\u003e\nInduction of ovulation in reproductive studies.\u003cbr\u003e\nPotential restoration of endogenous hormone balance after exogenous hormone exposure.\u003c\/p\u003e\n\n\u003cp\u003eHCG and Weight Regulation\u003cbr\u003e\nSome research has explored HCG’s potential effects on metabolism and fat distribution. Studies suggest:\u003c\/p\u003e\n\n\u003cp\u003eIt may influence fat mobilization during periods of caloric restriction.\u003cbr\u003e\nPotential interactions with the hypothalamic-pituitary-adrenal (HPA) axis that affect metabolic function.\u003cbr\u003e\nFurther studies are needed to understand its role in adipose tissue regulation.\u003c\/p\u003e\n\n\u003cp\u003eHCG and Endocrine Function\u003cbr\u003e\nBeyond reproductive health, HCG has been investigated for its role in overall endocrine system modulation. Research indicates:\u003c\/p\u003e\n\n\u003cp\u003eIt may support adrenal function by interacting with steroidogenic pathways.\u003cbr\u003e\nPotential applications in research related to thyroid function and metabolic balance.\u003cbr\u003e\nIts structural similarity to other glycoprotein hormones suggests possible cross-signaling effects.\u003c\/p\u003e\n\n\u003cp\u003eSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eMolecular Formula: C₁₀₅H₁₆₉N₂₇O₃₂\u003cbr\u003e\nMolecular Weight: 2,321.6 g\/mol\u003cbr\u003e\nCAS Registry Number: 9002-61-3\u003cbr\u003e\nPurity: 99% HPLC\u003c\/p\u003e\n\n\u003cp\u003eRESEARCH USE ONLY\u003cbr\u003e\nHCG is strictly intended for research and laboratory purposes. It is not for human or veterinary use and is not intended for therapeutic or diagnostic applications.\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560725737,"sku":"PS048","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/HCG-10000IU-scaled.jpg?v=1778509475"},{"product_id":"hcg-5000iu","title":"HCG 5000IU","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nHuman Chorionic Gonadotropin (HCG) is a naturally occurring glycoprotein hormone primarily produced during pregnancy. It plays a crucial role in stimulating the production of sex hormones, particularly testosterone and estrogen. In research settings, HCG has been studied for its ability to:\u003c\/p\u003e\n\n\u003cp\u003eStimulate testosterone production by mimicking luteinizing hormone (LH)\u003cbr\u003e\nSupport spermatogenesis and fertility-related functions\u003cbr\u003e\nMaintain testicular size and function in prolonged androgen therapy research\u003cbr\u003e\nPotentially aid in weight regulation by influencing metabolic activity\u003cbr\u003e\nModulate endocrine function in hormone-related studies\u003c\/p\u003e\n\n\u003cp\u003eHCG is commonly investigated in endocrinology and reproductive research due to its ability to regulate hormone production and influence metabolic processes.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\nHCG and Testosterone Production\u003cbr\u003e\nHCG interacts with luteinizing hormone receptors in the Leydig cells of the testes, promoting the production of testosterone. Studies indicate that:\u003c\/p\u003e\n\n\u003cp\u003eHCG mimics LH, stimulating natural testosterone synthesis.\u003cbr\u003e\nIt may help preserve testicular function during long-term anabolic hormone research.\u003cbr\u003e\nResearch suggests it plays a role in hormone balance and androgen regulation.\u003c\/p\u003e\n\n\u003cp\u003eHCG and Fertility Research\u003cbr\u003e\nHCG has been extensively studied for its potential role in male and female fertility. Findings include:\u003c\/p\u003e\n\n\u003cp\u003eStimulation of sperm production in hypogonadal models.\u003cbr\u003e\nInduction of ovulation in reproductive studies.\u003cbr\u003e\nPotential restoration of endogenous hormone balance after exogenous hormone exposure.\u003c\/p\u003e\n\n\u003cp\u003eHCG and Weight Regulation\u003cbr\u003e\nSome research has explored HCG’s potential effects on metabolism and fat distribution. Studies suggest:\u003c\/p\u003e\n\n\u003cp\u003eIt may influence fat mobilization during periods of caloric restriction.\u003cbr\u003e\nPotential interactions with the hypothalamic-pituitary-adrenal (HPA) axis that affect metabolic function.\u003cbr\u003e\nFurther studies are needed to understand its role in adipose tissue regulation.\u003c\/p\u003e\n\n\u003cp\u003eHCG and Endocrine Function\u003cbr\u003e\nBeyond reproductive health, HCG has been investigated for its role in overall endocrine system modulation. Research indicates:\u003c\/p\u003e\n\n\u003cp\u003eIt may support adrenal function by interacting with steroidogenic pathways.\u003cbr\u003e\nPotential applications in research related to thyroid function and metabolic balance.\u003cbr\u003e\nIts structural similarity to other glycoprotein hormones suggests possible cross-signaling effects.\u003c\/p\u003e\n\n\u003cp\u003eSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eMolecular Formula: C₁₀₅H₁₆₉N₂₇O₃₂\u003cbr\u003e\nMolecular Weight: 2,321.6 g\/mol\u003cbr\u003e\nCAS Registry Number: 9002-61-3\u003cbr\u003e\nPurity: 99% HPLC\u003c\/p\u003e\n\n\u003cp\u003eRESEARCH USE ONLY\u003cbr\u003e\nHCG is strictly intended for research and laboratory purposes. It is not for human or veterinary use and is not intended for therapeutic or diagnostic applications.\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560758505,"sku":"PS047","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/HCG-5000IU-scaled.jpg?v=1778509476"},{"product_id":"sermorelin-5mg","title":"Sermorelin 5mg","description":"OVERVIEW\nSermorelin is a synthetic analogue of growth hormone-releasing hormone (GHRH), designed to stimulate natural growth hormone (GH) secretion. It has been widely researched for its clinical applications in GH assessment and its potential benefits in cardiovascular health, cognitive function, and metabolic regulation.\nStudies suggest Sermorelin may:\n\nIncrease bone density, supporting skeletal health.\nReduce scarring, aiding in tissue healing and wound repair.\nEnhance renal function, potentially benefiting kidney health.\nSupport cognitive function and fight dementia, by promoting neuronal protection.\nReduce seizure activity, through GABA receptor interactions.\nImprove cardiovascular health, by aiding heart tissue repair after damage.\n\nUnlike direct GH therapy, Sermorelin preserves the body’s natural GH regulation, reducing the risk of overdosing, joint pain, and GH desensitization.\nRESEARCH\nSermorelin and Heart Health\n\nResearch in pig models demonstrated that Sermorelin reduces cardiac remodeling following a heart attack.\nMechanisms include:\n\nDecreasing cardiomyocyte (heart cell) death.\nEnhancing extracellular matrix production, supporting tissue healing.\nStimulating new blood vessel growth, improving circulation.\nReducing inflammation, lowering further heart damage risk.\n\n\nPotential application: Post-myocardial infarction recovery and heart failure management.\n\nSermorelin and Epilepsy\n\nStudies suggest Sermorelin activates GABA receptors, which help suppress seizures.\nAnimal models of epilepsy showed that GHRH analogues reduce seizure activity.\nPotential application: Adjunctive therapy for seizure disorders.\n\nSermorelin and Sleep Regulation\n\nSermorelin may influence orexin, a key neurochemical regulating wakefulness and sleep cycles.\nExogenous administration of Sermorelin has been linked to more stable sleep patterns.\nPotential application: Treatment for sleep disturbances and optimization of sleep quality.\n\nSermorelin as a Preferred Alternative to GH Therapy\nUnlike direct GH administration, Sermorelin:\n\nPreserves physiological GH feedback mechanisms, reducing the risk of GH overdose and dysregulation.\nDoes not cause tachyphylaxis (desensitization), meaning the body does not lose sensitivity to its effects over time.\nHas a lower risk of side effects, such as edema, joint pain, and metabolic imbalances.\nPotential application: Safer alternative for individuals requiring GH optimization.\n\nSTRUCTURE\n\nMolecular Formula: C₁₄₉H₂₄₆N₄₄O₄₂S\nMolecular Weight: 3357.933 g\/mol\nAmino Acid Sequence:\nTyr-DL-Ala-DL-Asp-DL-Ala-DL-xiIle-DL-Phe-DL-xiThr-DL-Asn-DL-Ser-DL-Tyr-DL-Arg-DL-Lys-DL-Val-DL-Leu-Gly-DL-Gln-DL-Leu-DL-Ser-DL-Ala-DL-Arg-DL-Lys-DL-Leu-DL-Leu-DL-Gln-DL-Asp-DL-xiIle-DL-Met-DL-Ser-DL-Arg\nCAS Registry Number: 86168-78-7\n\nCITATIONS\n\nL. L. Bagno et al. Growth Hormone–Releasing Hormone Agonists Reduce Myocardial Infarct Scar in Swine With Subacute Ischemic Cardiomyopathy. J. Am. Heart Assoc. Cardiovasc. Cerebrovasc. Dis. (2015).\nR. M. Kanashiro-Takeuchi et al. New therapeutic approach to heart failure due to myocardial infarction based on targeting growth hormone-releasing hormone receptor. Oncotarget (2015).\nS. Tang et al. Interactions between GHRH and GABAARs in the brains of patients with epilepsy and in animal models of epilepsy. Sci. Rep. (2017).\nB. S. Shepherd et al. Endocrine and orexigenic actions of growth hormone secretagogues in rainbow trout (Oncorhynchus mykiss). Comp. Biochem. Physiol. A. Mol. Integr. Physiol. (2007).\nR. F. Walker. Sermorelin: A better approach to management of adult-onset growth hormone insufficiency? Clin. Interv. Aging (2006).\nS. T. Wahid et al. Partial tachyphylaxis to somatostatin (SST) analogues in a patient with acromegaly: the role of SST receptor desensitisation and circulating antibodies to SST analogues. Eur. J. Endocrinol. (2002).","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560791273,"sku":"PS045","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/Sermorelin-5mg-scaled.jpg?v=1778509477"},{"product_id":"peg-mgf-2mg","title":"PEG-MGF 2mg","description":"PEG-MGF (Pegylated Mechano Growth Factor)\nOverview\nPEG-MGF is a modified form of MGF that has been pegylated to increase half-life and stability, allowing for systemic effects rather than localized response. It is widely used in muscle recovery, tissue repair, and anti-aging applications.\nKey Benefits:\n✔ Extended half-life compared to MGF\n✔ Systemic muscle repair and growth\n✔ Improved nitrogen retention and protein synthesis\n✔ Reduced muscle atrophy and enhanced recovery\nMechanism of Action:\n\nPegylation increases MGF’s stability, allowing it to circulate longer in the bloodstream.\nEnhances muscle satellite cell activation and IGF-1 expression for greater muscle recovery and growth.\n\nCitations:\n\nBarton-Davis, E. R. et al. “PEGylation of MGF enhances muscle repair in aged mice,” Am J Physiol Cell Physiol, 2010.\nHarridge, S. D. et al. “Mechano growth factor’s role in muscle adaptation,” Exp Physiol, 2007.","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560824041,"sku":"PS044","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/PEG-MGF-2mg-scaled.jpg?v=1778509478"},{"product_id":"mgf-2mg","title":"MGF 2mg","description":"MGF (Mechano Growth Factor)\nOverview\nMechano Growth Factor (MGF) is a splice variant of IGF-1 (Insulin-Like Growth Factor-1) that is primarily involved in muscle repair and regeneration. It is produced in response to mechanical stress (e.g., resistance training, injury)and stimulates satellite cell activation, which is crucial for muscle growth and hypertrophy.\nKey Benefits:\n✔ Enhances muscle repair and recovery\n✔ Promotes satellite cell activation\n✔ Increases muscle hypertrophy and strength\n✔ Supports injury rehabilitation and tissue repair\nMechanism of Action:\n\nMGF activates satellite cells, which fuse with existing muscle fibers to promote growth and repair.\nEnhances protein synthesis and nitrogen retention, essential for muscle regeneration.\nIncreases local IGF-1 expression, further accelerating tissue recovery.\n\nCitations:\n\nGoldspink, G. et al. “Mechano growth factor: a potent stimulator of muscle regeneration,” J Cell Biol, 2002.\nMcKoy, G. et al. “MGF and its role in muscle growth and repair,” Clin Sci, 2005.","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560856809,"sku":"PS043","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/MGF-2mg-scaled.jpg?v=1778509478"},{"product_id":"ghrp-6-10mg","title":"GHRP-6 10mg","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nGHRP-6 (Growth Hormone-Releasing Peptide-6) is a synthetic peptide that stimulates growth hormone (GH) release by acting as a ghrelin receptor agonist. It has been extensively studied for its potential benefits in appetite regulation, tissue repair, cognitive function, and cardiovascular health.\u003cbr\u003e\nResearch suggests GHRP-6 may:\u003c\/p\u003e\n\n\u003cp\u003eEnhance memory and cognitive function, supporting long-term memory formation.\u003cbr\u003e\nPromote neuroprotection, reducing inflammation and cell death in brain tissue.\u003cbr\u003e\nAccelerate skin and wound healing, increasing collagen production and reducing scar formation.\u003cbr\u003e\nProtect heart tissue, minimizing oxidative stress and damage following myocardial infarction.\u003cbr\u003e\nRegulate mood and sexual motivation, possibly reducing symptoms of depression and stress.\u003cbr\u003e\nSupport metabolic balance, potentially influencing obesity and energy homeostasis.\u003c\/p\u003e\n\n\u003cp\u003eDue to these properties, GHRP-6 has potential therapeutic applications in neurological disorders, cardiovascular health, and tissue regeneration.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\nGHRP-6 and Cognitive Function\u003c\/p\u003e\n\n\u003cp\u003eStudies suggest GHRP-6 enhances memory formation and spatial learning, indicating potential cognitive benefits.\u003cbr\u003e\nMay aid in converting short-term memories into long-term storage, improving overall cognitive performance.\u003cbr\u003e\nPotential application: Treatment for age-related cognitive decline and neurodegenerative disorders.\u003c\/p\u003e\n\n\u003cp\u003eGHRP-6 and Neuroprotection\u003c\/p\u003e\n\n\u003cp\u003eResearch indicates that GHRP-6 reduces inflammation and prevents apoptosis (programmed cell death) in brain tissue.\u003cbr\u003e\nMay play a role in stroke recovery and neurodegenerative diseases such as Parkinson’s disease.\u003cbr\u003e\nPotential application: Neuroprotection and post-stroke rehabilitation.\u003c\/p\u003e\n\n\u003cp\u003eGHRP-6 and Skin Healing\u003c\/p\u003e\n\n\u003cp\u003eGHRP-6 has been found to promote tissue regeneration and enhance wound closure.\u003cbr\u003e\nIncreases collagen production, reducing the risk of hypertrophic scarring.\u003cbr\u003e\nPotential application: Treatment for burn injuries, post-surgical healing, and chronic wounds.\u003c\/p\u003e\n\n\u003cp\u003eGHRP-6 and Cardiovascular Benefits\u003c\/p\u003e\n\n\u003cp\u003eStudies in animal models suggest GHRP-6 reduces heart damage following a myocardial infarction (heart attack).\u003cbr\u003e\nMay protect heart cells from oxidative stress, improving cardiac function.\u003cbr\u003e\nPotential application: Cardioprotection and post-heart attack recovery.\u003c\/p\u003e\n\n\u003cp\u003eGHRP-6 and Mood Regulation\u003c\/p\u003e\n\n\u003cp\u003eResearch in rats suggests that GHRP-6 influences sexual motivation and mood by interacting with ghrelin receptors in the brain.\u003cbr\u003e\nMay help regulate symptoms of depression and stress, offering mood-enhancing benefits.\u003cbr\u003e\nPotential application: Mental health support and stress management.\u003c\/p\u003e\n\n\u003cp\u003eGHRP-6 and Metabolism\u003c\/p\u003e\n\n\u003cp\u003eGHRP-6 has been investigated for its role in regulating metabolism, particularly in growth hormone secretion and energy balance.\u003cbr\u003e\nPotential application: Treatment for metabolic disorders and obesity management.\u003c\/p\u003e\n\n\u003cp\u003eSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eMolecular Formula: C₄₆H₅₆N₁₂O₆\u003cbr\u003e\nMolecular Weight: 873.032 g\/mol\u003cbr\u003e\nAmino Acid Sequence: His-D-Trp-Ala-Trp-D-Phe-Lys\u003cbr\u003e\nCAS Registry Number: 87616-84-0\u003cbr\u003e\nMechanism of Action: GHRP-6 binds to the ghrelin receptor (GHS-R1a) in the hypothalamus and pituitary gland, stimulating growth hormone release, which enhances cell growth, tissue repair, and metabolic function.\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003c\/p\u003e\n\n\u003cp\u003eHuang, C.-C., Chou, D., Yeh, C.-M., \u0026amp; Hsu, K.-S. Acute food deprivation enhances fear extinction but inhibits long-term depression in the lateral amygdala via ghrelin signaling. Neuropharmacology (2016).\u003cbr\u003e\nBeheshti, S., \u0026amp; Shahrokhi, S. Blocking the ghrelin receptor type 1a in the rat brain impairs memory encoding.Neuropeptides (2015).\u003cbr\u003e\nTóth, K., László, K., \u0026amp; Lénárd, L. Role of intraamygdaloid acylated-ghrelin in spatial learning. Brain Res. Bull. (2010).\u003cbr\u003e\nSubirós, N. et al. (Additional references pending completion)\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560889577,"sku":"PS042","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/GHRP6-10MG-scaled.jpg?v=1778509479"},{"product_id":"ghrp-6-5mg","title":"GHRP-6 5mg","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nGHRP-6 (Growth Hormone-Releasing Peptide-6) is a synthetic peptide that stimulates growth hormone (GH) release by acting as a ghrelin receptor agonist. It has been extensively studied for its potential benefits in appetite regulation, tissue repair, cognitive function, and cardiovascular health.\u003cbr\u003e\nResearch suggests GHRP-6 may:\u003c\/p\u003e\n\n\u003cp\u003eEnhance memory and cognitive function, supporting long-term memory formation.\u003cbr\u003e\nPromote neuroprotection, reducing inflammation and cell death in brain tissue.\u003cbr\u003e\nAccelerate skin and wound healing, increasing collagen production and reducing scar formation.\u003cbr\u003e\nProtect heart tissue, minimizing oxidative stress and damage following myocardial infarction.\u003cbr\u003e\nRegulate mood and sexual motivation, possibly reducing symptoms of depression and stress.\u003cbr\u003e\nSupport metabolic balance, potentially influencing obesity and energy homeostasis.\u003c\/p\u003e\n\n\u003cp\u003eDue to these properties, GHRP-6 has potential therapeutic applications in neurological disorders, cardiovascular health, and tissue regeneration.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\nGHRP-6 and Cognitive Function\u003c\/p\u003e\n\n\u003cp\u003eStudies suggest GHRP-6 enhances memory formation and spatial learning, indicating potential cognitive benefits.\u003cbr\u003e\nMay aid in converting short-term memories into long-term storage, improving overall cognitive performance.\u003cbr\u003e\nPotential application: Treatment for age-related cognitive decline and neurodegenerative disorders.\u003c\/p\u003e\n\n\u003cp\u003eGHRP-6 and Neuroprotection\u003c\/p\u003e\n\n\u003cp\u003eResearch indicates that GHRP-6 reduces inflammation and prevents apoptosis (programmed cell death) in brain tissue.\u003cbr\u003e\nMay play a role in stroke recovery and neurodegenerative diseases such as Parkinson’s disease.\u003cbr\u003e\nPotential application: Neuroprotection and post-stroke rehabilitation.\u003c\/p\u003e\n\n\u003cp\u003eGHRP-6 and Skin Healing\u003c\/p\u003e\n\n\u003cp\u003eGHRP-6 has been found to promote tissue regeneration and enhance wound closure.\u003cbr\u003e\nIncreases collagen production, reducing the risk of hypertrophic scarring.\u003cbr\u003e\nPotential application: Treatment for burn injuries, post-surgical healing, and chronic wounds.\u003c\/p\u003e\n\n\u003cp\u003eGHRP-6 and Cardiovascular Benefits\u003c\/p\u003e\n\n\u003cp\u003eStudies in animal models suggest GHRP-6 reduces heart damage following a myocardial infarction (heart attack).\u003cbr\u003e\nMay protect heart cells from oxidative stress, improving cardiac function.\u003cbr\u003e\nPotential application: Cardioprotection and post-heart attack recovery.\u003c\/p\u003e\n\n\u003cp\u003eGHRP-6 and Mood Regulation\u003c\/p\u003e\n\n\u003cp\u003eResearch in rats suggests that GHRP-6 influences sexual motivation and mood by interacting with ghrelin receptors in the brain.\u003cbr\u003e\nMay help regulate symptoms of depression and stress, offering mood-enhancing benefits.\u003cbr\u003e\nPotential application: Mental health support and stress management.\u003c\/p\u003e\n\n\u003cp\u003eGHRP-6 and Metabolism\u003c\/p\u003e\n\n\u003cp\u003eGHRP-6 has been investigated for its role in regulating metabolism, particularly in growth hormone secretion and energy balance.\u003cbr\u003e\nPotential application: Treatment for metabolic disorders and obesity management.\u003c\/p\u003e\n\n\u003cp\u003eSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eMolecular Formula: C₄₆H₅₆N₁₂O₆\u003cbr\u003e\nMolecular Weight: 873.032 g\/mol\u003cbr\u003e\nAmino Acid Sequence: His-D-Trp-Ala-Trp-D-Phe-Lys\u003cbr\u003e\nCAS Registry Number: 87616-84-0\u003cbr\u003e\nMechanism of Action: GHRP-6 binds to the ghrelin receptor (GHS-R1a) in the hypothalamus and pituitary gland, stimulating growth hormone release, which enhances cell growth, tissue repair, and metabolic function.\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003c\/p\u003e\n\n\u003cp\u003eHuang, C.-C., Chou, D., Yeh, C.-M., \u0026amp; Hsu, K.-S. Acute food deprivation enhances fear extinction but inhibits long-term depression in the lateral amygdala via ghrelin signaling. Neuropharmacology (2016).\u003cbr\u003e\nBeheshti, S., \u0026amp; Shahrokhi, S. Blocking the ghrelin receptor type 1a in the rat brain impairs memory encoding.Neuropeptides (2015).\u003cbr\u003e\nTóth, K., László, K., \u0026amp; Lénárd, L. Role of intraamygdaloid acylated-ghrelin in spatial learning. Brain Res. Bull. (2010).\u003cbr\u003e\nSubirós, N. et al. (Additional references pending completion)\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560922345,"sku":"PS041","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/GHRP6-5MG-scaled.jpg?v=1778509480"},{"product_id":"ghrp-2-10mg","title":"GHRP-2 10mg","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nGHRP-2 (Growth Hormone-Releasing Peptide-2), also known as Pralmorelin, is a synthetic growth hormone secretagogue (GHS) that binds to the ghrelin\/GHS receptor (GHS-R1a). It is one of the most potent GH-releasing peptides and has been extensively studied for its ability to enhance muscle growth, regulate metabolism, and support immune function.\u003cbr\u003e\nResearch suggests GHRP-2 may:\u003c\/p\u003e\n\n\u003cp\u003eStimulate growth hormone (GH) release, enhancing muscle repair and growth.\u003cbr\u003e\nBoost immune function through thymus stimulation and T-cell production.\u003cbr\u003e\nRegulate appetite and metabolism by mimicking ghrelin.\u003cbr\u003e\nImprove sleep quality and recovery.\u003cbr\u003e\nProvide cardioprotective and neuroprotective benefits.\u003c\/p\u003e\n\n\u003cp\u003eGHRP-2 is widely studied for its strong GH-releasing effects, with multiple administration options, including oral, sublingual, and injectable routes.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\nGHRP-2 and Muscle Growth\u003c\/p\u003e\n\n\u003cp\u003eResearch on Yaks (Bos grunniens) found that GHRP-2 enhances muscle growth by increasing protein deposition and reducing protein degradation.\u003cbr\u003e\nGHRP-2 suppresses Atrogin-1 and MuRF1, two key proteins involved in muscle breakdown.\u003cbr\u003e\nPotential application: Treatment for muscle-wasting conditions such as cachexia and sarcopenia.\u003c\/p\u003e\n\n\u003cp\u003eGHRP-2 and Appetite Regulation\u003c\/p\u003e\n\n\u003cp\u003eGHRP-2 activates ghrelin receptors, leading to increased food intake.\u003cbr\u003e\nClinical studies in healthy men showed that GHRP-2 significantly increased caloric intake, highlighting its role as an appetite stimulant.\u003cbr\u003e\nPotential application: Treatment for chronic illness-related appetite loss and malnutrition.\u003c\/p\u003e\n\n\u003cp\u003eGHRP-2 and Cardioprotection\u003c\/p\u003e\n\n\u003cp\u003eResearch in fetal heart cell cultures demonstrated that GHRP-2 and its analogs (GHRP-1, GHRP-6) protect heart cells from apoptosis (cell death).\u003cbr\u003e\nPotential application: Post-heart attack recovery, reducing cardiac tissue damage.\u003c\/p\u003e\n\n\u003cp\u003eGHRP-2 and Immune System Function\u003c\/p\u003e\n\n\u003cp\u003eGHRP-2 stimulates the thymus, an organ responsible for T-cell production and immune function.\u003cbr\u003e\nAging reduces thymus function, leading to weakened immune responses.\u003cbr\u003e\nStudies show that GHRP-2 rejuvenates the thymus, increasing T-cell production and immune resilience.\u003cbr\u003e\nPotential application: Treatment for age-related immune decline and autoimmune conditions.\u003c\/p\u003e\n\n\u003cp\u003eGHRP-2 and Sleep Quality\u003c\/p\u003e\n\n\u003cp\u003eGHRP-2 has been shown to increase deep sleep (slow-wave sleep) by 50% and REM sleep by 20%.\u003cbr\u003e\nImproved sleep enhances cognitive function, physical recovery, and hormonal balance.\u003cbr\u003e\nPotential application: Treatment for sleep disorders and sleep optimization.\u003c\/p\u003e\n\n\u003cp\u003eGHRP-2 and Pain Perception\u003c\/p\u003e\n\n\u003cp\u003eResearch suggests GHRP-2 interacts with opioid receptors, modulating pain perception.\u003cbr\u003e\nStudies in osteoarthritis models found that GHRP-2 reduced pain independently of tissue healing.\u003cbr\u003e\nPotential application: Alternative pain management therapy with reduced opioid dependency.\u003c\/p\u003e\n\n\u003cp\u003eSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eMolecular Formula: C₄₅H₅₅N₉O₆\u003cbr\u003e\nMolecular Weight: 817.97 g\/mol\u003cbr\u003e\nAmino Acid Sequence: D-Ala-D-2Nal-Ala-Trp-D-Phe-Lys\u003cbr\u003e\nCAS Registry Number: 158861-67-7\u003cbr\u003e\nPubChem Identifier: 6918245\u003cbr\u003e\nSynonyms: KP-102, GPA-748, WAY-GPA-748\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003c\/p\u003e\n\n\u003cp\u003eHu, R. et al. Effects of GHRP-2 and Cysteamine Administration on Growth Performance, Somatotropic Axis Hormone, and Muscle Protein Deposition in Yaks (Bos grunniens) with Growth Retardation. PLOS One (2016).\u003cbr\u003e\nYamamoto, D. et al. GHRP-2, a GHS-R agonist, directly acts on myocytes to attenuate the dexamethasone-induced expressions of muscle-specific ubiquitin ligases, Atrogin-1 and MuRF1. Life Sci. (2008).\u003cbr\u003e\nPhung, L. T. et al. The effects of growth hormone-releasing peptide-2 (GHRP-2) on the release of growth hormone and growth performance in swine. Domest. Anim. Endocrinol. (2000).\u003cbr\u003e\nLaferrère, B. et al. Growth hormone releasing peptide-2 (GHRP-2), like ghrelin, increases food intake in healthy men. J. Clin. Endocrinol. Metab. (2005).\u003cbr\u003e\nBodart, V. et al. Identification and characterization of a new growth hormone-releasing peptide receptor in the heart. Circ. Res. (1999).\u003cbr\u003e\nTaub, D. D. et al. Rejuvenation of the aging thymus: growth hormone-mediated and ghrelin-mediated signaling pathways. Curr. Opin. Pharmacol. (2010).\u003cbr\u003e\nCopinschi, G. et al. Prolonged oral treatment with MK-677, a novel growth hormone secretagogue, improves sleep quality in man. Neuroendocrinology (1997).\u003cbr\u003e\nZeng, P. et al. Ghrelin receptor agonist, GHRP-2, produces antinociceptive effects at the supraspinal level via the opioid receptor in mice. Peptides (2014).\u003cbr\u003e\nFehrentz, J. A. et al. Recent Developments in Ghrelin Receptor Ligands. ChemMedChem (2007).\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560955113,"sku":"PS040","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/Untitled_design_-_2026-05-17T062356.748.png?v=1779024256"},{"product_id":"ghrp-2-5mg","title":"GHRP-2 5mg","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nGHRP-2 (Growth Hormone-Releasing Peptide-2), also known as Pralmorelin, is a synthetic growth hormone secretagogue (GHS) that binds to the ghrelin\/GHS receptor (GHS-R1a). It is one of the most potent GH-releasing peptides and has been extensively studied for its ability to enhance muscle growth, regulate metabolism, and support immune function.\u003cbr\u003e\nResearch suggests GHRP-2 may:\u003c\/p\u003e\n\n\u003cp\u003eStimulate growth hormone (GH) release, enhancing muscle repair and growth.\u003cbr\u003e\nBoost immune function through thymus stimulation and T-cell production.\u003cbr\u003e\nRegulate appetite and metabolism by mimicking ghrelin.\u003cbr\u003e\nImprove sleep quality and recovery.\u003cbr\u003e\nProvide cardioprotective and neuroprotective benefits.\u003c\/p\u003e\n\n\u003cp\u003eGHRP-2 is widely studied for its strong GH-releasing effects, with multiple administration options, including oral, sublingual, and injectable routes.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\nGHRP-2 and Muscle Growth\u003c\/p\u003e\n\n\u003cp\u003eResearch on Yaks (Bos grunniens) found that GHRP-2 enhances muscle growth by increasing protein deposition and reducing protein degradation.\u003cbr\u003e\nGHRP-2 suppresses Atrogin-1 and MuRF1, two key proteins involved in muscle breakdown.\u003cbr\u003e\nPotential application: Treatment for muscle-wasting conditions such as cachexia and sarcopenia.\u003c\/p\u003e\n\n\u003cp\u003eGHRP-2 and Appetite Regulation\u003c\/p\u003e\n\n\u003cp\u003eGHRP-2 activates ghrelin receptors, leading to increased food intake.\u003cbr\u003e\nClinical studies in healthy men showed that GHRP-2 significantly increased caloric intake, highlighting its role as an appetite stimulant.\u003cbr\u003e\nPotential application: Treatment for chronic illness-related appetite loss and malnutrition.\u003c\/p\u003e\n\n\u003cp\u003eGHRP-2 and Cardioprotection\u003c\/p\u003e\n\n\u003cp\u003eResearch in fetal heart cell cultures demonstrated that GHRP-2 and its analogs (GHRP-1, GHRP-6) protect heart cells from apoptosis (cell death).\u003cbr\u003e\nPotential application: Post-heart attack recovery, reducing cardiac tissue damage.\u003c\/p\u003e\n\n\u003cp\u003eGHRP-2 and Immune System Function\u003c\/p\u003e\n\n\u003cp\u003eGHRP-2 stimulates the thymus, an organ responsible for T-cell production and immune function.\u003cbr\u003e\nAging reduces thymus function, leading to weakened immune responses.\u003cbr\u003e\nStudies show that GHRP-2 rejuvenates the thymus, increasing T-cell production and immune resilience.\u003cbr\u003e\nPotential application: Treatment for age-related immune decline and autoimmune conditions.\u003c\/p\u003e\n\n\u003cp\u003eGHRP-2 and Sleep Quality\u003c\/p\u003e\n\n\u003cp\u003eGHRP-2 has been shown to increase deep sleep (slow-wave sleep) by 50% and REM sleep by 20%.\u003cbr\u003e\nImproved sleep enhances cognitive function, physical recovery, and hormonal balance.\u003cbr\u003e\nPotential application: Treatment for sleep disorders and sleep optimization.\u003c\/p\u003e\n\n\u003cp\u003eGHRP-2 and Pain Perception\u003c\/p\u003e\n\n\u003cp\u003eResearch suggests GHRP-2 interacts with opioid receptors, modulating pain perception.\u003cbr\u003e\nStudies in osteoarthritis models found that GHRP-2 reduced pain independently of tissue healing.\u003cbr\u003e\nPotential application: Alternative pain management therapy with reduced opioid dependency.\u003c\/p\u003e\n\n\u003cp\u003eSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eMolecular Formula: C₄₅H₅₅N₉O₆\u003cbr\u003e\nMolecular Weight: 817.97 g\/mol\u003cbr\u003e\nAmino Acid Sequence: D-Ala-D-2Nal-Ala-Trp-D-Phe-Lys\u003cbr\u003e\nCAS Registry Number: 158861-67-7\u003cbr\u003e\nPubChem Identifier: 6918245\u003cbr\u003e\nSynonyms: KP-102, GPA-748, WAY-GPA-748\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003c\/p\u003e\n\n\u003cp\u003eHu, R. et al. Effects of GHRP-2 and Cysteamine Administration on Growth Performance, Somatotropic Axis Hormone, and Muscle Protein Deposition in Yaks (Bos grunniens) with Growth Retardation. PLOS One (2016).\u003cbr\u003e\nYamamoto, D. et al. GHRP-2, a GHS-R agonist, directly acts on myocytes to attenuate the dexamethasone-induced expressions of muscle-specific ubiquitin ligases, Atrogin-1 and MuRF1. Life Sci. (2008).\u003cbr\u003e\nPhung, L. T. et al. The effects of growth hormone-releasing peptide-2 (GHRP-2) on the release of growth hormone and growth performance in swine. Domest. Anim. Endocrinol. (2000).\u003cbr\u003e\nLaferrère, B. et al. Growth hormone releasing peptide-2 (GHRP-2), like ghrelin, increases food intake in healthy men. J. Clin. Endocrinol. Metab. (2005).\u003cbr\u003e\nBodart, V. et al. Identification and characterization of a new growth hormone-releasing peptide receptor in the heart. Circ. Res. (1999).\u003cbr\u003e\nTaub, D. D. et al. Rejuvenation of the aging thymus: growth hormone-mediated and ghrelin-mediated signaling pathways. Curr. Opin. Pharmacol. (2010).\u003cbr\u003e\nCopinschi, G. et al. Prolonged oral treatment with MK-677, a novel growth hormone secretagogue, improves sleep quality in man. Neuroendocrinology (1997).\u003cbr\u003e\nZeng, P. et al. Ghrelin receptor agonist, GHRP-2, produces antinociceptive effects at the supraspinal level via the opioid receptor in mice. Peptides (2014).\u003cbr\u003e\nFehrentz, J. A. et al. Recent Developments in Ghrelin Receptor Ligands. ChemMedChem (2007).\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989560987881,"sku":"PS039","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/GHRP2-5MG-scaled.jpg?v=1778509482"},{"product_id":"epithalon-10mg","title":"Epithalon 10mg","description":"Epithalon (Epitalon) Peptide\nOverview\nEpithalon (also known as Epitalon) is a synthetic tetrapeptide derived from Epithalamin, a naturally occurring peptide in the pineal gland. It has been extensively studied for its anti-aging, telomerase-activating, and lifespan-extending properties. Epithalon is primarily known for its ability to:\n✔ Activate telomerase, promoting DNA repair and longevity\n✔ Enhance melatonin production, regulating circadian rhythms and sleep\n✔ Reduce oxidative stress and inflammation\n✔ Support immune function and cellular repair\n✔ Improve cardiovascular and metabolic health\nMechanism of Action\n1. Telomerase Activation \u0026amp; Longevity\n\nTelomeres shorten with age, leading to cellular aging and senescence.\nEpithalon activates telomerase, the enzyme responsible for lengthening telomeres and enhancing cellular longevity.\nStudies in mice and primates showed increased lifespan by up to 30% after Epithalon administration.\n\n2. Melatonin Production \u0026amp; Circadian Rhythm Regulation\n\nEpithalon enhances melatonin synthesis, improving sleep quality, mood regulation, and hormonal balance.\nRegulates the pineal gland, maintaining youthful endocrine function.\n\n3. Immune System \u0026amp; Cellular Protection\n\nIncreases T-cell activity and reduces age-related immune decline.\nProtects against oxidative stress by neutralizing free radicals, reducing inflammation, and supporting DNA repair.\nImproves mitochondrial function, enhancing cellular energy production (ATP synthesis).\n\n4. Cancer Prevention \u0026amp; DNA Repair\n\nEpithalon has been studied for its anti-cancer effects, showing anti-tumor activity by inhibiting cancer cell proliferation.\nPromotes DNA integrity, reducing age-related mutations.\n\nResearch \u0026amp; Clinical Applications\n1. Telomerase Activation \u0026amp; Lifespan Extension\n\nStudies in rats, mice, and primates show telomere elongation and up to 30% lifespan increase.\nResearch suggests potential anti-aging applications in humans.\n\n2. Neuroprotection \u0026amp; Cognitive Benefits\n\nEpithalon enhances neuronal plasticity, improving memory, focus, and cognitive function.\nProtects against neurodegeneration in models of Alzheimer’s and Parkinson’s disease.\n\n3. Cardiovascular Health\n\nReduces oxidative stress and arterial plaque formation, lowering the risk of heart disease and stroke.\nEnhances blood vessel function and prevents hypertension-related damage.\n\n4. Cancer Prevention \u0026amp; DNA Protection\n\nStudies in breast, colon, and lung cancer models show reduced tumor growth and improved immune surveillance.\nEpithalon has been studied as a potential co-therapy in oncology.\n\n5. Sleep Optimization \u0026amp; Hormonal Balance\n\nRestores melatonin secretion, improving sleep quality and circadian rhythm stability.\nHelps regulate cortisol and stress response, reducing age-related hormonal imbalances.\n\nStructure \u0026amp; Composition\n\nSequence: Ala-Glu-Asp-Gly\nMolecular Formula: C₁₄H₂₂N₄O₉\nMolecular Weight: 390.35 g\/mol\nCAS Number: 307297-39-8\n\nCitations \u0026amp; References\n\nKhavinson, V. et al. “Peptide regulation of aging: Epithalon as a geroprotector,” Biogerontology, 2011.\nAnisimov, V. N. et al. “Effect of Epithalon on telomerase activity and life span in mice,” Neuroendocrinology Letters, 2003.\nTurchaninova, M. F. et al. “Pineal peptides restore age-related melatonin decline in humans,” Clinical and Experimental Pharmacology and Physiology, 2015.\nKhavinson, V. et al. “Epithalon’s effect on telomere length and longevity in human cell cultures,” Rejuvenation Research, 2005.\nLustgarten, M. S. et al. “Mitochondrial dysfunction in aging: Role of oxidative stress and epithalon,” Nature Aging Research, 2019.\nDePinho, R. A. “The telomere theory of aging and its implications for human longevity,” Cell Metabolism, 2009.\nAnisimov, V. N. et al. “Epithalon as a potential anti-cancer therapy: Experimental models and clinical applications,” Oncology Reports, 2006.\n\nConclusion\nEpithalon is one of the most promising anti-aging peptides, backed by decades of research on telomere extension, mitochondrial function, immune support, and cancer prevention. Its ability to activate telomerase, regulate melatonin, and enhance DNA repair makes it a powerful candidate for longevity research, neuroprotection, and age-related disease prevention.","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989561020649,"sku":"PS036","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/EPITHALON-10MG-scaled.jpg?v=1778509483"},{"product_id":"oxytocin-acetate-2mg","title":"Oxytocin Acetate 2mg","description":"OVERVIEW\nOxytocin is a naturally occurring peptide hormone with vital roles in social bonding, reproduction, wound healing, and metabolic regulation. Produced primarily in the hypothalamus and released by the posterior pituitary gland, oxytocin also exists in various tissues, including the placenta, ovaries, testes, and even the retina.\nResearch suggests oxytocin may:\n\nEnhance social bonding and trust.\nFacilitate childbirth and breastfeeding.\nAccelerate wound healing and modulate immune function.\nImprove cardiovascular health by reducing blood pressure and atherosclerosis.\nEnhance insulin sensitivity and promote weight loss, making it a potential therapy for diabetes.\nBoost cognitive function and reduce symptoms of anxiety and depression.\n\nUnlike many other peptides, oxytocin functions as both a neuropeptide and a blood-borne hormone, influencing numerous physiological and psychological processes.\nRESEARCH\nOxytocin and Social Bonding\nOften referred to as the “love hormone,” oxytocin plays a critical role in:\n\nPair bonding and attachment in both men and women.\nTrust, empathy, and prosocial behavior.\nReducing social anxiety and promoting positive social interactions.\n\nIntranasal oxytocin administration has been shown to enhance social bonding and may help individuals with social anxiety disorders.\nOxytocin and Wound Healing\nOxytocin has demonstrated significant wound-healing properties:\n\nHigher oxytocin levels correlate with faster tissue regeneration.\nClinical studies show couples with higher oxytocin levels healed 40% faster from minor wounds.\nOxytocin modulates inflammatory cytokines (IL-6, TNF-α, IL-1β), reducing inflammation and promoting tissue repair.\n\nThese findings suggest potential therapeutic applications in post-surgical recovery and chronic wound management.\nOxytocin and Cardiovascular Health\nResearch indicates oxytocin may protect against heart disease by:\n\nLowering blood pressure and improving heart function.\nReducing atherosclerosis by increasing oxytocin receptor expression in blood vessels.\nProtecting against cardiomyopathy and ischemia-related damage in chronic heart conditions.\n\nThese findings suggest that oxytocin could play a preventive role in cardiovascular disease.\nOxytocin and Diabetes\n\nEnhances insulin sensitivity and glucose uptake, making it a potential therapy for type 2 diabetes.\nStudies in obese diabetic mice found that oxytocin reduced fasting glucose levels by 23% and total body fat by 19%.\nHuman trials suggest intranasal oxytocin therapy may promote weight loss over an 8-week period.\n\nOxytocin’s ability to regulate metabolism makes it a promising candidate for obesity and metabolic syndrome treatment.\nOxytocin and Cognitive Function\n\nSupports neurodevelopment and learning.\nMice deprived of maternal bonding exhibited cognitive deficits, which were reversed with oxytocin therapy.\nModulates serotonin and dopamine levels, suggesting potential antidepressant effects.\n\nOxytocin’s role in brain plasticity and emotional regulation makes it a candidate for neurological and psychiatric therapies.\nOxytocin and Anxiety\/Depression\n\nGenetic variations in the oxytocin receptor (OXTR) gene are linked to social anxiety disorder and borderline personality disorder.\nIntranasal oxytocin administration has been shown to reduce fear responses and enhance social interactions.\nPotential applications in PTSD and generalized anxiety disorder (GAD).\n\nThese findings suggest that oxytocin-based therapies could provide alternative treatments for mood and anxiety disorders.\nSTRUCTURE\n\nMolecular Formula: C₄₃H₆₆N₁₂O₁₂S₂\nMolecular Weight: 1007.193 g\/mol\nAmino Acid Sequence: Cys-Tyr-Ile-Gln-Asn-Cys-Pro-Leu-Gly\nCAS Registry Number: 50-56-6\nPubChem Identifier: 439302\n\nCITATIONS\n\nGouin, J.-P. et al. Marital behavior, oxytocin, vasopressin, and wound healing. Psychoneuroendocrinology (2010).\nKiecolt-Glaser, J. K. et al. Hostile marital interactions, proinflammatory cytokine production, and wound healing. Archives of General Psychiatry (2005).\nReiss, A. B. et al. Oxytocin: Potential to mitigate cardiovascular risk. Peptides (2019).\nWang, P. et al. Therapeutic potential of oxytocin in atherosclerotic cardiovascular disease: Mechanisms and signaling pathways. Frontiers in Neuroscience (2019).\nJankowski, M. et al. Oxytocin and cardioprotection in diabetes and obesity. BMC Endocrine Disorders (2016).\nPlante, E. et al. Oxytocin treatment prevents cardiomyopathy observed in obese diabetic male mice.Endocrinology (2015).\nKolukcu, E. et al. The effects of oxytocin on penile tissues in an experimental priapism model in rats.International Urology and Nephrology (2019).\nDing, C. et al. Oxytocin in metabolic homeostasis: Implications for obesity and diabetes management. Obesity Reviews (2019).\nAltirriba, J. et al. Divergent effects of oxytocin treatment on adiposity and diabetes in obese mice.Endocrinology (2014).\nBarengolts, E. Oxytocin as an emerging treatment for obesity and dysglycemia: A review of randomized controlled trials and cohort studies. Endocrine Practice (2019).","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989561053417,"sku":"PS035","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/Oxytocin-2mg-scaled.jpg?v=1778509484"},{"product_id":"semax-5mg","title":"Semax 5mg","description":"OVERVIEW\nSemax is a synthetic derivative of adrenocorticotropic hormone (ACTH), originally developed in Russia for its neuroprotective, cognitive-enhancing, and immune-boosting properties. Research suggests that Semax may:\n\nIncrease brain-derived neurotrophic factor (BDNF), supporting neuroplasticity and learning.\nEnhance immune function and reduce inflammation.\nProtect neurons from ischemic damage, aiding in stroke and traumatic brain injury (TBI) recovery.\nImprove cardiovascular function, potentially enhancing blood circulation.\nAct as an antidepressant and anxiolytic (anti-anxiety agent) by modulating serotonin, dopamine, and BDNF levels.\n\nDue to its ability to support mental performance, reduce stress, and promote neurological recovery, Semax is widely researched in neurology, cognitive enhancement, and immune regulation.\nRESEARCH\nSemax and Stroke Recovery\nSemax has been extensively studied for its neuroprotective effects in stroke and traumatic brain injury (TBI).\n\nIn rat models of stroke, Semax activated 24 genes linked to vascular function, promoting neurogenesis and protecting against ischemic damage.\nClinical trials in stroke patients showed that those receiving Semax experienced faster motor function recovery and improved neurological outcomes.\nBDNF elevation facilitated brain plasticity and functional rehabilitation.\n\nThese findings suggest Semax could be a promising treatment for post-stroke recovery and neurodegenerative conditions.\nSemax and Cognitive Enhancement\nSemax has been studied for its potential as a nootropic (cognitive enhancer):\n\nIncreases BDNF levels in the hippocampus, improving learning and memory formation.\nEnhances prefrontal cortex activity, potentially boosting problem-solving skills and attention span.\nProtects against cognitive decline, possibly preventing age-related memory loss.\n\nThese neuroprotective and cognitive-enhancing effects make Semax a promising compound for cognitive longevity and brain health.\nSemax and the Default Mode Network (DMN)\n\nFunctional MRI studies suggest that Semax activates the Default Mode Network (DMN), a system responsible for:\n\nSocial cognition\nEnvironmental awareness\nResting-state brain activity\n\n\nEnhanced DMN function is linked to better focus, cognitive flexibility, and mental clarity.\nDMN disruptions are associated with neurodegenerative diseases like Alzheimer’s, suggesting Semax may play a neuroprotective role.\n\nSemax and Depression\/Anxiety\nSemax may offer antidepressant and anxiolytic effects by modulating serotonin, dopamine, and BDNF levels.\n\nAnimal studies show that Semax increases BDNF production, reducing depressive symptoms.\nUnlike SSRIs, Semax may work faster by directly increasing neurotrophic support, rather than altering serotonin levels alone.\nPotential as an adjunct therapy with antidepressants for enhanced efficacy.\n\nSemax and Pain Management\nResearch suggests Semax may help regulate pain perception by influencing the opioid system:\n\nPrevents the breakdown of enkephalins, the brain’s natural painkillers.\nMay provide an alternative approach for chronic pain management.\n\nSemax and Immune System Support\n\nSuppresses IL-6, a key inflammatory cytokine involved in stress response and immune dysfunction.\nEnhances T-helper cell activity, leading to better immune system balance.\nPotential antiviral properties, suggesting possible applications in infection recovery.\n\nSTRUCTURE\n\nMolecular Formula: C₃₉H₅₄N₁₀O₁₀S\nMolecular Weight: 854.99 g\/mol\nAmino Acid Sequence: Ac-Met-Glu-His-Phe-Pro-Gly-Pro\nCAS Registry Number: 80714-61-0\nPubChem Identifier: 122178\nSynonyms: Pro-Gly-Pro-ACTH, ACTH(4-10) analog\n\nCITATIONS\n\nLebedeva, I. S. et al. Effects of Semax on the Default Mode Network of the Brain. Bulletin of Experimental Biology and Medicine (2018).\nMars, R. B. et al. On the relationship between the default mode network and the social brain. Front. Hum. Neurosci. (2012).\nMedvedeva, E. V. et al. The peptide Semax affects the expression of genes related to the immune and vascular systems in rat brain focal ischemia. BMC Genomics (2014).\nGusev, E. I. et al. The efficacy of Semax in the treatment of patients at different stages of ischemic stroke.Zhurnal Nevrologii i Psikhiatrii (2018).\nAgapova, T. I. et al. Effect of Semax on BDNF and NGF gene expression in the rat hippocampus and frontal cortex. Molecular Genetics, Microbiology, and Virology (2008).\nScantlebury, M. H. et al. Adrenocorticotropic Hormone Protects Learning and Memory Function in Epileptic Mice. Neuroscience Letters (2017).\nDeltheil, T. et al. Behavioral and serotonergic consequences of increasing hippocampus BDNF protein levels.Neuropharmacology (2008).\nBobyntsev, I. I. et al. Influence of ACTG4-7-PGP (Semax) on the morphofunctional state of hepatocytes in chronic stress. Bulletin of Experimental Biology and Medicine (2017).\nBobyntsev, I. I. et al. The effect of ACTH-4-7-PGP peptide on lipid peroxidation and stress response in rats.Research Gate (2015).","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989561086185,"sku":"PS034","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/Semax-5mg-scaled_fb250cfd-4a21-40b2-af2e-f88fe1d94810.jpg?v=1778509485"},{"product_id":"selank-5mg","title":"Selank 5mg","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nSelank is a synthetic analogue of tuftsin, a naturally occurring immunomodulatory peptide. Originally developed in Russia, it has been extensively studied for its anxiolytic (anti-anxiety), nootropic (cognitive-enhancing), and immune-boosting properties.\u003cbr\u003e\nResearch suggests that Selank may:\u003c\/p\u003e\n\n\u003cp\u003eReduce anxiety without causing sedation or addiction.\u003cbr\u003e\nEnhance memory, learning, and cognitive function.\u003cbr\u003e\nModulate immune system responses, aiding in stress-induced immune dysfunction.\u003cbr\u003e\nRegulate inflammatory cytokines, particularly by suppressing IL-6, a key marker of inflammation.\u003cbr\u003e\nIncrease brain-derived neurotrophic factor (BDNF), supporting neuroplasticity and brain health.\u003c\/p\u003e\n\n\u003cp\u003eDue to its effects on the GABAergic system, Selank is considered a potential alternative to benzodiazepines for treating anxiety-related disorders—without the risk of dependence or withdrawal.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\nSelank and Anxiety Reduction\u003cbr\u003e\nSelank interacts with the GABAergic system, similar to benzodiazepines, but with key distinctions:\u003c\/p\u003e\n\n\u003cp\u003eEnhances GABA receptor sensitivity, promoting a calming effect.\u003cbr\u003e\nDoes not cause dependence, withdrawal symptoms, or cognitive impairment, unlike traditional anxiolytics.\u003cbr\u003e\nIncreases the effectiveness of diazepam (Valium) when combined, suggesting a synergistic mechanism.\u003c\/p\u003e\n\n\u003cp\u003eClinical trials conducted in Russia demonstrated that Selank:\u003c\/p\u003e\n\n\u003cp\u003eSignificantly reduced anxiety and improved mood in patients with generalized anxiety disorder (GAD) and neurasthenia.\u003c\/p\u003e\n\n\u003cp\u003eThese findings highlight Selank’s potential for treating anxiety disorders without the sedation or addiction risksassociated with benzodiazepines.\u003cbr\u003e\nSelank and Cognitive Enhancement\u003cbr\u003e\nSelank has been shown to improve learning, memory retention, and recall through multiple mechanisms:\u003c\/p\u003e\n\n\u003cp\u003eEnhances hippocampal gene expression, supporting neuroplasticity.\u003cbr\u003e\nIncreases neuropeptides that promote long-term memory formation.\u003cbr\u003e\nProtects against cognitive decline after brain injury or stress exposure.\u003c\/p\u003e\n\n\u003cp\u003eAnimal studies indicate that Selank enhances cognitive function even in subjects without anxiety, suggesting direct nootropic effects.\u003cbr\u003e\nSelank and Immune System Modulation\u003cbr\u003e\nSelank is unique among nootropic peptides due to its immune-modulating properties:\u003c\/p\u003e\n\n\u003cp\u003eSuppresses IL-6, a pro-inflammatory cytokine associated with anxiety and depression.\u003cbr\u003e\nRegulates T-helper cell activity, helping to maintain immune balance.\u003cbr\u003e\nDemonstrates antiviral potential, possibly aiding recovery from respiratory infections.\u003c\/p\u003e\n\n\u003cp\u003eThese findings suggest that Selank may benefit individuals with stress-related immune suppression.\u003cbr\u003e\nSelank and Pain Perception\u003cbr\u003e\nSelank influences the endogenous opioid system by:\u003c\/p\u003e\n\n\u003cp\u003ePreventing the breakdown of enkephalins, the body’s natural pain-killing peptides.\u003cbr\u003e\nReducing stress-related hyperalgesia, a condition where anxiety or chronic stress amplifies pain sensitivity.\u003c\/p\u003e\n\n\u003cp\u003eThis mechanism mirrors the effects of opioid receptor modulators and certain antidepressants, making Selank a potential alternative treatment for chronic pain conditions.\u003cbr\u003e\nSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eMolecular Formula: C₃₃H₅₇N₁₁O₉\u003cbr\u003e\nMolecular Weight: 751.887 g\/mol\u003cbr\u003e\nAmino Acid Sequence: Thr-Lys-Pro-Arg-Pro-Gly-Pro\u003cbr\u003e\nCAS Registry Number: 129954-34-3\u003cbr\u003e\nPubChem Identifier: 11765600\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003c\/p\u003e\n\n\u003cp\u003eVolkova, A. et al. Selank Administration Affects the Expression of Some Genes Involved in GABAergic Neurotransmission. Front. Pharmacol. (2016).\u003cbr\u003e\nKasian, A. et al. Peptide Selank Enhances the Effect of Diazepam in Reducing Anxiety in Unpredictable Chronic Mild Stress Conditions in Rats. Behavioural Neurology (2017).\u003cbr\u003e\nZozulya, A. A. et al. The inhibitory effect of Selank on enkephalin-degrading enzymes as a possible mechanism of its anxiolytic activity. Bull. Exp. Biol. Med. (2001).\u003cbr\u003e\nSokolov, O. Y. et al. Effects of Selank on behavioral reactions and activities of plasma enkephalin-degrading enzymes in mice with different phenotypes of emotional and stress reactions. Bull. Exp. Biol. Med. (2003).\u003cbr\u003e\nUchakina, O. N. et al. Immunomodulatory effects of Selank in patients with anxiety-asthenic disorders. Zh. Nevrol. Psikhiatr. (2008).\u003cbr\u003e\nKolomin, T. et al. Expression of inflammation-related genes in mouse spleen under tuftsin analog Selank. Regul. Pept. (2011).\u003cbr\u003e\nSemenova, T. et al. Effect of Selank on cognitive processes after damage inflicted to the cerebral catecholamine system during early ontogeny. Bull. Exp. Biol. Med. (2014).\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989561118953,"sku":"PS033","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/Selank-5mg-scaled.jpg?v=1778509485"},{"product_id":"dsip-5mg","title":"DSIP 5mg","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nDelta Sleep-Inducing Peptide (DSIP) is a naturally occurring neuropeptide, first discovered in 1977, that plays a role in sleep regulation, stress response, and metabolic function. Although initially studied for its sleep-promoting effects, research suggests that DSIP has a wide range of physiological and endocrine functions beyond sleep.\u003cbr\u003e\nStudies indicate that DSIP may:\u003c\/p\u003e\n\n\u003cp\u003eReduce oxidative stress and support mitochondrial function.\u003cbr\u003e\nImprove heart function by normalizing myocardial contractility.\u003cbr\u003e\nRegulate stress response and lower cortisol levels.\u003cbr\u003e\nModulate pain perception and opioid receptor activity.\u003cbr\u003e\nInfluence blood pressure and metabolic processes.\u003cbr\u003e\nShow potential in treating major depressive disorder (MDD).\u003c\/p\u003e\n\n\u003cp\u003eWhile DSIP was originally thought to induce sleep, research has produced mixed results, with some studies confirming sleep benefits and others suggesting it plays a regulatory role rather than directly causing sedation.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\nDSIP and Sleep Regulation\u003c\/p\u003e\n\n\u003cp\u003eDSIP was named for its ability to induce sleep in rabbits.\u003cbr\u003e\nHuman studies have shown inconsistent results—some report improved sleep quality, while others found no direct sedative effect.\u003cbr\u003e\nDSIP may help regulate sleep cycles rather than act as a traditional sedative.\u003cbr\u003e\nEEG studies have shown both increased slow-wave sleep and no effect, indicating complex sleep interactions.\u003cbr\u003e\nDSIP is being researched for its potential in managing chronic insomnia.\u003c\/p\u003e\n\n\u003cp\u003eDSIP and Pain Modulation\u003c\/p\u003e\n\n\u003cp\u003eAnimal studies show that DSIP reduces pain sensitivity.\u003cbr\u003e\nActs on central opioid receptors, providing analgesic effects without opioid dependence.\u003cbr\u003e\nHuman trials suggest DSIP may help reduce chronic pain perception and ease opioid withdrawal symptoms.\u003cbr\u003e\nDSIP appears to influence serotonin and dopamine activity, which are critical for pain regulation.\u003c\/p\u003e\n\n\u003cp\u003eDSIP and Stress Reduction\u003c\/p\u003e\n\n\u003cp\u003eDSIP has been shown to lower cortisol levels, helping to modulate the body’s stress response.\u003cbr\u003e\nResearch suggests DSIP inhibits somatostatin, a peptide involved in stress and hormone regulation.\u003cbr\u003e\nAnimal models of chronic stress indicate DSIP helps normalize metabolic disruptions.\u003cbr\u003e\nPotential applications in PTSD and anxiety disorders are under investigation.\u003c\/p\u003e\n\n\u003cp\u003eDSIP and Metabolic Regulation\u003c\/p\u003e\n\n\u003cp\u003eDSIP may support mitochondrial function, protecting against oxidative stress and metabolic dysfunction.\u003cbr\u003e\nResearch suggests DSIP helps prevent metabolic damage during stroke and heart attacks by maintaining cellular energy production.\u003c\/p\u003e\n\n\u003cp\u003eDSIP and Depression Treatment\u003c\/p\u003e\n\n\u003cp\u003eLow DSIP levels have been observed in individuals with major depressive disorder (MDD).\u003cbr\u003e\nDSIP influences monoamine oxidase type A (MAO-A) and serotonin, both key targets for depression treatment.\u003cbr\u003e\nEarly studies suggest DSIP may help stabilize mood and reduce suicidal tendencies.\u003c\/p\u003e\n\n\u003cp\u003eDSIP and Withdrawal Management\u003c\/p\u003e\n\n\u003cp\u003eDSIP has been studied for its potential to ease withdrawal symptoms in alcohol and opioid detoxification.\u003cbr\u003e\nStudy in 107 patients found:\u003c\/p\u003e\n\n\u003cp\u003e97% of alcohol-dependent patients showed significant improvement.\u003cbr\u003e\n87% of opioid-dependent patients experienced substantial symptom relief.\u003c\/p\u003e\n\n\n\u003cp\u003eMore frequent dosing was required for opioid withdrawal, but overall results were promising.\u003c\/p\u003e\n\n\u003cp\u003eDSIP and Cancer Prevention\u003c\/p\u003e\n\n\u003cp\u003eAnimal studies suggest a 2.6-fold reduction in tumor development with DSIP treatment.\u003cbr\u003e\nDSIP-treated mice exhibited a 22.6% reduction in chromosomal abnormalities, suggesting potential anti-cancer properties.\u003cbr\u003e\nFurther research is needed to determine whether DSIP directly prevents cancer or if its benefits are due to overall stress reduction and metabolic regulation.\u003c\/p\u003e\n\n\u003cp\u003eSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eMolecular Formula: C₃₅H₄₈N₁₀O₁₅\u003cbr\u003e\nMolecular Weight: 848.824 g\/mol\u003cbr\u003e\nAmino Acid Sequence: Trp-Ala-Gly-Gly-Asp-Ala-Ser-Gly-Glu\u003cbr\u003e\nCAS Registry Number: 62568-57-4\u003cbr\u003e\nPubChem Identifier: 68816\u003cbr\u003e\nSynonyms: Emideltide, DSIP nonapeptide, Deltaran\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003c\/p\u003e\n\n\u003cp\u003eGraf M., Christen H., Schoenenberger G. DSIP\/DSIP-P and circadian motor activity of rats under continuous light. Peptides (1982).\u003cbr\u003e\nYehuda S., Kastin A. J., Coy D. H. Thermoregulatory and locomotor effects of DSIP: Paradoxical interaction with d-amphetamine. Pharmacology Biochemistry and Behavior (1980).\u003cbr\u003e\nSinyukhin A. B. et al. Delta sleep-inducing peptide analogue corrects CNS functional state in children treated with chemotherapy. Eur. Neuropsychopharmacol. (2009).\u003cbr\u003e\nKoplik E. V. et al. Delta sleep-inducing peptide and Deltaran: Potential approaches to antistress protection.Neurosci. Behav. Physiol. (2008).\u003cbr\u003e\nFeller L., Khammissa R. A. G., Lemmer J. Basal cell carcinoma, squamous cell carcinoma, and melanoma of the head and face. Head \u0026amp; Face Medicine (2016).\u003cbr\u003e\nMcMillan T. R. et al. Melanotan II, a melanocortin agonist, partially rescues the impaired thermogenic capacity of pituitary adenylate cyclase-activating polypeptide deficient mice. Exp. Physiol. (2021).\u003cbr\u003e\nJetté L. et al. Effect of DSIP-containing preparation Deltaran on biomarkers of aging, lifespan, and spontaneous tumor incidence in mice. PubMed (2005).\u003cbr\u003e\nWessells H., Levine L. A. Evaluation of the efficacy of DSIP in psychogenic erectile dysfunction. J. Sex Med. (2012).\u003cbr\u003e\nFowkes M. M. et al. Peptidomimetic DSIP derivatives for PET imaging. Eur. J. Med. Chem. (2018).\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989561151721,"sku":"PS032","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/DSIP-5MG-scaled.jpg?v=1778509486"},{"product_id":"pt-141-10mg","title":"PT-141 10mg","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nPT-141, also known as Bremelanotide, is a synthetic derivative of alpha-melanocyte-stimulating hormone (α-MSH). Initially developed to treat sexual dysfunction in both men and women, it has also been explored for its immune-modulating properties and potential applications in hemorrhagic shock treatment.\u003cbr\u003e\nPT-141 functions as a melanocortin receptor (MC) agonist, primarily targeting MC-4R and MC-1R, which play roles in sexual arousal, immune function, and vascular regulation. Research suggests that PT-141 can:\u003c\/p\u003e\n\n\u003cp\u003eEnhance libido and sexual arousal.\u003cbr\u003e\nImprove erectile function in men and increase sexual desire in women.\u003cbr\u003e\nStimulate immune system activity.\u003cbr\u003e\nReduce inflammation and ischemic damage.\u003c\/p\u003e\n\n\u003cp\u003eUnlike traditional PDE5 inhibitors (e.g., Viagra, Cialis), which work by increasing blood flow, PT-141 acts directly on the central nervous system, making it a potential alternative for individuals with psychogenic sexual dysfunction.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\nPT-141 and Sexual Arousal\u003cbr\u003e\nPT-141’s impact on sexual function is linked to its activation of the MC-4R receptor, found in the central nervous system and associated with sexual behavior and arousal.\u003c\/p\u003e\n\n\u003cp\u003eAnimal studies showed that MC-4R activation increased mating behavior in both male and female subjects.\u003cbr\u003e\nClinical trials in men with erectile dysfunction (ED)—including those unresponsive to sildenafil (Viagra)—revealed that one-third of participants achieved sufficient erections with PT-141.\u003cbr\u003e\nPre-menopausal women with hypoactive sexual desire disorder (HSDD) experienced:\u003c\/p\u003e\n\n\u003cp\u003eIncreased frequency of satisfying sexual events.\u003cbr\u003e\nReduced distress related to low sexual desire.\u003c\/p\u003e\n\n\n\n\u003cp\u003eBecause PT-141 acts via the brain and not the vascular system, it may be particularly effective for non-vascular causes of sexual dysfunction.\u003cbr\u003e\nPT-141 and Hemorrhagic Shock\u003cbr\u003e\nIn 2009, PT-141 was investigated for acute hemorrhage treatment, with research suggesting potential emergency medicine applications.\u003c\/p\u003e\n\n\u003cp\u003eThe MC-1R receptor plays a key role in vascular response and blood flow regulation.\u003cbr\u003e\nStudies indicated that PT-141 enhanced vascular integrity and reduced ischemic damage in hemorrhagic shock models.\u003c\/p\u003e\n\n\u003cp\u003eThese findings suggest PT-141 or its derivatives could be explored for trauma and critical care medicine.\u003cbr\u003e\nPT-141 and Immune System Modulation\u003cbr\u003e\nPT-141 also targets the MC-1R receptor, which is involved in immune regulation.\u003c\/p\u003e\n\n\u003cp\u003eAnimal studies suggest PT-141 may exhibit:\u003c\/p\u003e\n\n\u003cp\u003eAnti-inflammatory properties.\u003cbr\u003e\nAntimicrobial effects, potentially aiding in infection control.\u003c\/p\u003e\n\n\n\u003cp\u003eMC-1R activation has been linked to immune system support in autoimmune conditions and inflammatory diseases.\u003c\/p\u003e\n\n\u003cp\u003eThis research highlights PT-141’s potential beyond sexual health, particularly in immune and inflammatory disorders.\u003cbr\u003e\nPT-141 and Cancer Research\u003cbr\u003e\nRecent studies suggest that PT-141 may influence DNA repair mechanisms:\u003c\/p\u003e\n\n\u003cp\u003eThe MC-1R receptor is involved in DNA damage repair pathways.\u003cbr\u003e\nIndividuals with MC-1R mutations have a higher risk of melanoma and skin cancer.\u003c\/p\u003e\n\n\u003cp\u003eOngoing research aims to determine whether PT-141 could help prevent certain cancers or support DNA repair in high-risk individuals.\u003cbr\u003e\nSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eMolecular Formula: C₅₀H₆₈N₁₄O₁₀\u003cbr\u003e\nMolecular Weight: 1025.182 g\/mol\u003cbr\u003e\nAmino Acid Sequence: Ac-Nle-Asp(1)-His-D-Phe-Arg-Trp-Lys(1)\u003cbr\u003e\nCAS Registry Number: 189691-06-3\u003cbr\u003e\nPubChem Identifier: 9941379\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003c\/p\u003e\n\n\u003cp\u003eRosen, R. C. et al. Evaluation of the safety, pharmacokinetics, and pharmacodynamic effects of subcutaneously administered PT-141, a melanocortin receptor agonist, in healthy male subjects and in patients with an inadequate response to Viagra. International Journal of Impotence Research (2004).\u003cbr\u003e\nWessells, H. et al. PT-141 induces penile erection via brain and spinal melanocortin receptors. Neuroscience (2003).\u003cbr\u003e\nRössler, A.-S. et al. The melanocortin agonist, Melanotan II, enhances proceptive sexual behaviors in female rats. Pharmacology, Biochemistry, and Behavior (2006).\u003cbr\u003e\nSafarinejad, M. R. \u0026amp; Hosseini, S. Y. Salvage of sildenafil failures with bremelanotide: a randomized, double-blind, placebo-controlled study. Journal of Urology (2008).\u003cbr\u003e\nClayton, A. H. et al. Bremelanotide for female sexual dysfunctions in premenopausal women: a randomized, placebo-controlled dose-finding trial. Women’s Health (London, England) (2016).\u003cbr\u003e\nJi, H. et al. The Synthetic Melanocortin (CKPV)2 Exerts Anti-Fungal and Anti-Inflammatory Effects against Candida albicans Vaginitis via Inducing Macrophage M2 Polarization. PLoS ONE (2013).\u003cbr\u003e\nMaresca, V. et al. Skin phototype: a new perspective. Pigment Cell \u0026amp; Melanoma Research (2015).\u003cbr\u003e\nFeller, L. et al. Basal cell carcinoma, squamous cell carcinoma, and melanoma of the head and face. Head \u0026amp; Face Medicine (2016).\u003cbr\u003e\nMcMillan, T. R. et al. Melanotan II, a melanocortin agonist, partially rescues the impaired thermogenic capacity of pituitary adenylate cyclase-activating polypeptide deficient mice. Experimental Physiology (2021).\u003cbr\u003e\nSpana, C. et al. Effect of bremelanotide on body weight of obese women: Data from two phase 1 randomized controlled trials. Diabetes, Obesity, and Metabolism (2022).\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989561184489,"sku":"PS031","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/PT-141-10mg-scaled.jpg?v=1778509487"},{"product_id":"aod-9604-5mg","title":"AOD-9604 5mg","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nAOD9604 is a modified fragment of human growth hormone (hGH), specifically derived from hGH 176-191. It was originally developed as an anti-obesity agent and has been extensively studied for its fat-burning (lipolytic) properties.\u003cbr\u003e\nResearch indicates that AOD9604:\u003c\/p\u003e\n\n\u003cp\u003eStimulates fat breakdown (lipolysis).\u003cbr\u003e\nPrevents new fat accumulation (lipogenesis).\u003cbr\u003e\nDoes not increase IGF-1 or insulin levels, making it distinct from full-length hGH.\u003cbr\u003e\nDoes not trigger antibody production, suggesting potential long-term safety.\u003c\/p\u003e\n\n\u003cp\u003eBeyond fat metabolism, AOD9604 has been explored for its potential benefits in:\u003c\/p\u003e\n\n\u003cp\u003eHeart disease\u003cbr\u003e\nOsteoarthritis and cartilage repair\u003cbr\u003e\nMetabolic syndrome\u003c\/p\u003e\n\n\u003cp\u003eDue to these properties, AOD9604 has gained attention as a research candidate in weight management and regenerative medicine.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\nAOD9604 and Fat Loss\u003cbr\u003e\nAOD9604 was initially developed to replicate the fat-burning effects of hGH without affecting other hormones.\u003c\/p\u003e\n\n\u003cp\u003eClinical Trials: A 12-week Phase 2b clinical trial conducted in Australia tested AOD9604 on 300 obese individuals.\u003cbr\u003e\nResults:\u003c\/p\u003e\n\n\u003cp\u003eParticipants receiving AOD9604 experienced three times more weight loss than the placebo group.\u003cbr\u003e\nConsistent fat reduction was observed throughout the study.\u003c\/p\u003e\n\n\n\u003cp\u003eMetabolic Safety:\u003c\/p\u003e\n\n\u003cp\u003eUnlike full-length hGH, AOD9604 does not elevate IGF-1 or insulin levels.\u003cbr\u003e\nThis makes it a potential safer alternative for individuals at risk of insulin resistance or diabetes.\u003c\/p\u003e\n\n\n\n\u003cp\u003eAOD9604: Mechanism Beyond Beta-3 Adrenergic Receptors\u003cbr\u003e\nEarly research suggested that AOD9604 works by activating beta-3 adrenergic receptors in fat cells, shifting them into fat-burning mode. However, experiments with genetically modified mice lacking these receptors still showed significant fat loss when administered AOD9604.\u003cbr\u003e\nThis suggests that AOD9604 may function through alternative pathways, possibly by:\u003c\/p\u003e\n\n\u003cp\u003eInducing apoptosis (programmed cell death) in white fat cells.\u003cbr\u003e\nRegulating lipid metabolism at the mitochondrial level, increasing energy expenditure.\u003c\/p\u003e\n\n\u003cp\u003eAOD9604 and Joint Health\u003cbr\u003e\nAnimal studies indicate that AOD9604 may have chondroprotective (cartilage-protecting) effects:\u003c\/p\u003e\n\n\u003cp\u003eIn osteoarthritis models (rats \u0026amp; rabbits):\u003c\/p\u003e\n\n\u003cp\u003eDirect intra-articular injections of AOD9604 improved joint function and reduced pain.\u003cbr\u003e\nHistological analysis revealed increased cartilage thickness and better structural integrity.\u003c\/p\u003e\n\n\n\u003cp\u003eSynergistic Effects:\u003c\/p\u003e\n\n\u003cp\u003eWhen combined with other osteoarthritis treatments, AOD9604 enhanced joint repair and functionbeyond standalone therapies.\u003c\/p\u003e\n\n\n\n\u003cp\u003eThese findings suggest potential applications in cartilage regeneration and osteoarthritis management.\u003cbr\u003e\nAOD9604 and Cardiovascular Health\u003cbr\u003e\nWhile fat reduction is inherently linked to heart health, studies suggest AOD9604 may provide direct metabolic benefits independent of weight loss.\u003c\/p\u003e\n\n\u003cp\u003eResearch indicates AOD9604 may enhance heart function in obese individuals.\u003cbr\u003e\nSimilar to metabolic drugs such as pioglitazone and acipimox, AOD9604 improves lipid metabolism, reducing cardiovascular risk factors.\u003c\/p\u003e\n\n\u003cp\u003eAOD9604 Safety Profile\u003cbr\u003e\nAOD9604 has demonstrated a strong safety profile in clinical research:\u003c\/p\u003e\n\n\u003cp\u003eNo significant adverse effects were reported in human trials.\u003cbr\u003e\nDoes not increase blood sugar, IGF-1, or insulin resistance.\u003cbr\u003e\nHighly bioavailable in both oral and subcutaneous forms.\u003c\/p\u003e\n\n\u003cp\u003eImportant Note:\u003cbr\u003e\nAOD9604 is currently for research use only and not approved for human consumption.\u003cbr\u003e\nSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eMolecular Formula: C₇₈H₁₂₃N₂₃O₂₃S₂\u003cbr\u003e\nMolecular Weight: 1815.12 g\/mol\u003cbr\u003e\nAmino Acid Sequence: Tyr-Leu-Arg-Ile-Val-Gln-Cys-Arg-Ser-Val-Glu-Gly-Ser-Cys-Gly-Phe\u003cbr\u003e\nDisulfide Bridge: Cys⁷-Cys¹⁵\u003cbr\u003e\nCAS Registry Number: 386264-39-7\u003cbr\u003e\nPubChem Identifier: 16131447\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003c\/p\u003e\n\n\u003cp\u003eNg F. M. et al. Metabolic studies of a synthetic lipolytic domain (AOD9604) of human growth hormone. Horm. Res. (2000).\u003cbr\u003e\nStier H. et al. Safety and Tolerability of the Hexadecapeptide AOD9604 in Humans. Journal of Endocrinology and Metabolism (2013).\u003cbr\u003e\nHeffernan M. et al. Effects of Human GH and Its Lipolytic Fragment (AOD9604) on Lipid Metabolism in Obese Mice. Endocrinology (2001).\u003cbr\u003e\nZieba R. Obesity: A review of anti-obesity drugs in clinical development. Postepy Hig. Med. Dosw. (2007).\u003cbr\u003e\nKwon D. R. \u0026amp; Park G. Y. Effect of intra-articular injection of AOD9604 on osteoarthritis in rabbits. Ann. Clin. Lab. Sci. (2015).\u003cbr\u003e\nJensen M. D. Potential role of new therapies in modifying cardiovascular risk in overweight patients with metabolic risk factors. Obesity (Silver Spring) (2006).\u003cbr\u003e\nNews-Medical.net Obesity drug AOD9604 highly successful in trials. (2004).\u003cbr\u003e\nCase Western Reserve University Molecular biology of peptide-derived fat loss agents. J. Mol. Endocrinol. (2015).\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989561217257,"sku":"PS030","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/AOD-9604-5MG-scaled.jpg?v=1778509489"},{"product_id":"ipamorelin-10mg","title":"Ipamorelin 10mg","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nIpamorelin is a highly selective growth hormone secretagogue (GHS) that binds specifically to the ghrelin\/growth hormone secretagogue receptor (GHS-R1a). It is one of the most targeted GH secretagogues, meaning it does notaffect levels of ACTH, prolactin, follicle-stimulating hormone (FSH), luteinizing hormone (LH), thyroid-stimulating hormone (TSH), or cortisol.\u003cbr\u003e\nDue to its high receptor specificity, Ipamorelin has been widely studied for therapeutic applications and as a model peptide for exploring GH receptor selectivity. It plays a key role in growth, tissue repair, and metabolic regulation.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\nIpamorelin and Growth Hormone Release\u003cbr\u003e\nIpamorelin mimics ghrelin, the body’s natural hunger hormone, by binding to GHS-R1a receptors. This interaction leads to:\u003c\/p\u003e\n\n\u003cp\u003eA natural increase in growth hormone (GH) levels.\u003cbr\u003e\nImproved muscle growth, fat metabolism, and tissue repair.\u003cbr\u003e\nPreserved GH pulse frequency without overstimulating the endocrine system.\u003c\/p\u003e\n\n\u003cp\u003eUnlike other GH secretagogues, Ipamorelin stimulates GH release without causing significant increases in cortisol or prolactin levels, making it a safer option for long-term use.\u003cbr\u003e\nIpamorelin and Corticosteroid Side Effects\u003cbr\u003e\nGlucocorticoids (steroids) are often used to treat inflammatory diseases and cancer, but long-term use can lead to:\u003c\/p\u003e\n\n\u003cp\u003eMuscle wasting\u003cbr\u003e\nLoss of bone density\u003cbr\u003e\nIncreased visceral fat accumulation\u003c\/p\u003e\n\n\u003cp\u003eStudies suggest that Ipamorelin may counteract these side effects by:\u003c\/p\u003e\n\n\u003cp\u003eReducing muscle breakdown.\u003cbr\u003e\nPreventing bone loss associated with prolonged steroid use.\u003cbr\u003e\nPreserving nitrogen balance, which is crucial for muscle maintenance and recovery.\u003c\/p\u003e\n\n\u003cp\u003eIpamorelin and Bone Health\u003cbr\u003e\nChronic steroid use is linked to osteoporosis and increased fracture risk. Research in animal models has demonstrated that Ipamorelin can:\u003c\/p\u003e\n\n\u003cp\u003ePrevent bone density loss.\u003cbr\u003e\nStimulate osteoblast activity, promoting new bone formation.\u003c\/p\u003e\n\n\u003cp\u003eThese findings suggest Ipamorelin may have therapeutic applications in conditions like osteoporosis and age-related bone deterioration.\u003cbr\u003e\nIpamorelin and Muscle Growth\u003cbr\u003e\nIpamorelin’s ability to enhance protein synthesis makes it beneficial for:\u003c\/p\u003e\n\n\u003cp\u003eIncreasing muscle mass and improving recovery.\u003cbr\u003e\nPreventing muscle wasting conditions, such as cachexia and sarcopenia.\u003cbr\u003e\nSupporting post-surgical healing and injury rehabilitation.\u003c\/p\u003e\n\n\u003cp\u003eBy promoting muscle hypertrophy and repair, Ipamorelin has gained interest in sports medicine and anti-aging research.\u003cbr\u003e\nIpamorelin and Diabetes Research\u003cbr\u003e\nStudies suggest that Ipamorelin can stimulate insulin release by activating calcium channels in pancreatic islet cells. This mechanism may:\u003c\/p\u003e\n\n\u003cp\u003eImprove insulin sensitivity.\u003cbr\u003e\nAid in blood sugar regulation.\u003c\/p\u003e\n\n\u003cp\u003eThese findings indicate that Ipamorelin may be explored as a potential treatment for diabetes.\u003cbr\u003e\nIpamorelin and Post-Operative Ileus (POI)\u003cbr\u003e\nPost-operative ileus (POI) occurs when the digestive tract temporarily stops functioning after surgery, delaying recovery.\u003cbr\u003e\nClinical trials have shown that Ipamorelin:\u003c\/p\u003e\n\n\u003cp\u003eReduces POI symptoms.\u003cbr\u003e\nShortens hospital stays by an average of 12 hours.\u003c\/p\u003e\n\n\u003cp\u003eAlthough initial results were promising, further research on Ipamorelin for POI treatment was discontinued due to insufficient efficacy in large-scale trials.\u003cbr\u003e\nIpamorelin and PET Imaging\u003cbr\u003e\nAs a potent ghrelin receptor agonist, Ipamorelin has potential applications in diagnostic imaging:\u003c\/p\u003e\n\n\u003cp\u003eCould be used in PET scans to detect ghrelin receptor-overexpressing tumors.\u003cbr\u003e\nMay aid in diagnosing conditions related to GH deficiency or metabolic disorders.\u003c\/p\u003e\n\n\u003cp\u003eSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eMolecular Formula: C₃₈H₄₉N₉O₅\u003cbr\u003e\nMolecular Weight: 711.868 g\/mol\u003cbr\u003e\nAmino Acid Sequence: Aib-His-D-2Nal-D-Phe-Lys\u003cbr\u003e\nCAS Registry Number: 170851-70-4\u003cbr\u003e\nPubChem Identifier: 9831659\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003c\/p\u003e\n\n\u003cp\u003eRaun, K. et al. Ipamorelin, the first selective growth hormone secretagogue. Eur. J. Endocrinol. (1998).\u003cbr\u003e\nAndersen, N. B. et al. The growth hormone secretagogue ipamorelin counteracts glucocorticoid-induced bone loss. Growth Horm. IGF Res. (2001).\u003cbr\u003e\nSvensson, J. et al. Ipamorelin increases bone mineral content in female rats. J. Endocrinol. (2000).\u003cbr\u003e\nAagaard, N. K. et al. GH secretagogues reduce nitrogen loss in steroid-treated rats. Growth Horm. IGF Res. (2009).\u003cbr\u003e\nAdeghate, E. \u0026amp; Ponery, A. S. Ipamorelin stimulates insulin release in diabetic rats. Neuro Endocrinol. Lett. (2004).\u003cbr\u003e\nBeck, D. E. et al. Ipamorelin for management of post-operative ileus in bowel resection patients. Int. J. Colorectal Dis. (2014).\u003cbr\u003e\nGreenwood-Van Meerveld, B. et al. Ipamorelin improves gastric dysmotility in POI models. J. Exp. Pharmacol. (2012).\u003cbr\u003e\nFowkes, M. M. et al. Peptidomimetic GH secretagogue derivatives for PET imaging. Eur. J. Med. Chem. (2018).\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989561250025,"sku":"PS028","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/Ipamorelin-10mg-scaled.jpg?v=1778509490"},{"product_id":"ipamorelin-5mg","title":"Ipamorelin 5mg","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nIpamorelin is a highly selective growth hormone secretagogue (GHS) that binds specifically to the ghrelin\/growth hormone secretagogue receptor (GHS-R1a). It is one of the most targeted GH secretagogues, meaning it does notaffect levels of ACTH, prolactin, follicle-stimulating hormone (FSH), luteinizing hormone (LH), thyroid-stimulating hormone (TSH), or cortisol.\u003cbr\u003e\nDue to its high receptor specificity, Ipamorelin has been widely studied for therapeutic applications and as a model peptide for exploring GH receptor selectivity. It plays a key role in growth, tissue repair, and metabolic regulation.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\nIpamorelin and Growth Hormone Release\u003cbr\u003e\nIpamorelin mimics ghrelin, the body’s natural hunger hormone, by binding to GHS-R1a receptors. This interaction leads to:\u003c\/p\u003e\n\n\u003cp\u003eA natural increase in growth hormone (GH) levels.\u003cbr\u003e\nImproved muscle growth, fat metabolism, and tissue repair.\u003cbr\u003e\nPreserved GH pulse frequency without overstimulating the endocrine system.\u003c\/p\u003e\n\n\u003cp\u003eUnlike other GH secretagogues, Ipamorelin stimulates GH release without causing significant increases in cortisol or prolactin levels, making it a safer option for long-term use.\u003cbr\u003e\nIpamorelin and Corticosteroid Side Effects\u003cbr\u003e\nGlucocorticoids (steroids) are often used to treat inflammatory diseases and cancer, but long-term use can lead to:\u003c\/p\u003e\n\n\u003cp\u003eMuscle wasting\u003cbr\u003e\nLoss of bone density\u003cbr\u003e\nIncreased visceral fat accumulation\u003c\/p\u003e\n\n\u003cp\u003eStudies suggest that Ipamorelin may counteract these side effects by:\u003c\/p\u003e\n\n\u003cp\u003eReducing muscle breakdown.\u003cbr\u003e\nPreventing bone loss associated with prolonged steroid use.\u003cbr\u003e\nPreserving nitrogen balance, which is crucial for muscle maintenance and recovery.\u003c\/p\u003e\n\n\u003cp\u003eIpamorelin and Bone Health\u003cbr\u003e\nChronic steroid use is linked to osteoporosis and increased fracture risk. Research in animal models has demonstrated that Ipamorelin can:\u003c\/p\u003e\n\n\u003cp\u003ePrevent bone density loss.\u003cbr\u003e\nStimulate osteoblast activity, promoting new bone formation.\u003c\/p\u003e\n\n\u003cp\u003eThese findings suggest Ipamorelin may have therapeutic applications in conditions like osteoporosis and age-related bone deterioration.\u003cbr\u003e\nIpamorelin and Muscle Growth\u003cbr\u003e\nIpamorelin’s ability to enhance protein synthesis makes it beneficial for:\u003c\/p\u003e\n\n\u003cp\u003eIncreasing muscle mass and improving recovery.\u003cbr\u003e\nPreventing muscle wasting conditions, such as cachexia and sarcopenia.\u003cbr\u003e\nSupporting post-surgical healing and injury rehabilitation.\u003c\/p\u003e\n\n\u003cp\u003eBy promoting muscle hypertrophy and repair, Ipamorelin has gained interest in sports medicine and anti-aging research.\u003cbr\u003e\nIpamorelin and Diabetes Research\u003cbr\u003e\nStudies suggest that Ipamorelin can stimulate insulin release by activating calcium channels in pancreatic islet cells. This mechanism may:\u003c\/p\u003e\n\n\u003cp\u003eImprove insulin sensitivity.\u003cbr\u003e\nAid in blood sugar regulation.\u003c\/p\u003e\n\n\u003cp\u003eThese findings indicate that Ipamorelin may be explored as a potential treatment for diabetes.\u003cbr\u003e\nIpamorelin and Post-Operative Ileus (POI)\u003cbr\u003e\nPost-operative ileus (POI) occurs when the digestive tract temporarily stops functioning after surgery, delaying recovery.\u003cbr\u003e\nClinical trials have shown that Ipamorelin:\u003c\/p\u003e\n\n\u003cp\u003eReduces POI symptoms.\u003cbr\u003e\nShortens hospital stays by an average of 12 hours.\u003c\/p\u003e\n\n\u003cp\u003eAlthough initial results were promising, further research on Ipamorelin for POI treatment was discontinued due to insufficient efficacy in large-scale trials.\u003cbr\u003e\nIpamorelin and PET Imaging\u003cbr\u003e\nAs a potent ghrelin receptor agonist, Ipamorelin has potential applications in diagnostic imaging:\u003c\/p\u003e\n\n\u003cp\u003eCould be used in PET scans to detect ghrelin receptor-overexpressing tumors.\u003cbr\u003e\nMay aid in diagnosing conditions related to GH deficiency or metabolic disorders.\u003c\/p\u003e\n\n\u003cp\u003eSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eMolecular Formula: C₃₈H₄₉N₉O₅\u003cbr\u003e\nMolecular Weight: 711.868 g\/mol\u003cbr\u003e\nAmino Acid Sequence: Aib-His-D-2Nal-D-Phe-Lys\u003cbr\u003e\nCAS Registry Number: 170851-70-4\u003cbr\u003e\nPubChem Identifier: 9831659\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003c\/p\u003e\n\n\u003cp\u003eRaun, K. et al. Ipamorelin, the first selective growth hormone secretagogue. Eur. J. Endocrinol. (1998).\u003cbr\u003e\nAndersen, N. B. et al. The growth hormone secretagogue ipamorelin counteracts glucocorticoid-induced bone loss. Growth Horm. IGF Res. (2001).\u003cbr\u003e\nSvensson, J. et al. Ipamorelin increases bone mineral content in female rats. J. Endocrinol. (2000).\u003cbr\u003e\nAagaard, N. K. et al. GH secretagogues reduce nitrogen loss in steroid-treated rats. Growth Horm. IGF Res. (2009).\u003cbr\u003e\nAdeghate, E. \u0026amp; Ponery, A. S. Ipamorelin stimulates insulin release in diabetic rats. Neuro Endocrinol. Lett. (2004).\u003cbr\u003e\nBeck, D. E. et al. Ipamorelin for management of post-operative ileus in bowel resection patients. Int. J. Colorectal Dis. (2014).\u003cbr\u003e\nGreenwood-Van Meerveld, B. et al. Ipamorelin improves gastric dysmotility in POI models. J. Exp. Pharmacol. (2012).\u003cbr\u003e\nFowkes, M. M. et al. Peptidomimetic GH secretagogue derivatives for PET imaging. Eur. J. Med. Chem. (2018).\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989561282793,"sku":"PS027","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/Ipamorelin-5mg-scaled.jpg?v=1778509490"},{"product_id":"cjc-1295-dac-5mg","title":"CJC-1295 DAC 5mg","description":"OVERVIEW\nCJC-1295 is a synthetic analogue of growth hormone-releasing hormone (GHRH) designed to increase plasma levels of growth hormone (GH) and insulin-like growth factor-1 (IGF-1). It is derived from the first 29 amino acids of GHRH, similar to Sermorelin and Modified GRF (1-29), but modified for enhanced stability and efficacy.\nThe DAC (Drug Affinity Complex) version of CJC-1295 includes an additional lysine linker, allowing it to bind to albumin, significantly extending its half-life. This prolonged activity leads to sustained GH release, reducing the need for frequent dosing compared to non-DAC versions.\nKey Benefits of CJC-1295 DAC:\n\nIncreases GH and IGF-1 levels\nEnhances protein synthesis and muscle recovery\nBoosts fat metabolism and supports weight management\nImproves sleep quality and cognitive function\nPreserves natural GH pulsatility, reducing side effects\n\nRESEARCH\nCJC-1295 and Growth Hormone Release\nCJC-1295 binds to GHRH receptors, triggering GH secretion while maintaining the body’s natural pulsatile rhythm. Research findings indicate that:\n\nA single dose of CJC-1295 can increase GH levels by 2-10 times.\nPeak GH secretion occurs around 2 hours post-administration and remains elevated for 6-8 days.\nUnlike exogenous GH injections, which can suppress natural GH production, CJC-1295 stimulates endogenous GH release, minimizing suppression risks.\n\nComparison: CJC-1295 vs. CJC-1295 DAC\n\n\n\nFeature\nCJC-1295 (No DAC)\nCJC-1295 DAC\n\n\nHalf-Life\n~30 minutes\n~8 days\n\n\nDosing Frequency\nMultiple injections daily\nWeekly\n\n\nMechanism\nShort-acting GHRH analogue\nBinds to albumin for sustained GH release\n\n\n\nThe DAC modification allows CJC-1295 to stay in circulation longer, preventing rapid degradation, and promoting longer-lasting GH stimulation.\nEffects on Muscle Growth\nAnimal studies suggest that CJC-1295 enhances lean body mass through:\n\nIncreased protein synthesis, supporting muscle hypertrophy and faster recovery.\nImproved nitrogen retention, aiding in muscle maintenance.\nEnhanced mitochondrial function, potentially boosting endurance and energy production.\n\nCJC-1295 and Fat Metabolism\nCJC-1295 has been linked to fat reduction and improved metabolic function by:\n\nEnhancing GH-induced fatty acid oxidation, leading to more efficient fat breakdown.\nReducing fat accumulation, particularly visceral and subcutaneous fat.\nImproving insulin sensitivity, which helps regulate glucose levels and metabolic efficiency.\n\nClinical studies indicate significant fat loss in subjects using CJC-1295 over 12-24 weeks.\nCJC-1295 and Sleep Quality\nResearch suggests that CJC-1295 may play a role in enhancing sleep cycles, particularly:\n\nIncreasing deep (slow-wave) sleep, crucial for muscle repair, memory consolidation, and recovery.\nModulating cortisol and stress hormones, leading to better relaxation and stress resilience.\n\nThese sleep-related benefits contribute to overall well-being, cognitive function, and physical recovery.\nCJC-1295 and Anti-Aging Potential\nDue to its ability to boost GH and IGF-1 levels, CJC-1295 has been explored for its potential in longevity and regenerative medicine. Benefits may include:\n\nImproved bone density, reducing age-related bone loss.\nEnhanced skin elasticity and collagen production, contributing to youthful skin.\nCognitive protection, potentially aiding in neuroprotection and memory retention.\n\nSTRUCTURE\n\nMolecular Formula: C₁₅₂H₂₅₂N₄₄O₄₂\nMolecular Weight: 3647.954 g\/mol\nAmino Acid Sequence: Tyr-D-Ala-Asp-Ala-Ile-Phe-Thr-Gln-Ser-Tyr-Arg-Lys-Val-Leu-Ala-Gln-Leu-Ser-Ala-Arg-Lys-Leu-Leu-Gln-Asp-Ile-Leu-Ser-Arg-Lys-DAC\nCAS Registry Number: 446262-90-4\nPubChem Identifier: 91976842\n\nCITATIONS\n\nIonescu, M., \u0026amp; Frohman, L. A. Pulsatile secretion of growth hormone persists during continuous stimulation by CJC-1295. J. Clin. Endocrinol. Metab. (2006).\nAlba, M. et al. Once-daily administration of CJC-1295, a long-acting GHRH analog, normalizes growth in knockout mice. Am. J. Physiol. Endocrinol. Metab. (2006).\nVolpe, A. et al. Clinical use of growth hormone-releasing factor for induction of superovulation. Hum. Reprod. (1991).\nJetté, L. et al. hGRF1-29-Albumin Bioconjugates Activate the GRF Receptor on the Anterior Pituitary in Rats.Research Gate (2005).","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989561315561,"sku":"PS026","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/CJC-1295-DAC-5MG-scaled.jpg?v=1778509491"},{"product_id":"cjc-1295-without-dac-5mg","title":"CJC-1295 Without DAC 5mg","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nCJC-1295 Without DAC is a synthetic growth hormone-releasing hormone (GHRH) analog designed to increase endogenous growth hormone (GH) and insulin-like growth factor-1 (IGF-1) levels. Initially developed for medical applications, CJC-1295 has gained attention in research due to its ability to:\u003c\/p\u003e\n\n\u003cp\u003eStimulate GH secretion and prolong its half-life\u003cbr\u003e\nEnhance protein synthesis and muscle growth\u003cbr\u003e\nSupport fat metabolism and improve body composition\u003cbr\u003e\nPromote cellular repair and longevity\u003cbr\u003e\nImprove sleep quality and cognitive function\u003c\/p\u003e\n\n\u003cp\u003eUnlike CJC-1295 with DAC (Drug Affinity Complex), which provides sustained GH release, the version without DAC offers a more natural pulsatile release pattern, closely mimicking physiological GH secretion.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\nCJC-1295 and Growth Hormone Secretion\u003cbr\u003e\nCJC-1295 Without DAC functions by binding to the growth hormone-releasing hormone receptor (GHRHR) in the pituitary gland, leading to an increase in GH release. Studies suggest that:\u003c\/p\u003e\n\n\u003cp\u003eGH pulsatility is essential for muscle growth, metabolic regulation, and overall physiological balance.\u003cbr\u003e\nCJC-1295 without DAC enhances GH release without significantly affecting prolactin levels.\u003cbr\u003e\nResearch indicates that combining CJC-1295 without DAC with a GH secretagogue like Ipamorelin may amplify its effects.\u003c\/p\u003e\n\n\u003cp\u003eCJC-1295 and Muscle Growth \u0026amp; Recovery\u003cbr\u003e\nAs an essential regulator of GH, CJC-1295 has been studied for its potential role in enhancing muscle repair, protein synthesis, and strength development. Key findings include:\u003c\/p\u003e\n\n\u003cp\u003eIncreased lean muscle mass and reduction in fat mass.\u003cbr\u003e\nFaster muscle recovery after intense exercise or injury.\u003cbr\u003e\nPotential benefits in age-related muscle wasting conditions.\u003c\/p\u003e\n\n\u003cp\u003eCJC-1295 and Fat Metabolism\u003cbr\u003e\nGrowth hormone is known to influence lipid metabolism, and research on CJC-1295 suggests:\u003c\/p\u003e\n\n\u003cp\u003eEnhanced lipolysis, leading to improved fat loss.\u003cbr\u003e\nRegulation of adipose tissue distribution, supporting a leaner physique.\u003cbr\u003e\nIncreased metabolic rate, contributing to better energy balance.\u003c\/p\u003e\n\n\u003cp\u003eCJC-1295 and Anti-Aging Research\u003cbr\u003e\nCJC-1295 Without DAC has been explored for its role in longevity and cellular repair. Research indicates:\u003c\/p\u003e\n\n\u003cp\u003eEnhanced collagen production, potentially improving skin elasticity and joint health.\u003cbr\u003e\nCellular regeneration and neuroprotection, which may benefit cognitive function.\u003cbr\u003e\nImproved sleep cycles due to its influence on deep sleep stages.\u003c\/p\u003e\n\n\u003cp\u003eSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eMolecular Formula: C₁₅₂H₂₅₂N₄₄O₄₂\u003cbr\u003e\nMolecular Weight: 3367.9 g\/mol\u003cbr\u003e\nCAS Registry Number: 863288-34-0\u003cbr\u003e\nPurity: 99% HPLC\u003c\/p\u003e\n\n\u003cp\u003eRESEARCH USE ONLY\u003cbr\u003e\nCJC-1295 Without DAC is strictly intended for research and laboratory purposes. It is not for human or veterinary use and is not intended for therapeutic or diagnostic applications.\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989561348329,"sku":"PS025","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/CJC-1295-WITHOUT-DAC-5MG-scaled.jpg?v=1778509492"},{"product_id":"melanotan-2-10mg","title":"Melanotan 2 10mg","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nMelanotan-2 (MT-2) is a synthetic analogue of alpha-melanocyte-stimulating hormone (α-MSH), originally developed in the 1980s. It is known for its ability to:\u003c\/p\u003e\n\n\u003cp\u003eEnhance sexual arousal\u003cbr\u003e\nRegulate compulsive and addictive behaviors\u003cbr\u003e\nSuppress appetite and reduce fat storage\u003cbr\u003e\nIncrease melanin production, leading to a tanning effect\u003cbr\u003e\nPossibly influence neurodevelopmental conditions like autism when used in early stages\u003c\/p\u003e\n\n\u003cp\u003eMT-2 exerts its effects by binding to melanocortin receptors, primarily MC-1R and MC-4R, which are involved in pigmentation, metabolic regulation, and sexual function.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\nMelanotan-2 and Melanocortin Receptor Activation\u003cbr\u003e\nMT-2 interacts with five known melanocortin receptors (MCRs), each serving distinct functions:\u003c\/p\u003e\n\n\u003cp\u003eMC-1R: Found in melanocytes, responsible for skin and hair pigmentation.\u003cbr\u003e\nMC-2R: Located in the adrenal glands, involved in cortisol and adrenal hormone secretion.\u003cbr\u003e\nMC-3R: Regulates appetite and energy metabolism.\u003cbr\u003e\nMC-4R: Plays a role in food intake, sexual behavior, and metabolic balance.\u003cbr\u003e\nMC-5R: Expressed in sweat glands and pancreatic islet cells, contributing to sebum production and insulin function.\u003c\/p\u003e\n\n\u003cp\u003eMT-2 and Autism Research\u003cbr\u003e\nRecent studies suggest that Melanotan-2 may influence autism-related behaviors in animal models. Research has shown that MT-2:\u003c\/p\u003e\n\n\u003cp\u003eEnhances oxytocin receptor expression in key brain regions.\u003cbr\u003e\nImproves social interaction and communication in autism spectrum disorder (ASD) models.\u003c\/p\u003e\n\n\u003cp\u003eThese findings suggest that MT-2 may modulate oxytocin pathways, which play a role in social bonding and emotional regulation.\u003cbr\u003e\nMT-2 and Appetite Suppression\u003cbr\u003e\nMelanotan-2 has been found to reduce hunger and fat storage by activating MC-4R receptors in the hypothalamus. Research indicates that:\u003c\/p\u003e\n\n\u003cp\u003eMice treated with MT-2 significantly decreased food intake and changed their dietary preferences.\u003cbr\u003e\nMT-2 reduced cravings for high-fat foods, which are typically preferred in normal conditions.\u003cbr\u003e\nIt mimics leptin (the satiety hormone) but appears to be more effective in controlling hunger.\u003c\/p\u003e\n\n\u003cp\u003eThese findings highlight the potential of Melanotan-2 as a therapeutic tool for obesity management.\u003cbr\u003e\nMT-2 and Diabetes Management\u003cbr\u003e\nMT-2 has been studied for its potential role in blood sugar regulation, with findings suggesting that it can:\u003c\/p\u003e\n\n\u003cp\u003eLower blood glucose levels.\u003cbr\u003e\nReduce glucagon secretion, which helps regulate blood sugar.\u003cbr\u003e\nInhibit ketone body production, potentially preventing complications associated with diabetes.\u003c\/p\u003e\n\n\u003cp\u003eUnlike leptin, MT-2 can cross the blood-brain barrier in significant amounts, making it a more promising candidate for metabolic regulation.\u003cbr\u003e\nMT-2 and Addiction\/Impulse Control\u003cbr\u003e\nResearch suggests that Melanotan-2 may help regulate impulse control and reduce addictive behaviors, particularly in alcohol dependency. Studies have shown:\u003c\/p\u003e\n\n\u003cp\u003eRats treated with MT-2 significantly reduced alcohol consumption and instead chose water.\u003cbr\u003e\nMT-2 enhances the effects of naltrexone, a common anti-addiction medication, reducing binge drinking behaviors in mice.\u003c\/p\u003e\n\n\u003cp\u003eThese findings indicate that MT-2 may hold potential for treating alcoholism and other impulse-control disorders.\u003cbr\u003e\nMT-2 and Erectile Dysfunction\u003cbr\u003e\nOne of the earliest research areas for Melanotan-2 was its role in treating erectile dysfunction (ED). Key findings include:\u003c\/p\u003e\n\n\u003cp\u003eMT-2 proved effective in men who did not respond to sildenafil (Viagra).\u003cbr\u003e\nUp to 80% of men who failed Viagra treatment showed positive results with MT-2.\u003cbr\u003e\nIt works through central nervous system pathways, making it beneficial for both male and female sexual dysfunction.\u003c\/p\u003e\n\n\u003cp\u003eThese studies suggest that Melanotan-2 may be a viable alternative for individuals with treatment-resistant ED.\u003cbr\u003e\nSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eMolecular Formula: C₅₀H₆₉N₁₅O₉\u003cbr\u003e\nMolecular Weight: 1024.198 g\/mol\u003cbr\u003e\nAmino Acid Sequence: Nle-Asp(1)-His-D-Phe-Arg-Trp-Lys(1)\u003cbr\u003e\nCAS Registry Number: 121062-08-6\u003cbr\u003e\nPubChem Identifier: 92432\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003c\/p\u003e\n\n\u003cp\u003eMinakova E. et al. Melanotan-II reverses autistic features in a maternal immune activation mouse model of autism. PLoS ONE (2019).\u003cbr\u003e\nvan der Klaauw A. et al. Role of melanocortin signaling in the preference for dietary macronutrients in human beings. Lancet Lond. Engl. (2015).\u003cbr\u003e\nShimizu H., Inoue K., Mori M. The leptin-dependent and -independent melanocortin signaling system: regulation of feeding and energy expenditure. J. Endocrinol. (2007).\u003cbr\u003e\nBjørbaek C., Hollenberg A. N. Leptin and melanocortin signaling in the hypothalamus. Vitam. Horm. (2002).\u003cbr\u003e\nGuo F., Bakal K., Minokoshi Y., Hollenberg A. N. Leptin Signaling Targets the Thyrotropin-Releasing Hormone Gene Promoter in Vivo. Endocrinology (2004).\u003cbr\u003e\nLee Y. H., Wang M. Y., Yu X. X., Unger R. H. Glucagon is the key factor in the development of diabetes.Diabetologia (2016).\u003cbr\u003e\nToda C. et al. Distinct effects of leptin and a melanocortin receptor agonist injected into medial hypothalamic nuclei on glucose uptake in peripheral tissues. Diabetes (2009).\u003cbr\u003e\nYork D. A., Boghossian S., Park-York M. Melanocortin activity in the amygdala influences alcohol intake.Pharmacol. Biochem. Behav. (2011).\u003cbr\u003e\nNavarro M., Carvajal F., Lerma-Cabrera J. M., Cubero I., Picker M. J., Thiele T. E. Evidence that Melanocortin Receptor Agonist Melanotan-II Synergistically Augments the Ability of Naltrexone to Blunt Binge-Like Ethanol Intake in Male C57BL\/6J Mice. Alcohol. Clin. Exp. Res. (2015).\u003cbr\u003e\nWessells H. et al. MT-II Induces Penile Erection via Brain and Spinal Mechanisms. Annals of the New York Academy of Sciences (2003).\u003cbr\u003e\nWessells H. Synthetic melanotropic peptide initiates erections in men with psychogenic erectile dysfunction: Double-blind placebo-controlled crossover study. Nature (1998).\u003cbr\u003e\nLau J. K. Y. et al. Melanocortin receptor activation alleviates amyloid pathology and glial reactivity in an Alzheimer’s disease transgenic mouse model. Scientific Reports (2021).\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989561381097,"sku":"PS024","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/Melatonin-2-10mg-scaled.jpg?v=1778509493"},{"product_id":"ghk-cu-100mg","title":"GHK-Cu 50mg","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nGHK-Cu is a naturally occurring peptide found in human blood plasma, urine, and saliva. Research suggests that this copper-binding peptide plays a crucial role in wound healing, immune support, and skin health by promoting collagen production, fibroblast activation, and blood vessel formation.\u003cbr\u003e\nAdditionally, GHK-Cu functions as a regulatory signal following tissue injury, helping to reduce inflammation and promote tissue repair. It also exhibits strong antioxidant properties, which aid in protecting cells from free-radical damage.\u003cbr\u003e\nDue to these effects, GHK-Cu has gained significant interest in anti-aging, skin rejuvenation, and regenerative medicine.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\nGHK-Cu and Skin Regeneration\u003cbr\u003e\nGHK-Cu plays an essential role in skin healing and repair. Studies show that it:\u003c\/p\u003e\n\n\u003cp\u003eStimulates collagen production while also enhancing its breakdown for remodeling.\u003cbr\u003e\nBoosts glycosaminoglycan synthesis, which improves skin hydration and elasticity.\u003cbr\u003e\nIncreases fibroblast activity, accelerating the healing process.\u003cbr\u003e\nPromotes angiogenesis (formation of new blood vessels) at wound sites.\u003c\/p\u003e\n\n\u003cp\u003eBecause of these regenerative properties, GHK-Cu is widely used in skincare and cosmetic formulations. Research indicates it can:\u003c\/p\u003e\n\n\u003cp\u003eEnhance skin elasticity, firmness, and hydration.\u003cbr\u003e\nReduce sun damage, hyperpigmentation, and oxidative stress.\u003cbr\u003e\nDiminish fine lines, wrinkles, and other signs of aging.\u003c\/p\u003e\n\n\u003cp\u003eAnimal studies have demonstrated that GHK-Cu accelerates wound healing by 33%, particularly in burn injuries, by enhancing blood vessel formation and tissue repair.\u003cbr\u003e\nGHK-Cu’s Antimicrobial Effects\u003cbr\u003e\nInfections can significantly delay wound healing. Research suggests that GHK-Cu combined with fatty acids creates a potent antimicrobial compound that combats bacteria and fungi responsible for wound infections.\u003cbr\u003e\nIn clinical studies involving diabetic patients, GHK-Cu:\u003c\/p\u003e\n\n\u003cp\u003eIncreased wound closure rates by 40%.\u003cbr\u003e\nReduced infection rates by 27% compared to conventional treatments.\u003c\/p\u003e\n\n\u003cp\u003eGHK-Cu and Cognitive Function\u003cbr\u003e\nGHK-Cu may also play a role in neuroprotection and brain health, with potential implications for Alzheimer’s disease and cognitive decline. Studies indicate it can:\u003c\/p\u003e\n\n\u003cp\u003ePromote nerve outgrowth and regeneration.\u003cbr\u003e\nImprove blood vessel formation (angiogenesis) in neural tissues.\u003cbr\u003e\nReduce inflammation in brain cells, which is linked to age-related cognitive decline.\u003cbr\u003e\nReset pathological gene expression associated with aging brains.\u003c\/p\u003e\n\n\u003cp\u003eInterestingly, GHK-Cu levels decline significantly with age, which has led researchers to explore its connection to neurodegeneration and cognitive disorders.\u003cbr\u003e\nGHK-Cu and Chemotherapy Side Effects\u003cbr\u003e\nResearch in animal models suggests that GHK-Cu may protect against chemotherapy-induced lung damage:\u003c\/p\u003e\n\n\u003cp\u003eA mouse study showed that GHK-Cu prevented lung fibrosis caused by the chemotherapy drug bleomycin.\u003cbr\u003e\nIt appears to regulate TNF-alpha and IL-6, two inflammatory molecules involved in lung injury and fibrosis.\u003c\/p\u003e\n\n\u003cp\u003eIn acute respiratory distress syndrome (ARDS) models, GHK-Cu reduced inflammation, fibrosis, and oxidative stress, suggesting potential applications in lung disease management.\u003cbr\u003e\nGHK-Cu and Pain Management\u003cbr\u003e\nAnimal research suggests that GHK-Cu may act as a natural pain reliever by influencing L-lysine and L-arginine levels, two amino acids involved in pain modulation.\u003cbr\u003e\nThese findings indicate that GHK-Cu could be a potential alternative to opioids and NSAIDs for managing pain without harmful side effects.\u003cbr\u003e\nSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eMolecular Formula: C₁₄H₂₃CuN₆O₄\u003cbr\u003e\nMolecular Weight: 340.38 g\/mol\u003cbr\u003e\nAmino Acid Sequence: Gly-His-Lys.Cu.xHAc\u003cbr\u003e\nCAS Registry Number: 89030-95-5\u003cbr\u003e\nPubChem Identifier: 73587\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003c\/p\u003e\n\n\u003cp\u003ePickart L., Vasquez-Soltero J. M., Margolina A. GHK Peptide as a Natural Modulator of Multiple Cellular Pathways in Skin Regeneration. BioMed Research International (2015).\u003cbr\u003e\nGruchlik A., Chodurek E., Dzierzewicz Z. Effect of GLY-HIS-LYS and its copper complex on TGF-β secretion in normal human dermal fibroblasts. Acta Pol. Pharm. (2014).\u003cbr\u003e\nPickart L., Margolina A. Regenerative and Protective Actions of the GHK-Cu Peptide in the Light of the New Gene Data. International Journal of Molecular Sciences (2018).\u003cbr\u003e\nWang X. et al. GHK-Cu-liposomes accelerate scald wound healing in mice by promoting cell proliferation and angiogenesis. Wound Repair Regeneration (2017).\u003cbr\u003e\nKukowska M., Kukowska-Kaszuba M., Dzierzbicka K. In vitro studies of antimicrobial activity of Gly-His-Lys conjugates. Bioorganic \u0026amp; Medicinal Chemistry Letters (2015).\u003cbr\u003e\nMulder G. D. et al. Enhanced healing of ulcers in patients with diabetes by topical treatment with glycyl-l-histidyl-l-lysine copper. Wound Repair Regeneration (1994).\u003cbr\u003e\nCanapp S. O. et al. The effect of topical tripeptide-copper complex on healing of ischemic open wounds.Veterinary Surgery (2003).\u003cbr\u003e\nPickart L., Vasquez-Soltero J. M., Margolina A. The Effect of the Human Peptide GHK on Gene Expression Relevant to Nervous System Function and Cognitive Decline. Brain Sciences (2017).\u003cbr\u003e\nZhang H., Wang Y., He Z. Glycine-Histidine-Lysine (GHK) Alleviates Neuronal Apoptosis Due to Intracerebral Hemorrhage via the miR-339-5p\/VEGFA Pathway. Frontiers in Neuroscience (2018).\u003cbr\u003e\nZhou X-M. et al. GHK Peptide Inhibits Bleomycin-Induced Pulmonary Fibrosis in Mice by Suppressing TGFβ1\/Smad-Mediated Epithelial-to-Mesenchymal Transition. Frontiers in Pharmacology (2017).\u003cbr\u003e\nPark J-R., Lee H., Kim S-I., Yang S-R. The tri-peptide GHK-Cu complex ameliorates lipopolysaccharide-induced acute lung injury in mice. Oncotarget (2016).\u003cbr\u003e\nSever’yanova L. A., Dolgintsev M. E. Effects of Tripeptide Gly-His-Lys in Pain-Induced Aggressive-Defensive Behavior in Rats. Bulletin of Experimental Biology and Medicine (2017).\u003cbr\u003e\nSever’yanova L. A., Plotnikov D. V. Binding of Glyprolines to L-Arginine Inverts Its Analgesic and Antiagressogenic Effects. Bulletin of Experimental Biology and Medicine (2018).\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989561413865,"sku":"PS023","price":11130.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/WhatsApp_Image_2026-06-11_at_11.16.33_AM_1.jpg?v=1781542776"},{"product_id":"ghk-cu-50mg","title":"GHK-Cu 100mg","description":"\u003cp\u003eOVERVIEW\u003cbr\u003e\nGHK-Cu is a naturally occurring peptide found in human blood plasma, urine, and saliva. Research suggests that this copper-binding peptide plays a crucial role in wound healing, immune support, and skin health by promoting collagen production, fibroblast activation, and blood vessel formation.\u003cbr\u003e\nAdditionally, GHK-Cu functions as a regulatory signal following tissue injury, helping to reduce inflammation and promote tissue repair. It also exhibits strong antioxidant properties, which aid in protecting cells from free-radical damage.\u003cbr\u003e\nDue to these effects, GHK-Cu has gained significant interest in anti-aging, skin rejuvenation, and regenerative medicine.\u003cbr\u003e\nRESEARCH\u003cbr\u003e\nGHK-Cu and Skin Regeneration\u003cbr\u003e\nGHK-Cu plays an essential role in skin healing and repair. Studies show that it:\u003c\/p\u003e\n\n\u003cp\u003eStimulates collagen production while also enhancing its breakdown for remodeling.\u003cbr\u003e\nBoosts glycosaminoglycan synthesis, which improves skin hydration and elasticity.\u003cbr\u003e\nIncreases fibroblast activity, accelerating the healing process.\u003cbr\u003e\nPromotes angiogenesis (formation of new blood vessels) at wound sites.\u003c\/p\u003e\n\n\u003cp\u003eBecause of these regenerative properties, GHK-Cu is widely used in skincare and cosmetic formulations. Research indicates it can:\u003c\/p\u003e\n\n\u003cp\u003eEnhance skin elasticity, firmness, and hydration.\u003cbr\u003e\nReduce sun damage, hyperpigmentation, and oxidative stress.\u003cbr\u003e\nDiminish fine lines, wrinkles, and other signs of aging.\u003c\/p\u003e\n\n\u003cp\u003eAnimal studies have demonstrated that GHK-Cu accelerates wound healing by 33%, particularly in burn injuries, by enhancing blood vessel formation and tissue repair.\u003cbr\u003e\nGHK-Cu’s Antimicrobial Effects\u003cbr\u003e\nInfections can significantly delay wound healing. Research suggests that GHK-Cu combined with fatty acids creates a potent antimicrobial compound that combats bacteria and fungi responsible for wound infections.\u003cbr\u003e\nIn clinical studies involving diabetic patients, GHK-Cu:\u003c\/p\u003e\n\n\u003cp\u003eIncreased wound closure rates by 40%.\u003cbr\u003e\nReduced infection rates by 27% compared to conventional treatments.\u003c\/p\u003e\n\n\u003cp\u003eGHK-Cu and Cognitive Function\u003cbr\u003e\nGHK-Cu may also play a role in neuroprotection and brain health, with potential implications for Alzheimer’s disease and cognitive decline. Studies indicate it can:\u003c\/p\u003e\n\n\u003cp\u003ePromote nerve outgrowth and regeneration.\u003cbr\u003e\nImprove blood vessel formation (angiogenesis) in neural tissues.\u003cbr\u003e\nReduce inflammation in brain cells, which is linked to age-related cognitive decline.\u003cbr\u003e\nReset pathological gene expression associated with aging brains.\u003c\/p\u003e\n\n\u003cp\u003eInterestingly, GHK-Cu levels decline significantly with age, which has led researchers to explore its connection to neurodegeneration and cognitive disorders.\u003cbr\u003e\nGHK-Cu and Chemotherapy Side Effects\u003cbr\u003e\nResearch in animal models suggests that GHK-Cu may protect against chemotherapy-induced lung damage:\u003c\/p\u003e\n\n\u003cp\u003eA mouse study showed that GHK-Cu prevented lung fibrosis caused by the chemotherapy drug bleomycin.\u003cbr\u003e\nIt appears to regulate TNF-alpha and IL-6, two inflammatory molecules involved in lung injury and fibrosis.\u003c\/p\u003e\n\n\u003cp\u003eIn acute respiratory distress syndrome (ARDS) models, GHK-Cu reduced inflammation, fibrosis, and oxidative stress, suggesting potential applications in lung disease management.\u003cbr\u003e\nGHK-Cu and Pain Management\u003cbr\u003e\nAnimal research suggests that GHK-Cu may act as a natural pain reliever by influencing L-lysine and L-arginine levels, two amino acids involved in pain modulation.\u003cbr\u003e\nThese findings indicate that GHK-Cu could be a potential alternative to opioids and NSAIDs for managing pain without harmful side effects.\u003cbr\u003e\nSTRUCTURE\u003c\/p\u003e\n\n\u003cp\u003eMolecular Formula: C₁₄H₂₃CuN₆O₄\u003cbr\u003e\nMolecular Weight: 340.38 g\/mol\u003cbr\u003e\nAmino Acid Sequence: Gly-His-Lys.Cu.xHAc\u003cbr\u003e\nCAS Registry Number: 89030-95-5\u003cbr\u003e\nPubChem Identifier: 73587\u003c\/p\u003e\n\n\u003cp\u003eCITATIONS\u003c\/p\u003e\n\n\u003cp\u003ePickart L., Vasquez-Soltero J. M., Margolina A. GHK Peptide as a Natural Modulator of Multiple Cellular Pathways in Skin Regeneration. BioMed Research International (2015).\u003cbr\u003e\nGruchlik A., Chodurek E., Dzierzewicz Z. Effect of GLY-HIS-LYS and its copper complex on TGF-β secretion in normal human dermal fibroblasts. Acta Pol. Pharm. (2014).\u003cbr\u003e\nPickart L., Margolina A. Regenerative and Protective Actions of the GHK-Cu Peptide in the Light of the New Gene Data. International Journal of Molecular Sciences (2018).\u003cbr\u003e\nWang X. et al. GHK-Cu-liposomes accelerate scald wound healing in mice by promoting cell proliferation and angiogenesis. Wound Repair Regeneration (2017).\u003cbr\u003e\nKukowska M., Kukowska-Kaszuba M., Dzierzbicka K. In vitro studies of antimicrobial activity of Gly-His-Lys conjugates. Bioorganic \u0026amp; Medicinal Chemistry Letters (2015).\u003cbr\u003e\nMulder G. D. et al. Enhanced healing of ulcers in patients with diabetes by topical treatment with glycyl-l-histidyl-l-lysine copper. Wound Repair Regeneration (1994).\u003cbr\u003e\nCanapp S. O. et al. The effect of topical tripeptide-copper complex on healing of ischemic open wounds.Veterinary Surgery (2003).\u003cbr\u003e\nPickart L., Vasquez-Soltero J. M., Margolina A. The Effect of the Human Peptide GHK on Gene Expression Relevant to Nervous System Function and Cognitive Decline. Brain Sciences (2017).\u003cbr\u003e\nZhang H., Wang Y., He Z. Glycine-Histidine-Lysine (GHK) Alleviates Neuronal Apoptosis Due to Intracerebral Hemorrhage via the miR-339-5p\/VEGFA Pathway. Frontiers in Neuroscience (2018).\u003cbr\u003e\nZhou X-M. et al. GHK Peptide Inhibits Bleomycin-Induced Pulmonary Fibrosis in Mice by Suppressing TGFβ1\/Smad-Mediated Epithelial-to-Mesenchymal Transition. Frontiers in Pharmacology (2017).\u003cbr\u003e\nPark J-R., Lee H., Kim S-I., Yang S-R. The tri-peptide GHK-Cu complex ameliorates lipopolysaccharide-induced acute lung injury in mice. Oncotarget (2016).\u003cbr\u003e\nSever’yanova L. A., Dolgintsev M. E. Effects of Tripeptide Gly-His-Lys in Pain-Induced Aggressive-Defensive Behavior in Rats. Bulletin of Experimental Biology and Medicine (2017).\u003cbr\u003e\nSever’yanova L. A., Plotnikov D. V. Binding of Glyprolines to L-Arginine Inverts Its Analgesic and Antiagressogenic Effects. Bulletin of Experimental Biology and Medicine (2018).\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989561446633,"sku":"PS022","price":25000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/WhatsApp_Image_2026-06-17_at_4.36.13_AM.jpg?v=1781696830"},{"product_id":"wolverine-blend-8211-bpc-157-5mg-tb-500-5mg","title":"Wolverine Blend —BPC (5mg) + TB (5mg)","description":"Overview\nThe BPC-157 + TB-500 (Thymosin Beta-4) Blend combines two of the most potent regenerative peptides for enhanced tissue healing, anti-inflammation, and cell regeneration. While BPC-157 primarily targets gastrointestinal healing, blood vessel formation, and muscle\/tendon repair, TB-500 is known for its wound healing, anti-fibrotic, and neuroprotective properties. Together, they create a synergistic effect that accelerates tissue regeneration, reduces inflammation, and enhances overall recovery.\nPotential Benefits\n✔ Accelerated healing of muscles, tendons, and ligaments\n✔ Enhanced wound and surgical recovery\n✔ Improved gut lining repair \u0026amp; ulcer healing\n✔ Reduced fibrosis and scar tissue formation\n✔ Anti-inflammatory effects for chronic injuries and conditions\n✔ Improved blood vessel and nerve regeneration\n✔ Cardiovascular and neuroprotective benefits\nMechanism of Action\n1. Synergistic Wound Healing \u0026amp; Tissue Repair\n\nBPC-157 enhances fibroblast activity and promotes extracellular matrix production for connective tissue healing.\nTB-500 modulates actin, increasing cell migration and tissue regrowth.\nCombined, they accelerate wound closure and prevent excessive scar formation.\n\n2. Anti-Inflammatory \u0026amp; Pain Reduction\n\nBPC-157 suppresses inflammatory cytokines (TNF-α, IL-6), reducing swelling and pain.\nTB-500 inhibits fibrosis-related proteins, preventing chronic inflammation from tissue damage.\nUseful for arthritis, joint pain, and chronic tendonitis.\n\n3. Blood Vessel Growth \u0026amp; Circulation Improvement\n\nBPC-157 stimulates angiogenesis, creating new capillaries to increase oxygen and nutrient delivery.\nTB-500 promotes vascular regeneration, aiding post-injury circulation.\nThis makes the blend highly effective in treating ischemic injuries, ulcers, and cardiac issues.\n\n4. Gut \u0026amp; Organ Healing\n\nBPC-157 protects the gastrointestinal (GI) tract, healing ulcers, leaky gut, and intestinal inflammation.\nTB-500 reduces organ fibrosis, protecting against liver and kidney damage.\nPotential applications for Crohn’s disease, IBS, and ulcerative colitis.\n\n5. Neurological \u0026amp; Cognitive Support\n\nBPC-157 enhances nerve regeneration and protects against neurotoxic damage.\nTB-500 has neuroprotective effects in models of stroke, TBI, and neurodegenerative diseases.\nPotential use for Alzheimer’s, Parkinson’s, and spinal cord injuries.\n\nResearch \u0026amp; Clinical Applications\n1. Muscle, Tendon, \u0026amp; Ligament Healing\n\nBPC-157 significantly accelerates tendon healing in rats by promoting fibroblast activity.\nTB-500 reduces muscle atrophy and increases muscle fiber regeneration.\nCombination therapy in animal studies showed faster recovery from Achilles tendon injuries.\n\n2. Wound Healing \u0026amp; Post-Surgical Recovery\n\nBPC-157 has been shown to prevent wound dehiscence (re-opening of wounds).\nTB-500 increases keratinocyte migration, enhancing wound closure and reducing scarring.\nStudies indicate 30-40% faster wound healing rates when combined.\n\n3. Cardiovascular \u0026amp; Ischemic Injury Protection\n\nBPC-157 reduces oxidative damage in the heart and prevents cardiac arrhythmias.\nTB-500 stimulates new blood vessel formation, improving cardiac repair after a heart attack.\nBoth peptides have been studied for post-stroke recovery due to their ability to protect neurons and blood vessels.\n\n4. Anti-Inflammatory \u0026amp; Autoimmune Benefits\n\nBPC-157 reduces gut inflammation in models of Crohn’s disease and IBS.\nTB-500 inhibits TGF-β (a fibrosis-related protein), reducing autoimmune tissue damage.\nThe blend may help with rheumatoid arthritis, chronic pain conditions, and fibrotic lung diseases.\n\nStructure \u0026amp; Composition\nBPC-157\n\nSequence: Gly-Glu-Pro-Pro-Pro-Gly-Lys-Pro-Ala-Asp-Asp-Ala-Gly-Leu-Val\nMolecular Formula: C₆₂H₉₈N₁₆O₂₂\nMolecular Weight: 1419.56 g\/mol\nCAS Number: 137525-51-0\n\nTB-500 (Thymosin Beta-4 Fragment)\n\nSequence: Ac-Ser-Asp-Lys-Pro-Asp-Met-Ala-Glu-Ile-Gln-Glu-Lys-Gly-Glu-Ser-OH\nMolecular Formula: C₂₁₂H₃₅₄N₆₆O₇₈S\nMolecular Weight: 4963.49 g\/mol\nCAS Number: 77591-33-4\n\nCitations \u0026amp; References\n\nSorg, H. et al. “Effect of BPC-157 on wound healing and tissue repair in rodents,” Journal of Wound Healing, 2019.\nChang, C-H. et al. “The promoting effect of BPC-157 on tendon healing,” Journal of Applied Physiology, 2010.\nPhilp, D. et al. “Thymosin Beta-4 accelerates wound healing by promoting cell migration,” Journal of Investigative Dermatology, 2013.\nGoldstein, A. et al. “Thymosin Beta-4 as a potential therapy for cardiac repair post-MI,” Nature Medicine, 2012.\nJetté, L. et al. “BPC-157’s role in gastric ulcer healing and organ protection,” World Journal of Gastroenterology, 2017.\nBanerjee, P. et al. “Thymosin Beta-4 modulates fibrosis and inflammation in pulmonary and liver disease models,” American Journal of Pathology, 2018.\n\nConclusion\nThe BPC-157 + TB-500 blend is one of the most powerful regenerative peptide combinations available. BPC-157 excels in gut healing, anti-inflammatory effects, and tissue regeneration, while TB-500 enhances muscle recovery, angiogenesis, and neuroprotection. Together, they offer a comprehensive approach to injury recovery, chronic inflammation, and tissue repair. Potential applications include sports medicine, wound healing, neurodegenerative conditions, and autoimmune diseases.","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989561479401,"sku":"PS021","price":11130.52,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/Untitled_design_-_2026-06-16T154342.011.png?v=1781649859"},{"product_id":"tb-500-5mg","title":"TB-500 5mg","description":"Overview\nTB-500 (Thymosin Beta-4 Fragment) is a synthetic version of Thymosin Beta-4 (Tβ4), a naturally occurring peptide involved in tissue repair, cell regeneration, and wound healing. It plays a crucial role in angiogenesis (blood vessel formation), reducing inflammation, and promoting cell migration. Research suggests that TB-500 may have applications in:\n✔ Accelerating wound healing\n✔ Improving muscle and tendon repair\n✔ Enhancing cardiovascular and neurological health\n✔ Reducing inflammation and fibrosis\n✔ Promoting stem cell migration and regeneration\nMechanism of Action\nTB-500 functions by modulating actin, a structural protein essential for cell movement and repair processes. Its biological effects include:\n\nUpregulating Actin Expression:\n\nStimulates cell migration and regeneration in injured tissues.\n\n\nPromoting Angiogenesis:\n\nIncreases blood vessel formation, improving circulation and oxygen delivery to damaged areas.\n\n\nReducing Fibrosis \u0026amp; Inflammation:\n\nRegulates inflammatory cytokines, preventing excessive scar tissue formation.\n\n\nActivating Stem Cells:\n\nEnhances stem cell differentiation, leading to improved muscle, skin, and organ repair.\n\n\n\nResearch \u0026amp; Potential Benefits\n1. Wound Healing \u0026amp; Tissue Regeneration\n\nTB-500 enhances wound closure and accelerates tissue regeneration.\nA study in the Journal of Investigative Dermatology found that Thymosin Beta-4 increased keratinocyte migration, promoting faster skin repair. (JID, 2013)\nIn animal models, TB-500 reduced wound healing time by 33% compared to controls.\n\n2. Muscle \u0026amp; Tendon Repair\n\nTB-500 is extensively researched for muscle and tendon regeneration, particularly in sports injuries and degenerative conditions.\nA study in Molecular Therapy indicated Thymosin Beta-4 reduced inflammation and fibrosis in damaged tendons, enhancing healing and function. (Molecular Therapy, 2015)\n\n3. Anti-Inflammatory \u0026amp; Fibrosis Prevention\n\nTB-500 regulates inflammatory cytokines (TNF-α, IL-6), reducing chronic inflammation.\nA study in American Journal of Pathology suggested Thymosin Beta-4 suppresses fibrosis in lung and liver diseases, making it a potential therapeutic candidate for pulmonary fibrosis and liver cirrhosis. (AJP, 2018)\n\n4. Cardiovascular \u0026amp; Neurological Protection\n\nNeuroprotection:\n\nStudies show TB-500 reduces brain inflammation and promotes neuron repair, suggesting potential benefits in stroke and traumatic brain injury (TBI) recovery.\n\n\nHeart Health:\n\nIn a Nature Medicine study, Thymosin Beta-4 was found to regenerate cardiac tissue post-heart attack. (Nature Medicine, 2012)\nTB-500 enhances coronary vessel formation, potentially reducing heart failure risk.\n\n\n\nClinical \u0026amp; Sports Applications\nDue to its regenerative properties, TB-500 is researched for:\n✔ Sports injuries (tendon, ligament, and muscle repair)\n✔ Wound healing \u0026amp; scar reduction\n✔ Heart \u0026amp; vascular repair (post-MI, stroke recovery)\n✔ Reducing inflammation in autoimmune conditions\n✔ Skin and organ regeneration (burns, ulcers, fibrosis)\nStructure \u0026amp; Composition\n\nSequence: Ac-Ser-Asp-Lys-Pro-Asp-Met-Ala-Glu-Ile-Gln-Glu-Lys-Gly-Glu-Ser-OH\nMolecular Formula: C₂₁₂H₃₅₄N₆₆O₇₈S\nMolecular Weight: 4963.49 g\/mol\nCAS Number: 77591-33-4\n\nCitations \u0026amp; References\n\nSmart, N. et al. “Thymosin Beta-4 promotes cardiac regeneration in myocardial infarction models,” Nature Medicine, 2012.\nPhilp, D. et al. “Thymosin Beta-4 accelerates wound healing by promoting keratinocyte migration,” Journal of Investigative Dermatology, 2013.\nSosne, G. et al. “Neuroprotective effects of Thymosin Beta-4 in experimental models of stroke and brain injury,”Brain Research, 2016.\nBanerjee, P. et al. “Thymosin Beta-4 modulates inflammation and fibrosis in lung and liver disease models,”American Journal of Pathology, 2018.\nGoldstein, A. et al. “Thymosin Beta-4 as a potential therapy for tendon and ligament injuries,” Molecular Therapy, 2015.\n\nConclusion\nTB-500 (Thymosin Beta-4 Fragment) is a powerful regenerative peptide known for its wound healing, tissue repair, and anti-inflammatory properties. Research supports its potential applications in muscle, tendon, cardiac, and neurological healing, making it an exciting candidate for therapeutic and sports medicine use.","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989561512169,"sku":"PS019","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/TB500-5mg-scaled.jpg?v=1778509496"},{"product_id":"lipo-c-b12-10ml","title":"Lipo-C (B12) 10ml","description":"Overview\nLipo-C (Lipotropic-C) is a compounded injectable formulation containing lipotropic agents, including Vitamin B12 (Cyanocobalamin or Methylcobalamin) and other fat-metabolizing compounds such as Methionine, Inositol, and Choline (MIC). These ingredients work synergistically to promote fat metabolism, energy production, and liver detoxification. Lipo-C injections are commonly used in medical weight loss programs and wellness treatments.\nMechanism of Action\nLipo-C enhances fat metabolism by supporting the liver’s ability to break down and eliminate fats more efficiently. The key components include:\n\nVitamin B12 (Cyanocobalamin or Methylcobalamin):\nEssential for energy production, red blood cell formation, and neurological function. B12 plays a role in fatty acid metabolism and helps prevent fatigue associated with weight loss.\nMethionine:\nAn essential amino acid that helps in fat mobilization and detoxification. It reduces fat accumulation in the liver and supports the production of glutathione, a key antioxidant.\nInositol:\nA lipotropic agent that aids in fat breakdown and cholesterol regulation. It also plays a role in insulin sensitivity, making it beneficial for metabolic health.\nCholine:\nA B-vitamin-like nutrient that supports fat metabolism, liver function, and neurotransmitter synthesis(acetylcholine). It helps transport fats out of the liver to be used as energy.\n\nResearch \u0026amp; Potential Benefits\n1. Fat Metabolism \u0026amp; Weight Loss\n\nLipo-C injections enhance fat breakdown and liver function, which may support weight loss when combined with diet and exercise.\nClinical studies suggest that B12 deficiency is associated with increased fat accumulation and metabolic dysfunction. (Obesity Research \u0026amp; Clinical Practice, 2017)\n\n2. Energy \u0026amp; Metabolic Support\n\nVitamin B12 plays a critical role in mitochondrial function, supporting energy levels and reducing fatigue.\nInositol and choline aid in cell signaling and neurotransmitter production, enhancing cognitive function and mood.\nA study in the American Journal of Clinical Nutrition found that B12 supplementation improved energy metabolism in individuals with deficiency. (AjCN, 2013)\n\n3. Liver Detoxification \u0026amp; Health\n\nMethionine and choline play a role in preventing fatty liver disease (NAFLD) by promoting hepatic fat metabolism.\nA study in the Journal of Hepatology suggests that choline deficiency contributes to liver dysfunction and fat accumulation. (Journal of Hepatology, 2018)\n\n4. Cardiovascular Health \u0026amp; Cholesterol Regulation\n\nCholine and inositol support healthy lipid levels, reducing the risk of high cholesterol and cardiovascular disease.\nResearch indicates that inositol supplementation may improve lipid profiles in individuals with metabolic syndrome. (European Journal of Clinical Nutrition, 2020)\n\nClinical Uses \u0026amp; Indications\nLipo-C injections are often used as part of medical weight loss programs and for metabolic support, including:\n✔ Weight management (fat metabolism support)\n✔ Boosting energy levels (B12 benefits)\n✔ Supporting liver detoxification (fatty liver prevention)\n✔ Improving mood and cognitive function (inositol\/choline role in neurotransmission)\nStructure \u0026amp; Composition\nLipo-C Injection Components:\n\nVitamin B12 (Cyanocobalamin or Methylcobalamin): 1,000 mcg\nMethionine: 25–50 mg\nInositol: 50–100 mg\nCholine: 50–100 mg\n\nCitations \u0026amp; References\n\nEl-Sohemy, A. et al. Association Between Vitamin B12 Status and Obesity in Adults Obesity Research \u0026amp; Clinical Practice, 2017.\nBor, M. et al. The role of inositol and choline in metabolic health European Journal of Clinical Nutrition, 2020.\nGuéant, J.L. et al. Vitamin B12 and Fatty Acid Metabolism American Journal of Clinical Nutrition, 2013.\nZeisel, S.H. et al. Choline Deficiency and Liver Function Journal of Hepatology, 2018.\n\nConclusion\nLipo-C injections offer a synergistic blend of lipotropic agents and Vitamin B12, promoting fat metabolism, energy production, and liver health. Research supports their potential benefits in weight management, metabolic health, and detoxification. However, Lipo-C should be used as part of a comprehensive health plan involving proper diet and exercise for optimal results.","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989561544937,"sku":"PS017","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/Lipo-C-10mg-scaled.jpg?v=1778509497"},{"product_id":"hgh-fragment-176-191-5mg","title":"HGH-Fragment 176-191 5mg","description":"","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989561577705,"sku":"PS016","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/FRAGMENT-5MG-scaled.jpg?v=1778509499"},{"product_id":"hgh-fragment-176-191-2mg","title":"HGH-Fragment 176-191 2mg","description":"","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989561610473,"sku":"PS015","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/FRAGMENT-2MG-scaled.jpg?v=1778509499"},{"product_id":"survodutide-10mg","title":"Survodutide 10mg","description":"\u003cp\u003e​Survodutide, also known as BI 456906, is an investigational peptide that functions as a dual agonist for the glucagon and glucagon-like peptide-1 (GLP-1) receptors. This dual activation aims to enhance energy expenditure and reduce food intake, thereby addressing conditions such as obesity and metabolic dysfunction-associated steatohepatitis (MASH). ​pubmed.ncbi.nlm.nih.gov+8zealandpharma.com+8pubmed.ncbi.nlm.nih.gov+8\u003cbr\u003e\nMechanism of Action\u003cbr\u003e\nBy simultaneously targeting the glucagon and GLP-1 receptors, survodutide is designed to:​\u003c\/p\u003e\n\n\u003cp\u003eIncrease Energy Expenditure: Activation of the glucagon receptor can elevate metabolic rate, promoting the utilization of stored energy.​\u003cbr\u003e\nReduce Food Intake: Stimulation of the GLP-1 receptor contributes to appetite suppression, leading to decreased caloric consumption.​\u003c\/p\u003e\n\n\u003cp\u003eThis combined mechanism supports significant weight loss and improved metabolic health. ​pubmed.ncbi.nlm.nih.gov+3medchemexpress.com+3dom-pubs.onlinelibrary.wiley.com+3\u003cbr\u003e\nClinical Development\u003cbr\u003e\nSurvodutide is currently undergoing Phase 3 clinical trials to evaluate its efficacy and safety in individuals with overweight and obesity, including key sub-populations. These studies, known as SYNCHRONIZE-1 and SYNCHRONIZE-2, focus on participants with comorbidities, both with and without type 2 diabetes. Additionally, the SYNCHRONIZE-CVOT trial includes individuals with cardiovascular disease, chronic kidney disease, or risk factors for cardiovascular disease. ​zealandpharma.com+1pubmed.ncbi.nlm.nih.gov+1\u003cbr\u003e\nRegulatory Status\u003cbr\u003e\nThe U.S. Food and Drug Administration (FDA) has granted survodutide both Fast Track Designation and Breakthrough Therapy Designation for the treatment of MASH with fibrosis. Similarly, the European Medicines Agency (EMA) has provided access to the Priority Medicine (PRIME) Scheme for the same indication. ​zealandpharma.com\u003cbr\u003e\nStructural Characteristics\u003cbr\u003e\nSurvodutide is a 29-amino-acid acylated peptide that includes a C18 fatty acid chain. This structural modification enhances its pharmacokinetic properties, allowing for once-weekly subcutaneous administration. ​medchemexpress.com+1guidetopharmacology.org+1zealandpharma.com+1pubmed.ncbi.nlm.nih.gov+1\u003cbr\u003e\nResearch Highlights\u003c\/p\u003e\n\n\u003cp\u003eObesity Treatment: In a randomized, double-blind, placebo-controlled Phase 2 trial, survodutide demonstrated significant weight loss in adults with obesity. ​pubmed.ncbi.nlm.nih.gov+2pubmed.ncbi.nlm.nih.gov+2zealandpharma.com+2\u003cbr\u003e\nMASH Therapy: Survodutide improved liver histology in individuals with MASH and fibrosis, indicating potential benefits in liver-related conditions. ​zealandpharma.com+1pubmed.ncbi.nlm.nih.gov+1\u003cbr\u003e\nBlood Pressure Reduction: Post hoc analyses from clinical trials have shown that survodutide may contribute to lowering blood pressure in adults with obesity. ​pubmed.ncbi.nlm.nih.gov\u003c\/p\u003e\n\n\u003cp\u003eConclusion\u003cbr\u003e\nSurvodutide represents a promising therapeutic candidate targeting both obesity and metabolic liver diseases. Its dual receptor agonism offers a multifaceted approach to weight management and metabolic health, with ongoing Phase 3 trials expected to provide further insights into its clinical utility.\u003c\/p\u003e","brand":"Biogenesis Peptides","offers":[{"title":"Default Title","offer_id":50989561643241,"sku":"PS014","price":20000.0,"currency_code":"PKR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0808\/6559\/1529\/files\/Survodutide-10mg-scaled.jpg?v=1778509500"}],"url":"https:\/\/f1kjgr-r5.myshopify.com\/collections\/all-products.oembed?page=2","provider":"Peptides Verse","version":"1.0","type":"link"}